THE YIELD AND QUAEITY OF RED BEET SEED AS AFFECTED BY DESICCANT SPRAYS AND HARVEST DATE

Summary. Diquat and sodium monochloroacetate (SMA) were used to desiccate seed crops of red beet in experiments from 1963 to 1965. The seed plants became brown and dry about 7 days after spraying and were then judged suitable for combine harvesting, although this was not done in these experiments. Yields of seed 7 days after spraying were as good as those from the control plants, but were lower than the control yields when harvested 14 days after spraying. In laboratory tests in moist sand at 20° C the percentage of the embryos which germinated was not markedly affected by diquat or SMA treatment. The emergence of seedlings in the field, however, was affected adversely by treatment of the seed crop with 11 and 22 oz/ac of diquat ion in 1963 and by SMA at 20 lb/ac in 1964 and 1965. Diquat at 6·6 oz/ac in 120 gal/ac water in 1964 and 1965 had no serious adverse effects on embryo emergence. Residues of diquat ion in seed varied from 2 to 4 ppm, but the impairment of embryo emergence by diquat was believed to be due to the premature arrest of growth of the seed on the desiccated plants.
It is concluded that when harvesting conditions are poor, desiccants could be valuable.
Influence des traitements dessicants et de la date de récolte sur le rendement et la qualité de la sentence de betterave rouge     

Epizootiology of Renibacterium salmoninarum, the causal agent of bacterial kidney disease in salmonid fish

Abstract. By use of a selective medium, Renibacterium salmoninarum was recovered from rainbow trout, Salmo gairdneri Richardson. With asymptomatic animals, small numbers of Renibacterium colonies were recovered only from the anterior part of the kidney; whereas in clinically diseased fish, dense pure culture growth was obtained from kidney, spleen, heart, blood, ascitic fluid and faeces. There was no evidence for the presence of R. salmoninarum in the water or sediment of 56 freshwater fish farms in England and Wales. Nevertheless, laboratory–based experiments showed that the pathogen was excreted in the faeces of clinically diseased fish. Moreover, there was survival of the bacterial cells for up to 21 days in sediment/faecal material.     

EFFECTS OF ANESTHETIC PROTOCOL ON NORMAL CANINE BRAIN UPTAKE OF 18F‐FDG ASSESSED BY PET/CT

The purpose of this study was to assess the effects of four anesthetic protocols on normal canine brain uptake of 2‐deoxy‐2‐[¹⁸F]fluoro‐d ‐glucose (FDG) using positron emission tomography/computed tomography (PET/CT). Five clinically normal beagle dogs were anesthetized with (1) propofol/isoflurane, (2) medetomidine/pentobarbital, (3) xylazine/ketamine, and (4) medetomidine/tiletamine–zolazepam in a randomized cross‐over design. The standard uptake value (SUV) of FDG was obtained in the frontal, parietal, temporal and occipital lobes, cerebellum, brainstem and whole brain, and compared within and between anesthetic protocols using the Friedman test with significance set at P<0.05. Significant differences in SUVs were observed in various part of the brain associated with each anesthetic protocol. The SUV for the frontal and occipital lobes was significantly higher than in the brainstem in all dogs. Dogs receiving medetomidine/tiletamine–zolazepam also had significantly higher whole brain SUVs than the propofol/isoflurane group. We concluded that each anesthetic protocol exerted a different regional brain glucose uptake pattern. As a result, when comparing brain glucose uptake using PET/CT, one should consider the effects of anesthetic protocols on different regions of the glucose uptake in the dog's brain.