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Adhesion problems sometimes occur during the production of laminated wood products. To minimize such quality problems, there
is a need for a nondestructive test that can provide continuous control of the process and the product. This study presents
results from measurements performed to evaluate the potential of pulse thermography as a method to detect glue deficiency
in laminated wood. Defect depth, defect size, and degree of glue deficiency have been varied. The surface layer was made of
merbau (Intsia bijuga) and the substrate of Scots pine (Pinus silvestris). The results showed that pulse thermography is a promising tool for detecting glue deficiency underneath the thin laminated
wood surface layers, mainly because of the short inspection time. Lack of glue with a minimum thermal defect size of 3 was
detectable (thermal defect size is defined as the quotient of defect size and defect depth). The penetration depth was 1.0 mm
and the highest contrast, 0.62°C, was achieved for one of the largest defects (24 mm) below the thinnest (0.5 mm) surface
layer after 1 second. Starved glue joints showed about half the contrast compared to areas with total lack of glue.
Received: April 24, 2002 / Accepted: July 26, 2002
Acknowledgments We gratefully acknowledge the support of this work from the Knowledge Foundation and The Swedish Wood Association. 相似文献
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以砂光处理、等离子体处理、羟甲基间苯二酚(HMR)处理为不同因素,研究了纤维增强树脂复合材料(FRP)/竹、FRP/木胶合界面处理工艺。结果表明胶合界面优化工艺为:FRP不做处理,而竹材和木材表面以150 g/m~2的涂布量涂布HMR。 相似文献
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WEN Hao-min LI Yan WANG Guang ZHANG Xiao-tan WANG Xiao-yu ZHANG Zhao-long YANG Xue-song 《园艺学报》2012,28(7):1269-1274
AIM: To investigate the role of N-cadherin in the delamination of neural crest cells. METHODS: The normal expression of N-cadherin in neural tube was identified using in situ hybridization. The cells with N-cadherin over expression were obtained by transfection of wild-type N-cadherin (wt-N-cadherin),and the cells with N-cadherin silencing expression were obtained by transfection of dominant-negative N-cadherin (dn-N-cadherin). The migration of cranial neural crest cells was determined by the technique of immunohistochemistry. RESULTS: Either overexpression or down-regulation of N-cadherin significantly affected the migration of cranial neural crest cells. CONCLUSION: Delamination and migration of the cranial neural crest cells rely on the relative N-cadherin expression in the neural tube during neurulation. 相似文献
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