Implant sequence effects in intact male Holstein veal calves: live and slaughter traits

Seven sequences of growth promotant implants were used in special-fed intact male Holstein veal calves (n = 443). Calves received implants 4 d after arrival at the veal barn, 42, and 84. The following implants were used: placebo (0), Z (36 mg zeranol), ET (20 mg estradiol, 200 mg testosterone), EP/2 (10 mg estradiol, 100 mg progesterone), EP (20 mg estradiol, 200 mg progesterone), and EBA (24 mg estradiol, 120 mg trenbolone acetate). The following sequences were compared: 0-0-0 (negative control), 0-ET-ET, Z-ET-ET, 0-EP-EP, Z-EP-EP, 0-EP/2-EBA, and Z-0-EBA. From 0 to 42 d, Z implants increased (P<.05) ADG by 3.4% compared to placebo. However, implant schemes without an initial Z implant (0-ET-ET and 0-EP-EP) had higher (P<.05) mean ADG for the period from d 42 to 84. From 84 d to the end of the experiment, only the 0-EP/2-EBA treatment increased (P<.05) ADG compared to 0-0-0. Over the entire trial 0-ET-ET, 0-EP-EP, Z-EP-EP, and 0-EP/2-EBA implant sequences increased (P<.05) ADG by 3.2, 3.2, 2.4, and 4.7%, respectively, compared to the 0-0-0 sequence. Blood traits measured within 2 wk before slaughter were not affected by implant sequence, except that sequences with EP had higher (P<.05) leukocyte counts than were observed for the other sequences. Testicular weight was less (P<.01) for all of the implant sequences than for the negative control and less (P<.05) for Z-ET-ET than for 0-ET-ET, 0-EP-EP, 0-EP/2-EBA, and Z-0-EBA. The type and frequency of medical treatments did not differ among implant sequences for any of the 42-d phases, or over the entire trial. Generally, the growth promotant implants currently approved for beef cattle resulted in approximately 50% of the increase in growth rate in Holstein intact bull calves, as has been observed in beef-type steers or heifers.     

Comparison of models to estimate genetic effects of weaning weight of Angus cattle

Weaning weights from nine sets of Angus field data from three regions of the United States were analyzed. Six animal models were used to compare two approaches to account for an environmental dam-offspring covariance and to investigate the effects of sire x herd-year interaction on the genetic parameters. Model 1 included random direct and maternal genetic, maternal permanent environmental, and residual effects. Age at weaning was a covariate. Other fixed effects were age of dam and a herd-year-management-sex combination. Possible influence of a dam's phenotype on her daughter's maternal ability was modeled by including a regression on maternal phenotype (fm) (Model 3) or by fitting grandmaternal genetic and grandmaternal permanent environmental effects (Model 5). Models 2, 4, and 6 were based on Models 1, 3, and 5, respectively, and additionally included sire x herd-year (SH) interaction effects. With Model 3, estimates of fm ranged from -.003 to .014, and (co)variance estimates were similar to those from Model 1. With Model 5, grandmaternal heritability estimates ranged from .02 to .07. Estimates of maternal heritability and direct-maternal correlation (r(am)) increased compared with Model 1. With models including SH, estimates of the fraction of phenotypic variance due to SH interaction effects were from .02 to .10. Estimates of direct and maternal heritability were smaller and estimates of r(am) were greater than with models without SH interaction effects. Likelihood values showed that SH interaction effects were more important than fm and grandmaternal effects. The comparisons of models suggest that r(am) may be biased downward if SH interaction and(or) grandmaternal effects are not included in models for weaning weight.