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41.
缺磷胁迫对温州蜜柑光合作用的光抑制研究(一) 总被引:3,自引:2,他引:3
经缺磷处理后,温州蜜柑叶片的表观量子效率(AQY),最大荧光(Fm),光化学效率(Fv/Fm),光量子产量(Y)和电子传递速率(ETR)下降,而初始荧光(Fo)上升。净光合速率(Pn)和光呼吸速率(Pr)均下降。但Pr/Pn比值升高。表明缺磷处理后,温州蜜柑更易发生光合作用的光抑制。 相似文献
42.
头足类是最具开发潜力的渔业种群之一,然而其资源易受环境变化影响,复杂的交互作用致使二者空间关系呈现非均一性。中国枪乌贼 (Uroteuthis chinensis) 是南海北部近海重要的经济物种,且在渔业群落结构中占据优势种地位,故正确理解与掌握该物种资源-环境关系的空间特征,有助于该资源的保护与利用。基于南海北部近海2014年夏季的渔业资源调查数据,构建了地理权重回归模型 (Geographical weighted regression, GWR),探索该海域中国枪乌贼的资源分布与海洋环境关系的空间特征,阐述主要影响因子。模型评价指标结果表明,GWR的最小赤池信息准则 (Akaike Information Criterion, AIC) 和校正决定系数 (Adjusted R-Squared, Uroteuthis chinensis is an important economic species in the offshore waters of northern South China Sea, occuping a dominant position in the fishery community structure. Therefore, understanding the spatial characteristics of the resource-environment relationship is beneficial to its development, utilization and protection. Based on the fishery resources survey data in the offshore waters of northern South China Sea in summer of 2014, we established a geographically weighted regression (GWR) model to explore the spatial characteristics of the relationship between the resource distribution of U. chinensis and the marine environment in this area, and to reveal the main influencing factors. The results of model evaluation indexes show that the minimum Akaike information criterion (AIC) and adjusted R-Square (K>P.For some middle part of Zhejiang province,the soil of which deficient in Zn,supplement of Zn-fertilizer could promote early maturity ... 相似文献
43.
以番茄灰霉病菌为供试菌种,采用电导率法、吸光度法和电镜分析法对经过喜树碱(CPT)处理的番茄灰霉病菌进行病理反应研究。试验结果表明经200 mg/L CPT溶液处理后,番茄灰霉病菌菌丝细胞膜的通透性增加了30.7%,细胞外蛋白质浓度提高了13.5%。番茄灰霉病菌经CPT溶液处理后,还原糖和可溶性蛋白的含量随着处理时间的增长先升高后降低最后趋于平缓,在0~1 h之间增长最快。CPT处理后的番茄灰霉病菌的菌丝畸形,出现膨大或缢缩,菌丝生长点缢缩,细胞中的空腔增多,线粒体增多且线粒体嵴变得模糊,细胞核有降解的现象。 相似文献
44.
45.
HE Yan-jing CHEN Ya-hong QI Yong-fen YAO Wan-zhen BI Xiao-rui GUAN Yu-hong ZHU Rui-xia TANG Chao-shu 《园艺学报》2011,27(6):1169-1175
AIM: To investigate the role of hydrogen sulfide (H2S) in the protection against oxidative stress in rats with chronic obstructive pulmonary disease (COPD).METHODS: The rat model of COPD was established by cigarette smoking (CS) combined with lipopolysaccharide (LPS) instillation. Thirty-two healthy male Sprague-Dawley rats were randomly divided into 4 groups: control group, CS+LPS group, CS+LPS+NaHS (H2S donor) group and CS+LPS+PPG (DL-propargylglycine, an inhibitor of cystathionine-γ-lyase) group. After 30 days, the lung functions of the rats were measured, the histological changes of lungs were observed under light microscope and the pathological scores were calculated. The H2S level in plasma and the protein expression of cystathionine-γ-lyase (CSE) in the lung tissues were measured. The content of malondialdehyde (MDA), the activity of superoxide dismutase (SOD) and catalase (CAT) were detected to reflect oxidative stress.RESULTS: Compared with control group, the peak expiratory flow (PEF) decreased by 24% and intra-pressure (IP) increased by 66% in CS+LPS group. The pathological scores of the lung tissues also increased. Compared with CS+LPS group, no change in the lung function was observed after given NaHS or PPG, but the pathological scores decreased in CS+ LPS+ NaHS group. Compared with control group, the content of H2S in plasma was increased by 26% on day 16. Compared with CS+LPS group, the content of H2S in plasma of CS+LPS+PPG group was decreased by 22% after 30 days. Compared with control group, the protein expression of CSE increased, and no statistical difference among CS+LPS group, CS+ LPS+ NaHS group and CS+LPS+PPG group was observed. Compared with control group, MDA content in the lung tissues was increased by 24% in CS+LPS group, the activity of SOD was increased by 47% and the activity of CAT was increased by 52%. Compared with CS+LPS group, the MDA content in CS+LPS+NaHS group was decreased by 21%, and no statistical difference in the activity of SOD and CAT was observed. The activity of SOD decreased by 33% after given PPG.CONCLUSION: H2S plays a role as anti-oxidant in the rats with COPD. The CSE/ H2S pathway may be involved in the development of COPD. 相似文献