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1.
Carlos Garrido María Carbú Francisco Javier Fernández-Acero Giles Budge Inmaculada Vallejo Alison Colyer Jesús M. Cantoral 《European journal of plant pathology / European Foundation for Plant Pathology》2008,120(4):409-415
Anthracnose, caused by Colletotrichum spp., is a major disease of cultivated strawberry, Fragaria × ananassa. This study identifies the Colletotrichum spp. which causes strawberry anthracnose in the southwest of Spain. A survey of the region was carried out, and the strains
isolated were identified as C. acutatum by using the polymerase chain reaction (PCR) with genus and species-specific primers, demonstrating that this species is
currently the causal agent of strawberry anthracnose in the studied region. The pathogenicity of C. acutatum and C. gloeosporioides strains was evaluated on two principal strawberry cultivars (cvs Camarosa and Ventana) under field conditions, the latter
being more pathogenic than the former. 相似文献
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This study determined the uptake of three model compounds, applied in the presence and absence of surfactants, into the leaves of three plant species (Chenopodium album L, Hedera helix L and Stephanotis floribunda Brongn). The results with 2-deoxy-D-glucose, 2,4-dichlorophenoxyacetic acid and epoxiconazole in the presence ofsurfactants (the polyethylene glycol monododecyl ethers C12EO3, C12EO6, C12EO10 and a trisiloxane ethoxylate with mean EO of 7.5, all used at one equimolar concentration and therefore different percentage concentrations) illustrate that the initial dose (nmol mm(-2)) of xenobiotic applied to plant foliage is a strong positive determinant of uptake. This held true for all the xenobiotics studied over a wide concentration range in the presence of these surfactants. Uptake on a unit area basis (nmol mm(-2)) could be related to the initial dose of xenobiotic applied per unit area (ID) by an equation of the form: Uptake = a [ID]b at time t = 24h. ID is given by the mass of xenobiotic applied, M divided by the droplet spread area, A. Total mass uptake is then calculated from an equation of the form: Total Uptake = a [ID]b x A. 相似文献
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Susceptibility to equine hyperlipaemia is increased by poor food intake. To assess the contribution of changes in insulin sensitivity, plasma glucose and cortisol responses to an intravenous insulin challenge (0·4
kg−1 bodyweight) were compared with those observed after saline administration in six donkeys fasted either overnight or for three days. Three days of fasting decreased both the rate of insulin-induced hypoglycemia and the maximal hypoglycemic response. A transitory increase in plasma cortisol which peaked within one to four hours of insulin administration was observed in three of the six overnight-fasted donkeys and in all of the three-day fasted donkeys; inter-animal variation appeared to exist in the responsiveness of the hypothalamic-pituitary-adrenocortical (
) axis to stimulation by insulin-induced hypoglycemia. Fasting is likely to present a risk of equine hyperlipaemia, at least in part, by the reduction in tissue sensitivity to the glucoregulatory action of insulin. 相似文献
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Abigail L. Fowden Juanita K. JellymanOrlando A. Valenzuela DVM MSc Alison J. Forhead PhD 《Journal of Equine Veterinary Science》2013
Human epidemiological observations and studies of experimental animals have shown that low birth weight is associated with adult phenotypes characterized by abnormalities in cardiovascular, metabolic, and endocrine function. In human populations, these changes can lead to overt degenerative diseases such as hypertension and type 2 diabetes. Because fetal growth depends primarily on the nutrient supply, the associations between birth weight and adult phenotype have been linked to poor nutrition in utero. The fetal supply of nutrients and oxygen depends on both the availability of these substances in the mother and the functional capacity of the placenta to supply them to the fetus. This review takes a comparative approach to examining the nutritional programming of adult physiological phenotype in mammals with emphasis on the horse where possible. It considers the role of maternal nutritional state and placental function in these processes and discusses the cellular and molecular mechanisms operating in utero that are responsible for the epigenetic regulation of phenotypical diversity. 相似文献