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11.
This report describes the use of 99m technetium pertechnetate (99m TcO4 − ) and 131 [for imaging of a metastatic thyroid carcinoma in a dog. The 131 ] imaging showed metastatic lesions undetected by the 99m TcO4 − imaging on 2 separate occasions. The possible mechanisms for the discrepancies between 131 I and 99m TcO4 − imaging of thyroid carcinomas are discussed. The use of 131 I for the imaging of functional thyroid carcinomas in the dog is recommended. 相似文献
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Christopher L. Mariani DVM Simon R. Platt BVMS MRCVS Susan M. Newell DVM MS Scott P. Terrell DVM Cheryl L. Chrisman DVM MS EDS Roger M. Clemmons DVM PhD 《Veterinary radiology & ultrasound》2001,42(6):524-531
A 3-year-old neutered female mixed breed dog was examined because of severe, generalized seizure activity, tetraparesis, and encephalopathic signs. Cerebrospinal fluid (CSF) evaluation was unremarkable except for a mild increase in protein. Serum and CSF titers for infectious diseases were negative. Magnetic resonance (MR) imaging examination of the brain was performed and lesions were found within the cerebral gray matter of the temporal and parietal lobes. The lesions had increased signal intensity on T1, T2, and proton density-weighted images. There was mild inhomogeneous enhancement following intravenous contrast medium administration. Neurologic status improved and the seizures were well controlled, but the dog never regained normal mentation and euthanasia was performed 10 weeks after initial evaluation. At necropsy, severe cerebral cortical necrosis was found in the regions corresponding to the lesions seen on MR imaging examination. Large numbers of fat-containing macrophages (gitter cells) were found within these areas, and are thought to be responsible for the characteristic hyperintensity seen on the MR images. 相似文献
15.
YOSHIAKI HIKASA DVM PhD MASAAKI KUBOTA DVM MS KATSUAKI TAKASE dvm PhD TOMOKO KAKUTA mt SHIGEO OGASAWARA dvm PhD 《Veterinary surgery : VS》1993,22(4):311-317
The effects of thiopental, ketamine, diazepam, xylazine, and nitrous oxide, and the combinations of thiopental-nitrous oxide and ketamine-nitrous oxide, on both enflurane-induced electroencephalographic (EEG) spike activity and convulsive behavior were measured quantitatively in atropinized cats receiving enflurane with controlled ventilation. Pretreatments with thiopental, ketamine, and diazepam reduced both EEG spike frequency and amplitude at 2.5% to 4.5% inspired enflurane but did not abolish spike activity. Nitrous oxide (66% of inspired gas) did not significantly alter spike frequency or amplitude during 2.5% to 4.5% inspired enflurane, but the combination of thiopental-nitrous oxide or ketamine-nitrous oxide reduced EEG spike activity during 2.5% inspired enflurane. Enflurane-induced convulsive score was markedly suppressed by thiopental and ketamine and was significantly reduced by diazepam, xylazine and nitrous oxide. The combinations of thiopental-nitrous oxide and ketamine-nitrous oxide greatly reduced behavioral-convulsive responses induced by 2.5% to 4.5% inspired enflurane. 相似文献
16.
Hepatic Lobe Torsion as a Cause of Colic in a Horse 总被引:2,自引:0,他引:2
TRACY A. TURNER DVM MS Diplomate ACVS CYNTHIA A. BROWN dvm JULIA H. WILSON DVM Diplomate ACVIM LOIS ROTH DVM Diplomate ACVP DIANA S. ROSENSTEIN DVM CHRISTINE M. SCHWEIZER dvm KRISTINE A. WOODAMAN dvm 《Veterinary surgery : VS》1993,22(4):301-304
A 14-year-old Arabian gelding was examined for colic. An exploratory celiotomy was subsequently performed and the left lobe of the liver was found to be twisted. The lobe was resected using a TA-90 surgical stapling instrument. Histologic examination of the resected liver indicated portal vein and sinusoid dilation and congestion with blood. There were focal areas of necrosis and bacterial cocci and rods throughout the section. The histologic findings were consistent with hepatic lobe torsion. After surgery, the horse was treated with broad spectrum antibiotics, anti-inflammatory drugs, heparin, and intravenous fluids. The horse recovered without complications, although serum liver enzymes remained elevated for more than 1 week after surgery. Seven months after surgery the horse showed no adverse affects from the disease. 相似文献
17.
