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排序方式: 共有101条查询结果,搜索用时 15 毫秒
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H. G. Zuckerman C. H. Metzger B. A. Brown W. B. Ward C. L. Fitch John S. Gardner Frank W. Hussey Ralph W. Donaldson H. C. Moore H. O. Werner W. H. Martin K. H. Fernow E. V. Hardenburg W. G. Been E. B. Tussing J. B. R. Dickey K. W. Lauer T. E. Odland H. L. Bailey Chas D. Gaines J. G. Milward John Tucker 《American Journal of Potato Research》1935,12(8):222-234
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L.W.N. Fitch B.V.Sc. 《New Zealand veterinary journal》2013,61(4):118-127
Extract This disease was first observed in sheep on a property near Bulls, and has since been found on five other properties in the vicinity. It is an insidious paradontal disease, characterized by a chronic inflammatory process in the periodontal membrane of one or more teeth, resulting in looseness of teeth and in unwillingness to graze and consequent starvation. 相似文献
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Fourth branchial arch defects (4‐BAD) is a syndrome that consists of aplasia, or varying degrees of hypoplasia, of one or more of the cartilaginous or muscular structures derived from the fourth branchial arch, unilaterally or bilaterally. This case report describes the cases of 2 directly related siblings admitted with a complaint of abnormal respiratory noise at rest and complete exercise intolerance. Laryngeal palpation, resting upper airway (UA) endoscopy, lateral radiography of the larynx and cervical trachea, and laryngeal ultrasonography revealed multiple laryngeal and pharyngeal abnormalities. The clinical findings were compatible with the diagnosis of fourth branchial arch defects. A left partial arytenoidectomy and a unilateral ventriculocordectomy were performed in both cases. The follow‐up endoscopies revealed a functional airway aperture. The surgery enabled the horses to be used for riding. Considering the direct family relationship between the horses, these cases would support a genetic component of 4‐BAD. 相似文献
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Lanza RP Arrow KJ Axelrod J Baltimore D Benacerraf B Bloch KE Bloembergen N Brown HC Brown MS Cibelli JB Cohen S Cooper LN Corey EJ Dulbecco R Fischer EH Fitch VL Friedmen M Friedman M Furchgott RF Gell-Mann M Glaser DA Glashow SL Gilbert W Goldstein JL Wilson RW 《Science (New York, N.Y.)》1999,283(5409):1849-1850
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Brooks BA Foster J Sandwell D Wolfe CJ Okubo P Poland M Myer D 《Science (New York, N.Y.)》2008,321(5893):1177
We demonstrate that a recent dike intrusion probably triggered a slow fault-slip event (SSE) on Kilauea volcano's mobile south flank. Our analysis combined models of Advanced Land Observing Satellite interferometric dike-intrusion displacement maps with continuous Global Positioning System (GPS) displacement vectors to show that deformation nearly identical to four previous SSEs at Kilauea occurred at far-field sites shortly after the intrusion. We model stress changes because of both secular deformation and the intrusion and find that both would increase the Coulomb failure stress on possible SSE slip surfaces by roughly the same amount. These results, in concert with the observation that none of the previous SSEs at Kilauea was directly preceded by intrusions but rather occurred during times of normal background deformation, suggest that both extrinsic (intrusion-triggering) and intrinsic (secular fault creep) fault processes can lead to SSEs. 相似文献