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151.
152.
K-S. Dai C-S. Liang Y. T. Ch'iu P-C. Yang I. C. Cheng 《Veterinary research communications》1995,19(2):115-125
The purpose of this study was to determine whether myocardial adenosine triphosphatase (ATPase) activities were reduced in pigs with naturally occurring hypertrophic cardiomyopathy (HCM). The selection of hearts for the HCM and the normal control groups depended on histological examination. Specific ATPase activity and 5-nucleotidase activity were measured in left ventricular myocardium obtained from HCM (n=7) and normal control (n=7) animals. The histological features of HCM included marked disorientation of muscle cells, thickening of the intramural coronary arterial wall with a narrowed lumen, endocardial fibrosis and myocardial fibrosis. The HCM group showed significant increases in both heart weight (32%) and heart weight to body weight ratio (46%). The total ATPase activity in crude homogenates from the HCM group was significantly decreased by 16%. Azide-sensitive ATPase (mitochondrial ATPase) activity, ouabain-sensitive ATPase (Na+,K+-ATPase) activity, basal Mg2+-ATPase activity and Ca2+-ATPase activity were all significantly decreased by 18%, 30%, 20% and 50%, respectively. In contrast, no significant decrease was found in the mean values for 5-nucleotidase activity. These results suggest that myocardial ATPase activities are suppressed in pigs with naturally occurring HCMAbbreviations ATP
adenosine triphosphate
- gww
grams wet weight
- HCM
hypertrophic cardiomyopathy
- Pi
inorganic phosphate 相似文献
153.
1976—1977年河北晋县贺家寨大队小麦春季灰飞虱的传带病毒率为31—52%,不同地块同代灰飞虱的带毒率异差较大:而秋苗期则为12—35.4%,其中不同地块上同代(四代成虫)灰飞虱的带毒率的差异从33.3%至35.4%则不太大。灰飞虱的有效传毒指数(即虫口数×自然带毒率)同田间小麦发病率的相关性是非常显著的,其r=0.9769。1975—1976两年调查及计算分析结果,其直线回归公式如y=4.12x-2.577。在有效传毒指数0.7—20.9之间用于予测发病率有效。人工分期侵染试验证明小麦在生长发育过程中受侵愈早,病情愈重,其中死株率也愈大。秋季侵染的死株率达95.6%,病指为99.6;夏季侵染的死株率为0,病指为57.6。秋季小麦出苗前后的大量虫口形成小麦10月中下旬的一个发病高峰,第二年春季4月中旬的虫口高峰形成5月上旬的一个发病高峰。3月下旬的一个发病高峰是越冬前受侵的。病毒在小麦植株中的潜育期的长短随小麦的生长速度而异。温度不过其中因素之一,最短5—8天,最长30—40天。侵染后如小麦进入越冬期,即不表现症状,一直要到返青以后。介体灰飞虱在河北晋县以第五代3—4龄若虫在冬麦地及草荒中越冬。夏季在玉米上可以产卵并孵化,但不能长期存活,主要在地头沟边及夏作物荫蔽下的禾草上越夏。冬麦治虫防病的重点应在秋季麦苗出土前,春季则应在3月中旬左右。冬麦适当推迟播种有利于减少发病率。棉间作麦比平作的病情重六倍而粮麦间作的病情比平作的重九倍。 相似文献
154.
155.
草地是发展畜牧业的重要基础,退牧还草工程是恢复、更新天然草地和保护生态环境的重要手段。本文就新疆实施退牧还草工程中所出现的一些问题进行了探讨。 相似文献
156.
Wen-Quan Zhen Da-Wei Huang Jin-Hua Xiao Da-Rong Yang Chao-Dong Zhu Hui Xiao 《Phytoparasitica》2005,33(2):113-120
We investigated oviposition behavior in three non-pollinating fig wasps: the sympatric speciesApocrypta bakeri Joseph onFicus hispida L.,A. westwoodi Grandi onF. racemosa L., andApocrypta sp. onF. semicordata Buch.-Ham. The oviposition behavior differs significantly between one pair of species (A. bakeri andA. westwoodi) and the other species (Apocrypta sp. onF. semicordata).A. bakeri andA. westwoodi were similar in the following aspects: the posture of the abdomen and the action of the hind legs before penetration, and
the bending ovipositor sheath during penetration. In contrast, the oviposition behavior ofApocrypta sp. is quite different. This difference can be explained by the significant correlation between ovipositor length and syconial
thickness.Apocrypta sp. has a shorter ovipositor than the two other species, which correlates with the thinner syconial wall of its host figF. semicordata. It is deduced that the ovipositor length adapts to the syconial thickness and induces the oviposition behavior in the different
species to diverge. For all threeApocrypta species, the midleg length and hindleg length are significantly correlated with their ovipositor lengths. This may be explained
as due to the fact that body movement adjusting the hindlegs and midlegs up and down, or forward and backward, is also influenced
by the ovipositor length. 相似文献
157.
AIM: To investigate the contribution of angiotensin-converting enzyme inhibitor (ACEI) to the regulation of calpain system in infarcted myocardium. METHODS: Rat myocardial infarction (MI) model was established by permanent ligation of the left coronary artery. The treatment with the ACEI inhibitor rampril (1 mg·kg-1·d-1) was started 7 days prior to surgery. On day 1, 3, 7 and 14 after MI, protein levels of calpainⅠ, Ⅱ and calpastatin were determined in left ventricular free wall (LVFW), interventricular septum (IS) and right ventricule. RESULTS: CalpainⅠprotein level was increased in IS 14 d post MI, whereas the protein level of calpainⅡ was maximally increased in LVFW 3 d post MI. Rampril decreased protein up-regulation of calpainⅠ and Ⅱ, and reduced infarct size and interstitial fibrosis. Calpastatin protein expression was not affected by ACEI. CONCLUSIONS: CalpainⅠ is involved in cardiac remodelling in the late and calpainⅡ contributes to cardiac tissue damage in the early phase of MI. The heart protective effect of ACEI may be related to the inhibition of calpain system in the pathogenesis of myocardial infarction. 相似文献
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