Deborah R. Van Pelt DVM MS Wayne E. Wingfield MS DVM Timothy B. Hackett DVM Linda G Martin DVM 《Journal of Veterinary Emergency and Critical Care》1993,3(2):63-70
Over the past several decades, recognition of acute respiratory failure as the cause of death in patients suffering from various clinical conditions has prompted aggressiv investigation into the area of respiratory physiology and supportive respiratory care. With the evolution of emergency medicine and critical care services in both human and veterinary medicine, many patients previously considered unsalvageable due to the severity of their underlying disease are now being resuscitated and successfully supported, creating a new population of critically ill patients. Where only a decade ago these patients would have succumbed to their underlying disease, they now survive long enough to manifest the complications of shock and tissue injury in the form of acute respiratory failure. Investigation into the pathophysiology and treatment of this acute respiratory distress syndrom (ARDS) has facilitated increased clinical application of respiratory theerapy and machanical ventilation.1 The purpose of this paper is to provide a basic review of respiratory mechanics and the pathophysiology of hypoxemia as they relate to airway pressure therapy in veterinary patients and to review the use of airway pressure therapy in veterinary patients This paper is divided into two parts; part I reviews respiratory mechanics and hypoxemia as they apply to respiratory therapy, while part II deals specifically with airway pressure therapy andits use in clinical cases. 相似文献
18.
A Weber 《Tier?rztliche Praxis》1991,19(5):469-473
The significance of sheep and goats in this country in connection with the zoonoses rabies, tick-borne encephalitis, contagious ecthyma, Q-fever, chlamydiosis, brucellosis, campylobacteriosis, echinococcosis and toxoplasmosis is discussed. 相似文献
19.
Application of Airway Pressure Therapy in Veterinary Critical Care: Part II: Airway Pressure Therapy
Deborah R. Van Pelt DVM MS Wayne E. Wingfield MS DVM Timothy B. Hackett DVM Linda G. Martin DVM 《Journal of Veterinary Emergency and Critical Care》1993,3(2):71-81
As the specialties of emergency medicine and critical care have grown and evolved in both human and veterinary medicine, so has the need for more advanced care of patients with primary lung disease. Treatment of acute respiratory failure has been the focus of several articles in the human medical literature of the past few years.1,8 This paper deals with airway pressure therapy and its application in cases of acute respiratory failure in veterinary medicine. The reader is referred to part I of this paper for a reveiw of respiratory mechanics and hypoxemia as they apply to respiratory therapy. 相似文献
20.
Laurence O. Whiteley DVM PhD Samuel K. Maheswaran BVSc PhD Douglas J. Weiss DVM PhD Trevor R. Ames DVM MS Mathur S. Kannan BVSc PhD 《Journal of veterinary internal medicine / American College of Veterinary Internal Medicine》1992,6(1):11-22
The severe fibrinonecrotic pneumonia associated with pneumonic pasteurellosis usually results from colonization of the lower respiratory tract by Pasteurella haemolytica biotype A, serotype 1(A1). Despite recent research efforts, the authors lack a detailed understanding of the interactions and host response to P. haemolytica in the respiratory tract. The authors hypothesize that management and environmental stress factors or viral infection alters the upper respiratory tract (URT) epithelium allowing P. haemolytica to colonize the epithelium. Once the URT is colonized, large numbers of organisms enter the lung where they interact with alveolar macrophages. Endotoxin, released from the bacteria, crosses the alveolar wall where it activates pulmonary intravascular macrophages, endothelium, neutrophils, lymphocytes, platelets, complement, and Hageman factor leading to complex interactions of cells and mediators. It is the progression of this inflammatory response with neutrophil influx that is ultimately responsible for the pulmonary injury. Leukotoxin is a major virulence factor of P. haemolytica that allows it to survive by destroying phagocytic cells. At subcytolytic concentrations it may also enhance the inflammatory response by activating cells to produce mediators and release reactive oxygen metabolites and proteases. 相似文献