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991.
提高森林质量是江西省林业建设攻坚战 总被引:1,自引:1,他引:0
江西省林业提出“一个提高,两个调整,三个搞活”的施政方针,是三句话,六个字(提高、调整、搞活)组成的相互联系的一个整体,江西省林业建设现实中质量不高,结构不优、林业不活是个老大难问题,并集中表现在质量上,是计划经济与传统林业留下的后遗症.所以提高森林质量是林业建设攻坚战,是发展江西林业生产力的壮举,认真贯彻科教兴国发展战略,加大林业科教技投入,提高人材素质,选贤用能,组建一支精兵强将,夺取这一战役的辉煌胜利. 相似文献
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针对汽车产品投资的特殊不确定性和其投资数额大、分阶段投入的状况,应用产品生命周期理论判定汽车产品项目的投资阶段,进而获得准确的阶段投资决策点,据此构建了基于汽车产品生命周期的汽车产品投资阶段判定和汽车企业投资决策点识别模型,并就影响识别的因素、阶段、模型进行数学表述,就汽车产品投资决策点抉择问题提出了解决的思路和方法. 相似文献
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ZHANG Jing ZHOU Xu SHEN Jinglin DING Honghao ZHAO Yun TANG Hongyu SHI Xudong FANG Hengtong YAN Hailiang 《东北农业大学学报(英文版)》2008,15(2):33-36
A 45-day growth trial was conducted to investigate the effects of 4 concentrations (0, 40, 80, 120 mg·kg^-1 Zinc- Methionine (Zn-Met) supplementation on growth and serum biochemical indexes of Jirong rabbits (60-day-old). A total of 72 Jirong rabbits were randomly divided into 4 groups, 3 replicates with 6 rabbits each. The Met value in the basal diet was 0.60%. ADGs of group Ⅱ, Ⅲ and Ⅳ were significantly different than that of control, increased by 3.16%, 7.53%, 8.14% during 0-15 days and 7.58%, 15.60%, 15.71% during 16-30 days and 8.27%, 12.43%, 11.44% during 31-45 days (P〈0.05), but it was not significantly different in ADG among group Ⅱ, Ⅲ and Ⅳ (P〉0.05). The fur area and the density of hair of groups Ⅱ, Ⅲ and Ⅳ were not significantly different than that of control (P〉0.05). The content of serum zinc, AKP, ALB for groups Ⅱ, Ⅲ and Ⅳ were significantly different than that of control (P〈0.05). The serum TP and SUN in group Ⅱ, Ⅲ and IV were not significantly different than that of control (P〉0.05). The results show that dietary supplementation of Zn-Met is much more efficient than dietary without Zn-Met for 60-day-old Jirong rabbits. 相似文献
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Ding-biao LONG Ke-ying ZHANG Dai-wen CHEN Xue-mei DING Bing YU 《Animal Science Journal》2009,80(5):585-590
The study was conducted to investigate the effects of active immunization against myostatin on the titer of myostatin antibody, carcass evaluation, activity of creatine kinase and the expression of the myostatin gene in pigs. Eighteen pigs were allotted into three groups (six pigs per group), and pigs in treatment 1, 2 and 3 were immunized with physiological saline, 1 mg or 4 mg myostatin per pig, respectively. Six pigs were killed by electrical stunning followed by exsanguination at BW of 100 kg. The results indicated that the titer of myostatin antibody was increased in treated groups compared to the control group on day 42 ( P < 0.01) and d 84 ( P < 0.01). The carcass lean percentage was significantly increased in the treatment groups compared to the control group ( P < 0.01), and intramuscular fat was significantly decreased in the 4 mg group compared to the control group ( P < 0.05). The muscle creatine kinase activity of pigs treated with 1 mg and 4 mg myostatin was lower than the control group. The immunization of myostatin signofocantly decreased the myostatin gene expression levels in muscle. It was concluded that optimal active immunization against myostatin could increase the content of myostatin antibody, suppress the activity of creatine kinase and the expression of myostatin gene, and therefore improve the carcass lean percentage for pigs. 相似文献
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AIM: To investigate whether gold nanoparticles (GNPs) reverses adriamycin (ADM), resistance of human hepatocellular carcinoma drug-resistant cell line HepG2/ADM and to explore the potential mechanism. METHODS: The sensitivities of HepG2 cells and HepG2/ADM cells to ADM were tested by MTT assay before and after GNPs pretreatment. The apoptotic rate was examined by flow cytometry. The concentration of ADM in HepG2/ADM or HepG2 cells was determined by ultraviolet-visible spectrophotometer. The content of glutathione (GSH) in HepG2/ADM or HepG2 cells by DTNB method. RESULTS: The half maximal inhibitory concentrations (IC50) of ADM for HepG2/ADM cells were(29.46±1.73) mg/L and (15.18±0.85) mg/L before and after GNPs pretreatment,respectively. The IC50 of ADM for HepG2 cells was (9.16±2.03) mg/L before pretreatment. The apoptotic rate in GNPs+ADM group was higher than that in ADM group (P<0.05). The concentration of ADM in HepG2/ADM group was lower than that in HepG2 group (P<0.01). After GNPs pretreatment, the concentration of ADM in HepG2/ADM cells was higher than that before pretreatment. The content of GSH in HepG2/ADM group was higher than that in HepG2 group (P<0.01). After GNPs pretreatment, the content of GSH in the HepG2/ADM cells was lower than that before pretreatment. CONCLUSION: Gold nanoparticles can reverse ADM resistance of human hepatocellular carcinoma drug-resistant cell line HepG2/ADM, reduce the content of GSH and increase the concentration of ADM in HepG2/ADM cells. 相似文献
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HUANG Hua LI Yu-sheng JIN Xin WANG Jiang-tao LI Na HUANG Zhen-gui DING Bo-ping 《园艺学报》2015,31(8):1365-1370
AIM: To investigate the effect of rhynchophylline (Rhy) on blood pressure, cardiac hypertrophy and myocardial fibrosis in spontaneously hypertensive rats (SHR). METHODS: Spontaneously hypertensive rats were randomly divided into model group, high dose (10 mg·kg-1·d-1) and low dose (2.5 mg·kg-1·d-1) group of rhynchophylline, captopril group (17.5 mg·kg-1·d-1). Wistar-Kyoto rats were used as normal control. Respectively, systolic blood pressure was measured by tail cuff every 2 weeks. After 10 weeks, heart weight index and left ventricular weight index were calculated. The myocardial hydroxyproline and plasma angiotensin Ⅱ were detected. Moreover, basic myocardial histopathological changes and myocardial collagen fibres were observed by HE staining and Masson staining, respectively. The protein expression of TGF-β1 and Smad3 in the myocardium was measured by the methods of immunohistochemistry and Western blot. RESULTS: Compared with SHR model group, Rhy significantly reduced blood pressure (P<0.05), the levels of HYP in the myocardium (P<0.05) and the levels of AngⅡ in the plasma (P<0.01). The pathological damages of the myocardial tissues and collagen deposition were attenuated. The protein expression of TGF-β1 and Smad3 was significantly reduced by the treatment with Rhy (P<0.01). CONCLUSION: Rhynchophylline reduces blood pressure and adjusts to improve ventricular remodeling of SHR. The mechanism may be involved in the TGF-β1/Smad pathway and reducing AngⅡ content. 相似文献