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1.
配料混合试验的设计和分析 总被引:2,自引:0,他引:2
莫惠栋 《扬州大学学报(农业与生命科学版)》2004,25(1):1-4
系统地介绍了配料混合试验的基本概念.指出了混合试验与常规因子试验的区别。提出配料混合试验的3种设计,即顶点和边界点设计、矩心设计和极顶设计。以2个实例演示了混合试验资料的统计方法,包括回归和方差分析.驻点分析和等值线分析。 相似文献
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提出了一种在线测量混炼胶粘度的新方法.该方法以模糊建模技术为基础,综合考虑混炼过程各因素对胶料粘度的影响,建立起胶料粘度的在线测量模型.在模糊建模中,采用T-S模型描述胶料粘度变化的非线性过程,提出了一种基于相似性判别的模糊聚类算法以自动确定合适的聚类组数目,并用实数编码的遗传算法优化全局参数,从而获得了规则简化的、具有较高精度的模糊模型.根据此方法,设计了测量装置,并进行了现场试验.试验结果表明模糊模型输出与实验室测量值基本一致,平均误差较低且最大误差未超过1门尼.该方法较大地提高了橡胶混炼的生产效率,为粘度最优控制奠定了基础. 相似文献
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2种典型固体表面铺展剂SDS和TX100混合水溶液,无论在其表面层或在混合胶团中两组分之间均存在一定程度的相互作用,使得以廉价的SDS为主的溶液中加入少许TX100组成的复配体系表面活性提高,在植物叶面上的润湿性能改善,并从理论上计算了其相互作用参数,初步解释了实验现象,上述结果不仅对指导优良植物叶面铺展剂配方具有指导意义,也预示了潜在的实用价值. 相似文献
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Functional brain alterations induced by lindane were examined in Rat chronically exposed to low doses of lindane (1 and 2 μg L−1 in drinking water) from conception. Histological and electrophysiological investigations were conducted at 14 weeks of age. Lindane did not induce histological changes. It did not significantly modify the sleep-wakefulness cycle and EEG did not show major alterations. The spectral EEG analysis, by recording cortical variations in both energy spectrum and energy levels of different frequency bands, showed an increase in the 11-15 Hz activity in both groups exposed to lindane. This activity was characterised by spindles well-depicted during slow wave sleep or associated with fast activity during wakefulness and theta activity in paradoxical sleep. These data suggest that chronic exposure to lindane doses as low as 1 μg L−1 in drinking water could lead to central electrophysiological effects possibly involving GABA-benzodiazepine mechanisms. 相似文献
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Mario Suwalsky Maritza BenitesBeryl Norris Carlos P Sotomayor 《Pesticide biochemistry and physiology》2003,77(2):44-53
The current study examines the acute effects of the organophosphorus pesticide chlorpyrifos on a sympathetic synapse of the frog Caudiverbera caudiverbera. Nerve stimulation was followed immediately by a transient increase in the short-circuit current (SCC) and in the potential difference (PD), which consisted of a rapid and then a slow component. Chlorpyrifos concentrations from 5 μM to 1.0 mM caused a dose-dependent block of both components to a 10% of their control values, which was reversed by washout. The pesticide blockade did not affect the skin response to noradrenaline. X-ray diffraction and fluorescence spectroscopy studies on membrane models showed marked phospholipid perturbation, which favors changes in ion channel conformation and interferes with receptor proteins, thus altering noradrenergic transmission and Cl− secretion in the mucous glands. The foregoing results may be interpreted as a reversible inhibition of the neuroepithelial synapse to nerve stimulation, possibly due to non-specific lip μd-protein perturbation, interference with synaptic transmission, and transient Cl− channel inactivation. 相似文献
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AIM: To observe the therapeutic effect of Jiedu-Qingfei mixture on Mycoplasma pneumoniae (MP)-infected rat lung tissues and to explore its mechanism. METHODS: SD rats (n=40) were randomly divided into 4 groups:blank control group, model group, Jiedu-Qingfei group and positive control group, with 10 rats in each group. The rats in experimental groups were slowly dripped with 1×109 CFU/L MP solution into their nostrils for 4 d. One rat in each group was sacrificed for MP nucleic acid detection at the second day after inoculation, and the other rats were given gavage therapy. The rats in blank control group and model group were intragastrically given the same volume of normal saline, the rats in Jiedu-Qingfei group were given 8 mL/kg Jiedu-Qingfei mixture daily for 4 weeks, and the rats in psoitive control group were given dexmethasone sodium phosphate (0.5 mg·kg-1·d-1). After the experiment, the rats were killed. The serum and bronchoalveolar lavage fluid (BALF) were collected for detecting the levels of interleukin-12 (IL-12), IL-13 and TNF-α by ELISA. The right lung tissues were used for pathological observation and HE staining, while the left lung tissues were used to detect the expression of NF-κB p50, I-κBα and p38 mitogen-activated protein kinase (p38 MAPK) at mRNA and protein levels. RESULTS: The results of MP nucleic acid detection showed that all the rats except blank control group were MP nucleic acid positive, indicating that the rat model of MP infection was successfully established. On the 1st day of the treatment, the pathological scores of the lung tissues in model group and Jiedu-Qingfei group were significantly higher than those in blank control group (P<0.05). After treatment, the pathological scores of the lung tissues in mo-del group were significantly higher than those in blank control group and Jiedu-Qingfei group. The levels of IL-12 in the serum and BALF in model group were significantly lower than those in blank control group after MP infection (P<0.05), while those after treatment with Jiedu-Qingfei mixture were significantly higher than those in model group (P<0.05). The levels of IL-13 and TNF-α in the serum and BALF of MP-infected rats were increased significantly, while those after treatment with Jiedu-Qingfei mixture were significantly lower than those in model group (P<0.05). The mRNA expression levels of NF-κB p50 and p38 MAPK in model group were increased significantly (P<0.01). After treatment, the mRNA expression levels of NF-κB p50 and p38 MAPK were decreased significantly compared with model group (P<0.01). The mRNA expression level of I-κBα in model group was significantly lower than that in control group. After treatment, the mRNA expression of I-κBα in Jiedu-Qingfei group was significantly higher than that in model group (P<0.05). The protein levels of NF-κB p50 and p38 MAPK in the lung tissues of model group were significantly higher than those of blank control group. After treatment, the protein expression of NF-κB p50 and p38 MAPK was decreased significantly. The protein level of I-κBα in model group was significantly lower than that in blank control group, and after treatment with Jiedu-Qingfei mixture, the protein expression level of I-κBα was increased significantly (P<0.05). CONCLUSION: Jiedu-Qingfei mixture may attenuate lung tissue inflammation caused by MP through NF-κB and p38 MAPK pathways. 相似文献
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