术中穿刺抽脓法预防急性化脓性阑尾炎切口感染

目的：减少急性化脓性阑尾炎手术切口感染机会。方法：对21例急性化脓性阑尾炎开腹显露阑尾后，先穿刺抽脓。防止术中阑尾穿孔污染切口．再切除阑尾。结果：21例病人手术切口全部甲级愈口。结论：急性化脓性阑尾炎术中阑尾穿孔会污染切口．术中穿刺抽脓法可消除这一潜在的污染源。减少切口感染的机会。     

吻合器痔上粘膜环切术治疗重度痔病的临床研究

目的：探讨吻合器痔上粘膜环切术(PPH)治疗重度痔病的临床价值。方法：总结PPH治疗Ⅰ～Ⅳ度痔42例的临床资料。结果：手术时间10～20min，术后平均住院2～5d，术后需止痛剂6例，术后恢复正常生活的平均时问5．5d。术后4周随访，满意36例(85．7％)．基本满意6例(14．3％)，无严重并发症。结论：PPH具有安全、有效、创伤小、术后疼痛轻、并发症少、恢复快、对肛管直肠功能影响小等优点，但长期效果有待进一步观察。     

ASD Field Spec3野外便携式高光谱仪诊断冬小麦氮营养

氮素营养诊断关键在于氮营养指数(nitrogen nutrient index,NNI)预测。对于冬小麦氮营养指数预测模型而言,如何选取预处理方法和建模方法不一而足,不同预处理和模型选取对预测结果精度的影响程度目前还不清楚。该研究以ASD Field Spec3野外便携式高光谱仪采集乐陵市冬小麦冠层高光谱数据,采用10种光谱预处理方法并结合3种模型(偏最小二乘回归、BP神经网络和随机森林算法)建立多种冬小麦氮营养指数高光谱预测模型。对比模型预测精度表明最佳的高光谱建模方法为随机森林算法结合SG卷积平滑预处理所建模型(预测集R2=0.795,RMSE=0.125,RE=11.7%)精度高、可靠性强,是筛选出最佳的冬小麦氮营养指数高光谱预测模型。该研究结果对冬小麦氮营养指数高光谱预测建模具有科学价值,为筛选最优高光谱预处理方法和预测模型提供技术参考。     

Identification of Fusarium wilt resistance gene SiRLK1 in Sesamum indicum L.

Sesame Fusarium wilt(SFW), caused by Fusarium oxysporum f. sp. sesami(Fos), is one of the most devastating diseases affecting sesame cultivation. Deciphering the genetic control of SFW resistance is pivotal for effective disease management in sesame. An inheritance study on a cross between the highly resistant variety Yuzhi 11 and the highly susceptible accession Sp1 using a Fos pathogenicity group 1 isolate indicated that resistance was conferred by a single dominant allele. The target locus wa...     

离子注入浮床植物去除水富营养化研究

在农业和工业废水中,氮（N）和磷（P）是形成富营养化的主要原因,它们在大多数的淡水生态系统中都被认为是限制生物生存的因素,因此也是修复淡水生态系统时需要控制的目标。运用水生植物栽培去除湖泊、河流、水库、池塘和沼泽等富营养化水体中氮和磷的方法已被证明是可取的。已有研究表明,收割吸收了N和P 的水生植物能有效去除N、P污染物,能够有效控制淡水水体富营养化,并且收获的水生植物可用来提高经济效益和产生有价值的副产品,如沼气、生物肥料、生物材料或动物食物。     

MicroRNA regulates the toxicological mechanism of four mycotoxins in vivo and in vitroOA

Mycotoxins can cause body poisoning and induce carcinogenesis, often with a high mortality rate. Therefore,it is of great significance to seek new targets that indicate mycotoxin activity and to diagnose and intervene in mycotoxin-induced diseases in their early stages. MicroRNAs(miRNAs) are physiological regulators whose dysregulation is closely related to the development of diseases. They are thus important markers for the occurrence and development of diseases. In this review, consideration i...     

校企协同育人模式在牧医专业职业规划中的探索

自改革开放以来,我国经济实力、竞争力显著增强,同时职业教育不断发展壮大,但畜牧兽医高职教育在职业生涯教育培养体系还不够完善.在日益严峻的就业形势下,只有不断深化职业教学改革,提高学生创新创业能力,提升就业竞争力,构建校企协同育人的高职学生职业生涯规划教育的一种新模式.     

Roles of an extracellular matrix (ECM) receptor and ECM processing enzymes in demyelinating canine distemper encephalitis

Canine distemper virus (CDV) belongs to the genus Morbillivirus of the Paramyxoviridae family. Due to the central nervous system (CNS) tropism of the virus and associated neuropathological changes, demyelinating canine distemper encephalitis (CDE) represents a relevant model for human demyelinating diseases like multiple sclerosis. The present review decribes the role of CD44 antigen (CD44), the principle cell surface receptor for hyaluronate and extracellular matrix (ECM) processing enzymes (matrix metalloproteinases [MMPs]) and their inhibitors (TIMPs) in the pathogenesis of demyelination. In acute and subacute CDE, a plaque-associated CD44 up-regulation is found that parallels astrocyte activation. Likewise, MMPs and TIMPs are prominently up-regulated in these lesions and are expressed mostly by astrocytes and microglia. In chronic lesions, CD44 expression declines together with the number of glial fibrillary acidic protein (GFAP) positive astrocytes. In addition, in this plaque type, CD44 is expressed on the cell membrane of perivascular mononuclear cells. In this phase, a decrease of MMP and TIMP expressions apart from MMP-11, -12, and -13 is obvious. In summary, CD44 and MMPs might be associated with the onset of demyelination and may interact to initiate ECM disturbances. Ligation of CD44 in the early phase may induce chemokines and cytokines and hence initiate and perpetuate the inflammatory process. In the chronic phase, it is conceivable that a MMP-TIMP imbalance may be the motor for lesion progression with a simultaneous influx of CD44-positive activated immune cells.