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341.
Rui KANO Tomoko TAKAHASHI Takeshi HAYAKAWA Yoshiki YAMAYA Atsuhiko HASEGAWA Hiroshi KAMATA 《The Journal of veterinary medical science / the Japanese Society of Veterinary Science》2015,77(9):1183-1185
Feline upper respiratory tract infection due to Aspergillus spp. is
considered an emerging disease, with the number of reported cases continuing to rise. In
this study, we report the first case of feline sinonasal aspergillosis caused by
Aspergillus fischeri in Japan. The patient presented after 2 months of
progressive facial deformity around the nose and nasal discharge. The isolate from this
case was susceptible to itraconazole (ITZ), voriconazole and micafungin, but was resistant
to amphotericine B. However, the infected cat died approximately 1 month after referral,
despite treatment for 12 days ITZ administered orally at 10 mg/kg. 相似文献
342.
Dai Kusumoto Takashi Yonemichi Hiroki Inoue Tomonori Hirao Atsushi Watanabe Toshihiro Yamada 《Journal of Forest Research》2014,19(2):285-294
Pine wilt disease caused by the pine wood nematode (PWN), Bursaphelenchus xylophilus, has been epidemic and has had disastrous impacts on pine forests and forest ecosystems in eastern Asia. Many pine species in this area are susceptible to this disease. Pinus thunbergii is particularly susceptible. In Japan, tree breeders have selected surviving trees from severely damaged forests as resistant candidates, and have finally established several resistant varieties of P. thunbergii. However, this breeding procedure requires much time and effort due to the lack of physiological and phenotypical information about resistance. To investigate the resistance mechanisms of selected P. thunbergii, we compared histochemical responses, tissue damage expansion, and PWN distribution in resistant and susceptible clones of P. thunbergii after PWN inoculation. The results suggested that the mechanisms of resistance are as follows: damage expansion in the cortex, cambium, and xylem axial resin canals are retarded in resistant trees soon after inoculation, probably due to the induction of wall protein-based defenses. Suppression of PWN reproduction was particularly caused by inhibition of damage expansion in the cambium. The slow expansion of damage in each tissue provides time for the host to complete the biosynthesis of lignin in the walls of cells that surround the damaged regions. This lignification of cell walls is assumed to effectively inhibit the migration and reproduction of the PWNs. The mechanism of initial damage retardation is presumed to be a key for resistance. 相似文献