鸡腺胃炎及其研究进展

鸡腺胃炎是以鸡采食量低、生长不良、消瘦、粪便过料等外观症状,以及腺胃肿大或萎缩、腺胃黏膜溃疡、肌胃角质层溃疡或糜烂为主要特征的流行病,发病鸡群死亡率高达30%～50%,给家禽业带来了巨大威胁。该病病因复杂,尚无定论。为使广大禽类研究者更好地认识该病,本文就该病的研究进展进行综述。     

鸡传染性腺胃炎的流行及病因分析

鸡传染性腺胃炎是一种由多种原因引起的鸡腺胃综合征,主要以鸡的生长缓慢或阻滞、腺胃肿大充血、腺胃乳头出血、溃疡甚至糜烂为特征的传染性疾病。鸡传染性腺胃炎的病原较复杂,可能主要有禽网状内皮组织增殖病病毒、冠状病毒、鸡痘病毒及霉菌毒素等构成。一般认为该病的发生由病毒、霉菌及细菌等多种因素协同作用。由于病因尚无定论,加之该病防制困难,给养禽业造成了重大的经济损失。文章就鸡传染性腺胃炎几个主要的发病原因进行分析和叙述。     

鸡肌腺胃炎的目前状况与对策

<正>从2007年东北地区发生鸡病肌腺胃炎以后,很多专家都在研究该病的病因。众说纷纭。该病不但导致鸡采食量下降,鸡群生长发育不良,甚至导致其他疾病的产生。1发病特点1.1发病品种以前腺胃炎主要发生在青年蛋鸡上(以50～80日龄多发),慢慢地在杂交鸡、肉鸡上     

眼型鸡痘并发鸡腺胃炎的诊治

近段时间,一种以育雏、育成鸡生长不良,消瘦,整齐度低,病程长,死淘率高为主要特征的疾病,经流行病学调查和实验诊断确定是鸡痘并发腺胃炎。本病多发生于7-10月份,秋季是该病的高发时期。发病日龄多集中在30~60日龄,个别鸡群在80日龄以上和20日龄以内发病。该病病因复杂,但近2年秋季腺胃炎高发的主要原因是眼型鸡痘。     

鸡传染性腺胃炎病原学研究进展

鸡传染性腺胃炎是以鸡生长阻滞、腺胃肿大、腺胃乳头出血和溃疡等为主要特征的传染病,多发于雏鸡,以蛋用雏鸡发病较多,近年来,肉鸡发病也有逐渐增多的趋势,给养鸡业带来严重的经济损失。鸡传染性腺胃炎病原复杂,病因众说纷纭,目前尚无定论。为使广大禽病研究者更快速的认识本病,本文就国内外对该病有关的主要病原学的研究进展进行综述。     

鸡传染性腺胃炎病的防控

正鸡传染性腺胃炎是以腺胃肿大为特征的一种慢性消耗性传染疾病,因病因复杂,存在诊断延误和治疗效果不理想等问题。因此,对该病的发病原因、诊断以及防控措施进行规范,做到早诊断、早治疗,才能使该病得到有效地控制。1病因引发鸡传染性腺胃炎的病因较为复杂,概括起来主要有非传染性因素和传染性因素两方面。     

鸡传染性腺胃炎病因研究进展及协同作用分析

自1994年以来,在我国江苏、山东等地先后流行一种以腺胃肿胀、腺胃乳头溃疡为特征的传染病,现已蔓延至全国许多养鸡地区。本病多发生于雏鸡,主要表现生长停滞、羽毛生长不良和消瘦;剖检除较常见的腺胃炎变化外,还可见胸腺、法氏囊萎缩,胰腺坏死。常因继发感染而造成大批死亡。由于病因复杂,故该病有“鸡腺胃型传染性支气管炎”、“鸡传染性腺胃炎”、“鸡病毒性腺胃炎”等多种称呼。     

鸡腺胃炎致病因素研究进展

鸡腺胃炎在我国多个地区流行,给养鸡业造成严重损失。通过阐述鸡腺胃炎的流行范围、特点、临床症状和病理变化,分析了诱发该病的传染性与非传染性两大主要因素,提出了相应的防控思考,有助于增强行业对鸡腺胃炎的了解与重视,为该病的防治提供参考。     

鸡传染性腺胃炎的病因及防治

近年来伴随着我国集约化养殖的快速发展,鸡传染性腺胃炎的发生也呈逐年上升趋势,其临床症状主要表现为鸡的发育不良和生长迟缓,这严重影响着养鸡业的发展。由于该病病因复杂且病原尚未明确,目前尚未有针对该病的特效药,一旦有鸡只染上该病,由于短时间内无法根治,将会在鸡群中迅速传播,这给养鸡场带来极大的经济损失。文章通过对传染性鸡腺胃炎的发病原因、临床症状与治疗措施进行分析,以期为广大养鸡户提供参考。     

以鸡腺胃肿大为主征候群的鉴别诊断和综合防治

<正>临床剖检所见的腺胃肿大只是复杂病原在腺胃部位的一种表现,这种以腺胃肿大为主的、兼有肠道炎症变化、免疫器官损伤等病理变化为主的疾病应称之为鸡的腺胃肿大征候群。该病并不是一种独立的疾病,而是一种多病因的鸡病征候群。     

