构建高校图书馆与农家书屋的协作机制推动区域经济持续发展

从活跃农民群众文化生活、发展和繁荣农村文化的角度出发，从促进社会主义农村经济的协调发展、全面提高农民综合素质入手，对当前“农家书屋”发展现状以及存在的问题进行分析，探讨了高校图书馆对“农家书屋”工程建设的优势和促进“农家书屋”工程建设的有效方法和途径。     

凝结水精处理安全运行探讨

文章简要叙述了大唐华银株洲发电有限公司凝结水精处理混床和再生系统设备及流程,出水水质及树脂性能等。针对火电厂凝结水精处理系统的安全运行问题,在设计以及运行操作方面结合大唐华银株洲发电有限公司凝结水精处理系统实际进行探讨。     

贵州省烤烟自育品种繁育与推广现状

为了解贵州省自育品种选育与推广现状,对2008～ 2012近5年贵州省烤烟品种种植结构、自育品种种植面积、比例和分布进行了统计与分析.结果表明:贵州省繁育推广的烟草品种主要包括自育品种、自繁品种、外繁品种3大类群.2008～2011年,外引品种一直占主导地位,2012年自育品种种植比例首次突破50％,成为贵州省推广面积最大的一个类群.在自育品种中,南江3号、韭菜坪2号单产和上等烟比率分别较对照K 326显著增加,其中,南江3号在2008 ～ 2012期间有3年种植面积超过3万hm2；韭菜坪2号2010 ～2012年连续3年种植面积超过0.67 hm2,是提升自育品种种植比例的主要因素.从产区分布来看,除在省内推广外,贵州省自育品种另外辐射到湖南、四川、重庆、陕西、河南、山东、广西等省市烟叶产区,2012年省外推广面积为1.39万hm2.     

江西稻田蜘蛛区系及种群动态研究

赣中北平原稻区常见的稻田蜘蛛有9科25种,其数量占捕食性天敌的80％以上。早稻田以微蛛所占比例最大;晚稻田除微蛛外,狼蛛和球腹蛛也有较大的比例。稻田蜘蛛数量变动与稻叶蝉、稻飞虱的消长大体一致,对这类害虫有明显的抑制作用,但晚稻后期仍需药剂防治。     

温度对玉米纹枯病菌生长与发病的影响

室内控制性测定结果表明,玉米纹枯病菌(RhizoctoniasolaniAG1鄄IA)菌丝生长的温度10～35℃,最适温度25～30℃;菌核萌发温度18～35℃,最适温度25～30℃。在幼苗和离体叶鞘上由菌丝引起侵染和发病的温度15～35℃,以25～30℃最适宜,潜育期最短,病斑较大。田间分期播种观察分析,气温和地面温度与病害始见的历期(播种期至病害初见期)分别呈显著负相关和极显著负相关。在田间自然条件下,始病的温度为20℃左右。这些结果为了解玉米纹枯病的发生规律,开展预测预报提供了科学依据。     

内生细菌B_(47)菌株的鉴定及其对番茄青枯病的防效测定

通过形态特征、生理生化特征和16SrDNA序列分析,对分离于番茄茎部能较强抑制番茄青枯病菌生长的内生细菌B47菌株进行了鉴定。结果表明,该菌为枯草芽孢杆菌,其最适生长pH5~6,最适生长温度为35℃。室内防治试验结果表明,用淋根法先接种B47菌后接种病原菌和用注射法先接种B47菌后接种病原菌的处理可取得81.25%和92.10%的防效,而用淋根法、注射法同时接种B47菌与病原菌的处理防效较低。     

聚乙烯醇共混改性膜作为农药种衣剂成膜剂的性能研究

以水溶性聚合物聚乙烯醇(PVA)和水不溶性聚合物乙酸乙烯-乙烯共聚物(VAE)乳液为原料,采用溶液共混法制备了PVA-VAE共混膜,为农药成膜剂的制备开辟了新途径。红外光谱、透射电镜、激光粒度仪测试结果显示,PVA溶液中的胶束被打开,体系的比表面积急剧增大,两相间的界面消失,说明共混体系分子间具有良好的相容性;透水性与耐水性测定结果表明,PVA-VAE的质量比为80∶ 20、70∶ 30、60∶ 40时其共混膜的性能适用于农药种衣剂的成膜剂;种子发芽试验显示,与单独使用PVA膜相比,引入适当比例VAE后的共混膜用于种子包衣,其耐水性显著提高,渗水率适宜,对种子发芽无明显影响。     

