Hormonal status of pregnant and infertile high producing cows

The content of ovarian, adrenal, and thyroid hormones in the blood of Red Pied cows at various times of pregnancy and in the case of infertility is studied. The characteristics of hormonal interrelations in various nosologically differentiated pathologies of the sex organs are determined. Opinions are expressed about possible ways to normalized their reproductive function.     

Cerebrospinal fluid creatine kinase activity in horses with central nervous system disease: 69 cases (1984-1989)

The CSF creatine kinase (CK) activity was determined in 70 CSF samples from 69 horses with CNS disease. Abnormal values (greater than or equal to 1 IU/L) were determined from 32 CSF samples, and normal values (less than 1 IU/L) were found in 38 samples. Increased CK activity was most frequently associated with a diagnosis of equine protozoal myelitis; CK activity was not increased in 11 horses with cervical compressive myelopathy. Other diagnoses, in which CSF CK activity was increased included trauma (n = 1), idiopathic epilepsy (n = 2), botulism (n = 2), articular facet fracture (n = 1), intervertebral disk protrusion (n = 1), and toxemia (n = 1).     

Ram management for northeastern flocks

The ram is an important member of the flock, particularly in smaller, intensively managed flocks such as are found in the Northeast. An infertile ram can spell reproductive and financial failure for the producer. A highly fertile ram can settle more ewes in a shorter time, produce more twins, and confer valuable traits on his offspring. These include wool type, rate of gain and higher sperm production and ovulation rates. Details of the breeding soundness examination, including semen evaluation are discussed. Ram management techniques appropriate for Northeastern sheep producers are presented.     

Blood sugar level following intravenous glucose in rheumatoid arthritis

Since the majority of patients with rheumatoid arthritis show a slower fall in the blood sugar level after the intravenous injection of glucose than do the normal controls, the alteration cannot be explained on the basis of gastrointestinal dysfunction. Differences in the renal threshold of glucose do not explain the altered glucose tolerance, since approximately the same amount of glucose is lost in the urine in both groups. Blood samples taken at 3 and 5 minutes following the injection of the glucose showed the height of the blood sugar level to be approximately the same in the patients with rheumatoid arthritis and in normals. The slower fall in the blood sugar level of the former is therefore not a simple function of a greater rise following the intravenous administration of the glucose. Although the patients with severe poliomyelitis had as much or more atrophy than the rheumatoid arthritic patients, there was no delay in rate of fall of the blood sugar level after the intravenous administration of glucose. In view of the fact that the hepatic homeostatic control regulates the blood sugar level, faulty utilization of glucose by extrahepatic tissues cannot be considered the primary factor responsible for the alteration of the glucose tolerance. The altered glucose tolerance in rheumatoid arthritis is explainable on the basis of an altered threshold of the hepatic homeostatic control of the blood sugar. Additional studies must be done to determine whether this derangement emanates directly from extrahepatic influences.