甜菜产生叉根的原因初探

从种子芽势、作物茬口、播种量、镇压次数、育苗苗龄方面调查分析了甜菜产生叉根的原因。     

不同铁肥品种对苹果叶片叶绿素、铁含量及光合速率的影响

采用断根输液法对缺铁失绿症的苹果树分别施用不同浓度的柠檬酸铁、螯合铁和邻啡罗啉铁3种铁肥,试验结果表明,断根输液能在较短时间内提高叶片的叶绿素和铁的含量,提高叶片的光合速率,从而获得较好的复绿效果。邻啡罗啉铁是适合根系输液矫正苹果树缺铁失绿症的铁肥品种。     

亲水胶体对竹荚鱼鱼糜凝胶特性的影

研究了亲水胶体类型和添加量对竹荚鱼鱼糜凝胶特性的影响.通过对竹荚鱼鱼糜的质构特性、持水力和白度的测定,以及超微结构观察等结果表明,添加可得胶、罗望子胶、魔芋胶、卡拉胶、琼胶和CMC的鱼糜凝胶能形成均匀、致密的三维凝胶网络结构,因此能显著改善竹荚鱼鱼糜的凝胶特性,其中可得胶的效果最显著,其次是罗望子胶、魔芋胶、卡拉胶;添加瓜尔胶和海藻酸钠对竹荚鱼鱼糜的凝胶特性无显著影响;添加黄原胶和果胶后形成了较疏松、网孔较大的网络结构,降低了竹荚鱼鱼糜的凝胶特性.     

基于冠层温度的温室葡萄CWSI模型试验研究

探讨并建立了适合于镇江丘陵地区温室葡萄水分状况监测的作物水分胁迫指数(CWSI)模型.通过田间实验和观测,得到了适合温室葡萄的CWSI经验模型中的经验关系.初步的检验和分析表明,这一模型是合理的,可以用于温室内葡萄基于冠层温度信息的水分状况监测.     

木薯粉对罗非鱼生长、饲料利用和鱼体营养成分的影响

选择健康的吉富罗非鱼(体重1.29±0.25 g)为实验鱼,配制五种木薯粉含量分别为0%、10%、20%、30%和40%的等氮(粗蛋白为32%)、等能(总能为16 kJ·g-1)实验饲料,饲喂罗非鱼56 d,每饲料组设三个重复,研究不同含量木薯粉对吉富罗非鱼生长、饲料利用及全鱼营养成分的影响.结果表明:(1)木薯粉能提高罗非鱼的增重和蛋白质效率,其中以40%木薯粉组的效果最佳,其增重率、蛋白质效率和饲料系数分别为1470.99%(P＜0.05)、2.36(P＜0.05)和1.27(P＜0.05).(2)40%木薯粉组的干物质和蛋白质的表观消化率分别较对照组提高13.87%(P＜0.05)和7.93%(P＜0.05).(3)40%木薯粉组的全鱼粗蛋白、粗灰分分别较对照组显著提高7.07%(P＜0.05)和8.78%(P＜0.05),粗脂肪降低了13.00%(P＜0.05).     

饵料种类、温度和规格对硬壳蛤的同化率的影响

试验结果表明,温度和规格对硬壳蛤同化率的影响不显著(P>0.05);饵料种类对硬壳蛤同化率影响显著(P<0.05).其中小球藻和湛江等鞭金藻的同化率显著高于小新月菱形藻(P<0.05),而小新月菱形藻又显著高于扁藻(P<0.05).     

河北外向型渔业发展战略研究

渔业是资源性产业,也是市场化、国际化程度较高的产业,统筹国内发展和对外开放,充分利用“两个市场、两种资源”,不断提高对外开放水平,是本国渔业适应经济全球化和世界海洋制度变革的必然要求。为了拓展渔业发展的新空间,加速渔业的发展,研究河北省外向型渔业的现状、特点、潜力、发展思路意义重大。     

几种常用渔药对三疣梭子蟹Portunus trituberculatus(Miers)稚蟹的急性毒性试验

采用常温静水实验法.对Ⅰ～Ⅱ期梭子蟹稚蟹(平均体长0.2～0.3 cm)进行急性毒性试验,结果表明:在水温26.2～27.8℃F,6种渔用药物对梭子蟹Ⅰ～Ⅱ期稚蟹的96 h半致死浓度(96 h LC50)分别为硫酸铜1.4 mg/L、纤虫清2.5 mg/L、清苔净16 mg/L、二氧化氯3.7 mg/L、强力杀菌消毒剂4.2 mg/L、二溴海因36.8 mg/L;安全浓度(Sc)分别为硫酸铜0.47 mg/L、纤虫清0.62mg/L、清苔净4.1 mg/L、强力杀菌消毒剂1.1 mg/L、二氧化氯1.1 mg/L、二溴海因13 mg/L.梭子蟹对上述药物的敏感性分别为硫酸铜＞纤虫清＞三氯异氰尿酸钠＞二氧化氯＞二溴海因＞清苔净.其中硫酸铜、纤虫清的安全浓度相对较低,在梭子蟹养殖过程当中应谨慎使用.     

小型自排沉沙池沉沙工况的水力学特性试验研究

首先介绍了小型自排沉沙池的工作原理与工作过程,然后以物理模型对小型自排沉沙的水力学特性进行试验研究,通过试验资料深入分析了小型自排沉沙池内水流的流态特征,流速分布等水力特性。试验研究旨在为进一步研究小型自排沉沙池沉沙工况提供依据。     

Protective effect of C1q/tumor necrosis factor-related protein 3 on vascular endothelium in rats with hyperuricemia

AIM To study whether C1q/tumor necrosis factor （TNF）-related protein 3 （CTRP3）protect vascular endothelium in rats with hyperuricemia and its potential mechanisms. METHODS An animal model of hyperuricemia was established by using male SD rats drinking 10% fructose water （n=10）. The rats drinking normal water served as normal controls （n=10）. After 12 weeks, the rats were given a single injection with Ad-CTRP3 or Ad-GFP. The experiment was ended at 14th day after transfection.The serum levels of uric acid and nitric oxide （NO） were evaluated. The serum contents of TNF-α and interleukin-6 （IL-6） were measured by ELISA. HE staining and TUNEL assay were used to assess the morphological changes of intima and apoptosis of endothelial cells in thoracic aorta, respectively. The mRNA levels of endothelial nitric oxide synthase （eNOS）, TNF-α and IL-6 were detected by RT-qPCR. The protein levels of CTRP3 and Toll-like receptor 4 （TLR4） were determined by Western blot. RESULTS Compared with normal control group, the rats with hyperuricemia showed lower CTRP3 and higher TLR4 protein levels in the thoracic aorta （P<0.05）. Hyperuricemic rats had higher serum contents of uric acid, TNF-α and IL-6 （P<0.05）. Also, the intima structure disturbance of thoracic aorta, increased apoptotic rate, higher mRNA levels of TNF-α and IL-6 as well as lower mRNA levels of eNOS were observed （P<0.05）. By contrast, CTRP3 over-expression decreased TLR4 protein levels, reduced inflammatory cytokines, and obviously improved the morphology and function of thoracic aorta in the rats with hyperuricemia. CONCLUSION CTRP3 protect vascular endothelium in rats with hyperuricemia maybe via down-regulation of TLR4- mediated inflammatory signaling pathway.