羔羊腹泻细菌和病毒病原的研究进展

导致羔羊腹泻的因素多样,细菌和病毒感染是最关键的因素,其危害也最严重。已有很多专家、学者应用不同的方法对引起羔羊腹泻的病原开展了大量研究,旨在探明引起羔羊腹泻的主要病原。近年来,对病原的研究主要集中于病原主要编码基因和功能蛋白在致病过程中的作用及其分子流行病学。作者从分子水平将近年来导致羔羊腹泻的主要细菌和病毒病原进行综述,并据此为羔羊腹泻病原学发展提出看法。     

Research Progress on the Bacterial and Viral Etiology of Lamb Diarrhea

The etiology of lamb diarrhea disease is complex,however,bacterial and viral infection are the most critical factors and cause most serious harm.In order to explore the main pathogens of lambdiarrhea,many scholars and experts have undertaken extensive research on different aspects of viral and bacterial etiology of lamb diarrhea with various methods.Recently,the research about the etiology concentrates on the main code genes and functional proteins in the progress of pathogen invading host and molecular epidemiology.This paper reviewed the recent and main pathogens of lamb diarrhea caused by bacteria and viruses from the molecular level,and proposed some ideas about the development of etiology of lamb diarrhea in the future.     

鸭坦布苏病毒病研究进展

鸭坦布苏病毒病(Duck tembusu virus disease)自2010年暴发以来,众多科研工作者对于该病毒的病原学、病理学、致病机理、防治措施等方面进行研究,并取得丰硕的成果。本文针对鸭坦布苏病毒病的病原学、病理学、致病机理、防治措施等方面进行综述。     

Cirrhosis: a consequence of chronic liver disease

The definition, etiology, and pathophysiology of cirrhosis are discussed. The author also discusses therapy for cirrhosis, which is directed at treating the primary etiology as well as controlling the common secondary complications of the disease.     

牛疙瘩皮肤病概述

牛疙瘩皮肤病是20世纪发现的在牛群中传播的一种皮肤传染病,该病1929年最早发现于非洲,后传播到世界其他地区,常带来较大的经济损失.论文综述了牛疙瘩皮肤病病原学、流行病学、诊断和防控技术.     

New insights into degenerative mitral valve disease in dogs.

Degenerative mitral valve disease (DMVD) is the most common cardiac disease in dogs. Although the disease is frequently described in the veterinary literature, many aspects are still unknown or controversial. Based on recent research findings, this article addresses the etiology, pathogenesis, inheritance, diagnosis of early DMVD, diagnosis of mild decompensated heart failure, and efficacy of early medical intervention in clinically compensated dogs.     

浅谈鸡传染性贫血

本文介绍了由鸡传染性贫血病病毒侵害雏鸡骨髓、胸腺和法氏囊导致免疫抑制的一种疾病——鸡传染性贫血病,系统论述了自1979年以来国内外学者对该病病原、流行状况、临床诊断、病原学诊断、防治措施的研究概况.     

An emerging zoonosis in Scandinavia -- nephropathia epidemica.

The paper reviews as zoonosis the disease called Nephropathia epidemica in north European countries. The etiology is as yet unsolved but a viral one is highly suspected with small rodents (voles and mice) as the source of infection. Based on the epidemiological peculiarities of the disease, the ecology of the agent and hypothetical ways of transmission of infection from small rodents to man are discussed.     

Research Progress on Avian Tembusu Virus

Avian Tembusu virus(ATMUV) is a newly viral infectious disease of laying duck.Since April 2010,the disease has caused severe economic loss to the duck industry in China southeast coastal areas.ATMUV can also cause chickens,geese and other poultry disease.The present article summarized research progress on ATMUV regarding etiology,epidemiology,biological characteristics,genomics,methods of virus detection and vaccine developments,and provided references for further research ATMUV.     

Neuroendocrine evaluation of cardiac disease.

Current evidence favors the view that regardless of etiology, there is a predictable sequence of neuroendocrine activation that operates in most dogs and cats with progressive heart disease and that it is largely, but not entirely, independent of etiology. The natriuretic peptides and sympathetic nervous system seem to be early responders to developing cardiac and hemodynamic perturbations in both species. BNP plays a particularly prominent role in cats, possibly as a reflection of disease etiology. Shortly thereafter, plasma endothelin concentrations rise, reflecting the impact of the hemodynamic alterations on the vasculature. Endothelin and the natriuretic peptides directly suppress plasma renin release but have divergent effects on aldosterone. Activation of the tissue RAAS may operate early on to further the progression of heart failure, but evidence of plasma RAAS activation occurs comparatively late and near the time of development of overt CHF. Finally, in animals with severe CHF that are prone to hypotension,vasopressin levels may also rise, contributing to the retention of free water and congestion that is refractory to diuretics. Although oversimplified, this scenario seems to be consistent with data obtained in human, canine, and feline patients. These observations provide some impetus for evaluating ACE inhibitors in cats and beta-receptor-blocking drugs in dogs and cats. Perhaps we are also a little closer to identifying useful biochemical markers that can aid in the diagnosis of heart disease, guide therapy, and improve our understanding of the biologic processes occurring in our patients.