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排序方式: 共有3474条查询结果,搜索用时 553 毫秒
941.
942.
Mittermeier RA Gascon C Rajaobelina L Supriatna J Cardoso da Silva JM Rodríguez CM Zhi L Brandon K 《Science (New York, N.Y.)》2007,318(5855):1377-82; author reply 1377-83
943.
Rhesus Macaque Genome Sequencing Analysis Consortium Gibbs RA Rogers J Katze MG Bumgarner R Weinstock GM Mardis ER Remington KA Strausberg RL Venter JC Wilson RK Batzer MA Bustamante CD Eichler EE Hahn MW Hardison RC Makova KD Miller W Milosavljevic A Palermo RE Siepel A Sikela JM Attaway T Bell S Bernard KE Buhay CJ Chandrabose MN Dao M Davis C Delehaunty KD Ding Y Dinh HH Dugan-Rocha S Fulton LA Gabisi RA Garner TT Godfrey J Hawes AC Hernandez J Hines S Holder M Hume J Jhangiani SN 《Science (New York, N.Y.)》2007,316(5822):222-234
944.
mTORC1 senses lysosomal amino acids through an inside-out mechanism that requires the vacuolar H(+)-ATPase 总被引:3,自引:0,他引:3
Zoncu R Bar-Peled L Efeyan A Wang S Sancak Y Sabatini DM 《Science (New York, N.Y.)》2011,334(6056):678-683
The mTOR complex 1 (mTORC1) protein kinase is a master growth regulator that is stimulated by amino acids. Amino acids activate the Rag guanosine triphosphatases (GTPases), which promote the translocation of mTORC1 to the lysosomal surface, the site of mTORC1 activation. We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary for amino acids to activate mTORC1. The v-ATPase engages in extensive amino acid-sensitive interactions with the Ragulator, a scaffolding complex that anchors the Rag GTPases to the lysosome. In a cell-free system, ATP hydrolysis by the v-ATPase was necessary for amino acids to regulate the v-ATPase-Ragulator interaction and promote mTORC1 translocation. Results obtained in vitro and in human cells suggest that amino acid signaling begins within the lysosomal lumen. These results identify the v-ATPase as a component of the mTOR pathway and delineate a lysosome-associated machinery for amino acid sensing. 相似文献
945.
Habitat split and the global decline of amphibians 总被引:2,自引:0,他引:2
Becker CG Fonseca CR Haddad CF Batista RF Prado PI 《Science (New York, N.Y.)》2007,318(5857):1775-1777
The worldwide decline in amphibians has been attributed to several causes, especially habitat loss and disease. We identified a further factor, namely "habitat split"-defined as human-induced disconnection between habitats used by different life history stages of a species-which forces forest-associated amphibians with aquatic larvae to make risky breeding migrations between suitable aquatic and terrestrial habitats. In the Brazilian Atlantic Forest, we found that habitat split negatively affects the richness of species with aquatic larvae but not the richness of species with terrestrial development (the latter can complete their life cycle inside forest remnants). This mechanism helps to explain why species with aquatic larvae have the highest incidence of population decline. These findings reinforce the need for the conservation and restoration of riparian vegetation. 相似文献
946.
Effectiveness of parks in protecting tropical biodiversity 总被引:2,自引:0,他引:2
We assessed the impacts of anthropogenic threats on 93 protected areas in 22 tropical countries to test the hypothesis that parks are an effective means to protect tropical biodiversity. We found that the majority of parks are successful at stopping land clearing, and to a lesser degree effective at mitigating logging, hunting, fire, and grazing. Park effectiveness correlates with basic management activities such as enforcement, boundary demarcation, and direct compensation to local communities, suggesting that even modest increases in funding would directly increase the ability of parks to protect tropical biodiversity. 相似文献
947.
Ahn TK Avenson TJ Ballottari M Cheng YC Niyogi KK Bassi R Fleming GR 《Science (New York, N.Y.)》2008,320(5877):794-797
Energy-dependent quenching of excess absorbed light energy (qE) is a vital mechanism for regulating photosynthetic light harvesting in higher plants. All of the physiological characteristics of qE have been positively correlated with charge transfer between coupled chlorophyll and zeaxanthin molecules in the light-harvesting antenna of photosystem II (PSII). We found evidence for charge-transfer quenching in all three of the individual minor antenna complexes of PSII (CP29, CP26, and CP24), and we conclude that charge-transfer quenching in CP29 involves a delocalized state of an excitonically coupled chlorophyll dimer. We propose that reversible conformational changes in CP29 can "tune" the electronic coupling between the chlorophylls in this dimer, thereby modulating the energy of the chlorophyll-zeaxanthin charge-transfer state and switching on and off the charge-transfer quenching during qE. 相似文献
948.
Using a patterned, grating-like plate to control the electromagnetic near field, we demonstrate focusing well beyond the diffraction limit at approximately 1 gigahertz. The near-field plate consists of only capacitive elements and focuses microwaves emanating from a cylindrical source to a spot of size approximately lambda/20 (half-power beamwidth), where lambda is the free-space wavelength. These plates will find application in antennas, beam-shaping devices, nonradiative wireless power-transfer systems, microscopy, and lithography. 相似文献
949.
Lysosomal glycosphingolipid recognition by NKT cells 总被引:2,自引:0,他引:2
Zhou D Mattner J Cantu C Schrantz N Yin N Gao Y Sagiv Y Hudspeth K Wu YP Yamashita T Teneberg S Wang D Proia RL Levery SB Savage PB Teyton L Bendelac A 《Science (New York, N.Y.)》2004,306(5702):1786-1789
NKT cells represent a distinct lineage of T cells that coexpress a conserved alphabeta T cell receptor (TCR) and natural killer (NK) receptors. Although the TCR of NKT cells is characteristically autoreactive to CD1d, a lipid-presenting molecule, endogenous ligands for these cells have not been identified. We show that a lysosomal glycosphingolipid of previously unknown function, isoglobotrihexosylceramide (iGb3), is recognized both by mouse and human NKT cells. Impaired generation of lysosomal iGb3 in mice lacking beta-hexosaminidase b results in severe NKT cell deficiency, suggesting that this lipid also mediates development of NKT cells in the mouse. We suggest that expression of iGb3 in peripheral tissues may be involved in controlling NKT cell responses to infections and malignancy and in autoimmunity. 相似文献
950.
The Anopheles gambiae genome sequence will accelerate identification of new insect vector target genes leading to improved strategies for malaria control. 相似文献