乌兰布和沙漠流动沙丘不同部位水分动态研究

通过对乌兰布和沙漠流动沙丘的不同部位沙土含水量的测定研究，结果表明：（1）乌兰布和沙漠流动沙丘3个不同部位的水分垂直变化趋势是一致的，0～60cm含水量变化幅度大，60～100cm变化幅度小，含水量最大值出现在20～60CITI，降雨主要影响60cm以上的沙土水分；（2）3种部位在时间变化上都表现为4～5月含水量最小，从6月开始含水量升高；（3）迎风坡含水量较其他部位大0．65％～0．9％，较背风坡大1．8％～2．5％，其他部位较背风坡大1％～1．7％，且3种地貌部位20～60cm处较80cm和100cm处有较大差异，同时迎风坡的变化幅度最大。     

Effects of vasoconstrictor and endothelium-dependent relaxationagent on thoracic aortic rings of diabetes mice

AIM: To discuss the effects of vasoconstrictor and endothelium-dependent relaxation agent on the thoracic aortic rings in different stages of diabetes mellitus. METHODS: Streptozotocin (STZ, 40 mg/kg) was injected intraperitoneally to establish diabetic model in C57BL/6J mice. At the 17th, 22nd, 28th week, diabetic and age-matched control mice were sacrificed respectively and the effects of vasoconstrictors: phenylephrine (PE), 60 mmol/L KCl and endothelium-dependent relaxation agent respectively (ACh) were measured in two groups using thoracic aortic rings. RESULTS: The level of fasting plasma glucose concentration 2 weeks after STZ treatment increased higher (≥11.1 mol/L) in STZ-induced diabetic mice than that in age-matched controls, and maintained at this level in entire experiment course. On the contrary, the weight was decreased significantly. The responses of thoracic aortic rings in STZ-induced diabetic mice to PE were increased, unaltered and increased at the 17th, 22nd, 28th weeks, respectively compared to that in age-matched controls. The responses to 60 mmol/L KCl were also increased in all stages. While the responses to ACh were increased, unaltered and decreased at the 17th, 22nd, 28th weeks, respectively compared to that in age-matched controls. CONCLUSION: The responses of thoracic aortic rings to vasoconstrictor enhance in STZ-induced diabetic mice. However, the endothelial functions potentiate initially due to compensation, and then lower exhibiting endothelial damage.     

Antisense c-myc sensitizes osteosarcoma cells to cisplatin-induced apoptosis

AIM: To down-regulate expression of c-myc through antisense therapy and to investigate its effect on the sensitivity of osteosarcoma MG-63 cells to cisplatin-induced apoptosis. METHODS: The recombinant adenovirus (Ad-Asc-myc) encoding antisense c-myc fragment was constructed and transfected into osteosarcoma MG-63 cells in vitro in order to down-regulate the expression of c-myc, and the change in the sensitivity to cisplatin-induced apoptosis was observed. MTT, Western blot, RT-PCR, flow cytometry (FCM) and electron microscope were used to evaluate tumor cell proliferation in vitro, genes expression related to apoptosis regulation and effects on the sensitivity of osteosarcoma MG-63 cells to cisplatin-induced apoptosis. RESULTS: Ad-Asc-myc down-regulated the expression of c-myc protein after transfected MG-63 cells for 48 h, combined with the treatment of 2.0 mg/L cisplatin for 2 h inhibited tumor cell proliferation in vitro by 38.0%. RT-PCR revealed that Ad-Asc-myc down-regulated the expression of Bcl-2 and up-regulated the expression of Bax. No appreciable change was observed in the expression of E2F-1. FCM showed that Ad-Asc-myc induced apoptosis in intransfected cells, and rendered it more sensitive to cisplatin. CONCLUSION: Antisense c-myc is able per se to induce apoptosis and sensitize osteosarcoma cells to cisplatin-induced apoptosis.