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1. Observations were made on the use of food pans and food trays by commercially grown broiler chickens.

2. Birds fed by going inside the feeders on top of the food or from standing around the outside of the feeders. The feeding times of birds inside feeders were significantly longer than those of birds outside. Different reasons to account for this are proposed for the 2 types of feeder.

3. The total number of birds able to feed together from a tray or pan changed as the birds grew. With trays this change in the number of birds was a steady decline. With pans the change was non‐linear and reflected a changing ratio of birds eating in to birds eating out. This ratio was influenced by pan design features and by competition for feeding space.

4. The data suggest that a linear measure of feeding space, as used in research trials and Codes of Practice for Welfare, does not adequately describe birds’ access to and use of feeding space. Implications for both research and the commercial industry are discussed.  相似文献   

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The detection of Mycobacterium paratuberculosis organisms in bovine faeces by isolation was compared with that by the microscopical examination of Ziehl-Neelsen stained faecal smears for the presence of clumps of acid-fast M. paratuberculosis organisms. Faeces were obtained from cattle naturally or experimentally infected with M. paratuberculosis as well as from uninfected cattle. Microscopical examination was an unreliable method for the detection of M. paratuberculosis organisms, since the organisms were only detected in 99 (=55.9%) of 177 culturally positive faecal samples. 1111 addition, clumps of acid-fast organisms indistinguishable from M. paratuberculosis were also observed iin three of 18 samples from cattle free from Johne's disease and in 18 of 37 culturally negative samples from paratuberculous cattle. When M. paratuberculosis organisms were added to faeces from an uninfected cow, results showed that isolation attempts should be positive when 15 or more M. paratuberculosis organisms per gram of faeces are present.  相似文献   
958.
Corneal sequestrum was diagnosed upon clinical examination of 12 cats. The cornea of seven were examined histologically and had focal necrosis and inflammation of the stroma and epithelium. As the simple excision of the corneal lesions had been unsuccessful in two of the cats, repair of these was effected by construction of a tarso-conjunctival pedicle flap after the excision of the sequestrum.  相似文献   
959.
Equine polymorphonuclear (PMN) and mononuclear (MN) leucocytes were separated on Percoll gradients and used to study the chemoattractant properties of the polar ether-linked phospholipid, platelet activating factor (PAF). Six concentrations of PAF ranging from 1 ng/ml to 100 g/ml were studied in each of two in vitro assay systems, the agarose microdroplet and a microfilter technique. Very significant (p<0.01) increases in the movement of both PMN and MN cells were obtained with most concentrations of PAF. In two instances there was no apparent concentration-response relationship, although the action of PAF was approximately bell-shaped in two others. The possible significance of these findings for equine inflammatory conditions is discussed.  相似文献   
960.
In conscious pigs the influence of intravenous infusion of live E. coli (7×108/kg), of the equivalent amount of endotoxin (20 g/kg) or of a high dose of endotoxin (2.5 mg/kg) on the hemodynamic, clinical and pathological parameters and on survival rate was studied. E. coli and endotoxin infusion resulted in pulmonary hypertension, systemic arterial hypotension, a decrease in cardiac output and an increase in heart rate. Clinical signs were characterized by respiratory and nervous disturbances, whereas necropsy revealed hemorrhages and edema in several organs. Although these findings were similar in the three groups, a marked difference in lethality was observed. Infusion of E. coli or of the high dose of endotoxin resulted in a significant mortality, whereas all pigs survived the infusion of the low dose of endotoxin. This suggests that the lethal pathophysiological mechanisms may only become activated when a sufficient amount of endotoxin is released into the circulation.  相似文献   
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