渭河阶地全新世土壤粒度成分高分辨率研究

通过对渭河阶地全新世土壤的野外考察和粒度成分的高分辨率分析研究，揭示了该区域褐色土的成壤作用过程与季风气候变化的关系。研究表明渭河第二级阶地褐色土形成在全新世气候最适宜期（8500－3100a B.P.），从8500a B.P.开始该土壤开始发育，老官台文化兴起。从3100a B.P.开始的气候干旱化和沙尘暴堆积形成了黄土L。层。西周时期对应于该干旱期的初期。     

荔枝、龙眼园使用机油乳剂的安全性及其对荔枝蒂蛀虫的防治研究

利用机油乳剂对果树进行安全性和防治荔枝蒂蛀虫Conopomorpha sinensis Bradley研究试验，结果表明，喷施机油乳剂后荔枝和龙眼的落果率与对照树4无差异，喷清水的对照树其蒂蛀虫蛀果率比喷机油乳剂的处理树高，分别为150、200和250倍机油乳剂处理的2.8、3.0和2.7倍。在荔枝、龙眼园应用机油乳剂防治害虫不会导致落叶、落果、畸形叶和畸形果，也不会延缓果实的生长和成熟及降低单果重，而有利于控制介壳虫、螨类、蛀果类等害虫。机油乳剂可替代广谱性化学农药的使用，减少化学农药的污染，有利于果园天敌的生存、繁殖，提高生物多样性，改善果园生态环境。     

新型取代氨基(或芳氧)磺酰基苯氧丙酸酯的合成及其除草活性

由α-苯氧基丙酸酯出发,合成了取代氨基(或芳氧)磺酰基苯氧丙酸酯,并测定了它们的除草活性。所有的化合物均经1H NMR和元素分析确证,初步的生测结果表明上述化合物具有一定的除草活性。     

稻象甲的防治指标和防治适期研究

根据稻象甲虫量与水稻产量的关系 ,结合现行的稻谷价格 ,产量水平和防治费用等因素 ,进行了稻象甲危害损失的测定 ,制定了稻象甲的防治指标。采用累积虫日作为防治指数 ,确定了在江西境内稻象甲的防治适期     

苏云金芽孢杆菌cry基因在大肠杆菌中表达产物和生物活性

研究了几种Bt cry基因于大肠杆菌(Escherichia coli)中表达产物在pH 10.0的50mmol/L碳酸钠和20mmol/L乙醇胺溶解液中的溶解性 ,发现同样的Cry蛋白在碳酸钠中的溶解度大于乙醇胺。通过胰蛋白酶消化 ,明确Cry1Ca7、Cry1Ia8酶解产物为 38kD多肽 ;Cry1Ie1、Cry1Cb2、Cry2Ab4酶解产物为 41kD多肽 ;Cry1Ac酶解产物为60kD多肽。采用FPLC层析方法对 6种原毒素及其酶解后得到的毒素多肽进行了分离纯化 ,比较了原毒素和毒素的杀虫活性的差异。其结果表明 ,Cry1Ac的原毒素和毒素对棉铃虫初孵幼虫的校正死亡率均为 100% ,Cry2Ab4的原毒素的毒力高于其酶解毒素。     

转Bt基因棉在安庆市大面积种植后害虫发生特点调查

根据调查,转Bt基因棉大面积种植后,棉铃虫、红铃虫发生减轻;棉叶螨发生加重,为害时间延长;甜菜夜蛾、斜纹夜蛾发生面积扩大,为害程度加重。据此初步提出了当地转Bt基因棉害虫防治中应注意的问题和管理对策。     

梨果实若干性状的遗传倾向

 以梨12 个杂交组合和138 个栽培品种为试材, 研究几个果实性状的遗传倾向, 结果如下: 栽培品种果实含糖量集中在6 %～9 % , 平均为7. 39 %; 果实含糖量表现为数量性状, 呈正态分布, 杂种后代果实含糖量主要分布在6 %～10 % , 平均为7. 8 % , 遗传传递力平均为90. 8 %。栽培品种含酸量介于0. 08 %～0. 74 % , 平均为0. 238 %; 杂种后代果实含酸量为数量性状, 组合遗传传递力平均为69. 2 %。梨果实挥发性物质共检测出5 种酯类、8 种醇类和乙烯、乙醛、丙酮类共16 种化合物。杂种后代果实中发现了其亲本果实中所没有的戊醇成分。果实肉质性状表现为质量性状, 脆肉对软肉为显性。杂种后代表现为果点变大、密, 果柄变长, 果汁变少, 果皮厚度相当于亲中值。     

Effect of heat shock precondition on reperfusion arrhythmia in rats

AIM:To investigate the effect of the heat shock response on the reperfusion arrhythmias(RAs) and the possible mechanism involved. METHODS:Fifty-five Sprague Dawley rats were randomly divided into 2 groups: the heat shock group (group H,n=29) and the control group (group C,n=26). The rats in group H were preconditioned with heat shock 24 hours before, and that in group C were not. The hearts of 16 rats in group H and 16 in group C were exercised and mounted on a non-circulating Langendorff perfusion apparatus and perfused retrogradely with modified K-H buffer and mimic ischemia/reperfusion was applied. Additionally, conventional intracellular microelectrode techniques were used for recording such electrophysiological parameters as resting potential(RP), action potential amplitude(APA), over shot(OS), maximum depolarization velocity(Vmax) of the hearts of other 13 rats in group H and 10 in group C. RESULTS:①Prior heat stress significantly decreased reperfusion arrhythmia. ②The amount of CK release in the effluent in group H was much less than that in group C. ③Myocardial HSP70 content was elevated significantly in group H. ④Heat stress significantly increased myocardial anti-oxydases activity and decreased lipid peroxydative products. Additionally, heat stress significantly reduced the Vmax of action potential. It indicated that rapid Na⁺ channel of papillary muscles may be inhibited by heat shock. The degree of change of Vmax after ischemia in H group was significantly less than that in group C. And the time of reperfusoin with Tyrode's solution till the action potential appeared as large as that before perfusion with mimic ischemic solution is shorter in group H than in group C. CONCLUSION:Heat shock pretreatment markedly reduces ischemia/reperfusion-induced injury of heart and ventricular arrhythmias in rats and this effect may be associated with the inhibition of rapid Na⁺ channel of papillary muscles by heat shock and the increase in myocardial HSP70 and anti-oxydase activity.     

Effect of lansoprazole on gastric ulceration in rats

AIM:To investigate the effect of lansoprazole on gastric ulceration in rats. METHODS:Using the gastric ulcer model induced by hemorrhagic shock, restraint water-immersion stress and pylorus-ligature, the protective effect of lansoprazole (iv) on gastric ulceration was observed. RESULTS:Pretreatment with lansoprazole (7.5-60 mg/kg) significantly inhibited the formation of gastric ulcer in the three models in a dose-dependent manner. The autiulcer efficacy of lansoprazole was similar to that of omeprazole in the equal dose, but stronger than that of omeprazole for ulcer induced by water-immersion stress.CONCLUSION:The intravenously administered lansoprazole inhibited formation of experimental gastric ulcer in rats.