蛋鸡产蛋下降综合症的病因及防治

蛋鸡产蛋下降综合症却直接影响着蛋鸡饲养业经济效益的提高。因此,深入探讨蛋鸡产蛋下降综合症的病因,制定卓有成效的防治措施,已成为当今养鸡业的热门话题。 一、蛋鸡产蛋下降综合症的病因 蛋鸡产蛋下降综合症主要是由于家禽疾病、饲养不善、管理不当等多方面原因而引起。 １．引起蛋鸡产蛋下降综合症的疾病主要有以下几种：①由肠病毒感染引起的鸡脑脊髓炎。该病可使蛋鸡在１０天左右将产蛋率下降４０％左右。②由副粘病毒感染引起的鸡新城疫。该病可使产蛋率严重下降。③由疱疹病毒感染引起的鸡传染性喉气管炎,也可使产蛋率下降。④…     

蛋鸡脂肪肝综合症的临床防治

蛋鸡脂肪肝综合症,当前在国内报道较少。英国工作者(Goran 1977)提出鸡脂肪肝和肾综合症,主要发生在11—32日龄的肉仔鸡。丹麦工作者于1958年首次确诊。美国报道的脂肪肝综合症未见到明显肾变,并认为以笼养产蛋母鸡为多发,没有严格日龄关系,可能一是因为能量饲料过高;二是蛋鸡能量     

蛋鸡脂肪肝综合症的危害及防治措施

脂肪肝综合症，又称脂肝病，该病是因代谢障碍而导致肝脏脂肪的变性，腹腔中脂肪沉积。该病主要多发于体况良好的产蛋鸡，尤其是笼养蛋鸡则更易发。病鸡在临床上并无明显的临床症状，剖检时可见肝脏肥大，脂肪浸润，易碎，呈褐色，表面有出血现象。皮下及内脏的脂肪增多，鸡体较正常鸡偏大，但产蛋量下降，开产鸡产蛋率上升慢，且很难达到产蛋高峰。由于脂肪肝综合症临床症状不明显，此病虽然导致鸡死亡率很低，但对生产水平影响很大，因此该病在养鸡生产上应当及早引起注意。     

蛋鸡脂肪肝综合症防治措施

<正>蛋鸡脂肪肝综合症是一种营养代谢性疾病。主要引起蛋鸡不明原因的产蛋下降和死亡率上升,引起鸡肝脏发生脂肪变性、出血,有时导致急性死亡为特征的营养代谢性疾病。近年来,在养鸡业追求高效生产的条件下,该病的发生呈逐年增高的趋势,主要发生于蛋鸡,特别是笼养蛋鸡的产蛋高峰期,直接影响家禽业的经济效益,而越来越引起人们的高度重视。1发病机理鸡肝脏是合成体内脂肪的最主要场     

蛋鸡脂肪肝综合征的病因及其发病机理

蛋鸡脂肪肝综合症是一种营养代谢性疾病。它是由于鸡产蛋高峰期机体代谢旺盛 ,引起脂肪代谢紊乱 ,导致极低密度脂蛋白合成及转运受阻 ,使肝脏中合成的脂肪酸不能排出 ,造成甘油三酯在肝细胞内过度沉积。它导致青年蛋鸡不明原因的产蛋率下降和死亡率上升。本文对脂肪肝发病的症状、病因、发病机理进行综述     

鸡产蛋下降综合征

<正>秋冬是鸡只疾病高发季节,多种传染病可以引起鸡只产蛋下降,本文介绍鸡产蛋下降综合征的预防。1鸡产蛋下降综合症1.1流行病学鸡产蛋下降综合症(EDS)由鸡产蛋下降综合症病毒引起,主要侵害商品蛋鸡和种鸡,造成产蛋量下降及蛋品质降低。其他日龄鸡感染后无明显临床症状。     

蛋鸡脂肪肝综合症的病因与防治

脂肪肝综合症(FLS)是蛋鸡常见的一种脂类代谢障碍疾病,主要特征是肝脏脂肪变性、出血,普遍发生于6～9月炎热季节产蛋高峰期膘情较好的笼养蛋鸡。1病因1.1营养因素(1)能蛋比失衡:鸡的脂肪主要在肝脏合成,合成后必须与蛋白质结合才能从肝脏运出。当给     

蛋鸡产蛋下降综合征的临床症状与防治

<正>1蛋鸡产蛋下降综合症的临床症状蛋鸡产蛋下降综合症英文简称EDS-76,也称为鸡减蛋综合症,这种病主要是由腺病毒引起的,鸡一旦患这种病则使鸡产量下降,蛋品质也下降,且是一种传染性疾病,该病发生的季节是春冬季节,气温一旦变化严重,则会引起产蛋下降,感染疾病的鸡发病时并没有显著的症状,产蛋下降也是在26～36周龄时才真正表现出来,下降的比     

蛋鸡脂肪肝出血综合征的发病原因及其防治

蛋鸡脂肪肝出血综合征是蛋鸡养殖生产中的常见和多发的营养代谢性疾病综合症,临床表现以突然发病、产蛋量下降和高产蛋鸡突然死亡为主要特征。特别是进入产蛋高峰期的鸡发病,常会给养殖户带来很大的经济损失。 近年来,笔者在动物门诊工作中,发现蛋鸡脂肪肝出血综合征（FLHS）有逐年上升的趋势,现笔者对该病的发病及诊疗小结如下,供业界同仁参考     

鸡戊型肝炎病毒感染及其鉴别诊断

鸡戊型肝炎病毒(禽HEV)是导致鸡肝脏、脾脏肿大综合症(HS)的主要病原,近期在临床可疑病例发生增多.该病以病鸡肝脏、脾脏肿大以及蛋鸡、肉种鸡的产蛋下降、死淘率增加等为主要危害特征.鉴于引起肝脏病变的病因较为复杂,在此做一下鉴别.     

Sonography of the liver, gallbladder, and spleen

Focal hepatic and splenic lesions, vascular abnormalities, and disorders of the biliary system can readily be detected with ultrasonography. The sonographic appearance in certain cases may be nonspecific, but the differential diagnosis can be substantially narrowed when the presenting clinical signs are considered. Ultrasonography is less helpful when the liver or spleen is diffusely involved without parenchymal abnormalities. However, certain diseases may also be eliminated from diagnostic consideration on the basis of this finding. Percutaneous ultrasound guided biopsy markedly improves the success and safety of obtaining a definitive diagnosis when either diffuse or focal lesions are present. Sonography has also been very beneficial for serially evaluating the response to therapy once focal lesions of the liver or spleen have been identified.     

Sonography of the liver, gallbladder, and spleen

Focal hepatic and splenic lesions, vascular abnormalities, and disorders of the biliary system can readily be detected with ultrasonography. The sonographic diagnosis can be substantially narrowed when the presenting clinical signs are considered. Ultrasonography is less helpful when the liver or spleen is diffusely involved without parenchymal, abnormalities. However, certain diseases may also be eliminated from diagnostic consideration on the basis of this finding. Percutaneous ultrasound guided biopsy markedly improves the success and safety of obtaining a definitive diagnosis when either diffuse or focal lesions are present. Sonography has also been very beneficial for serially evaluating the response to therapy once focal lesions of the liver or spleen have been identified.     

Comparative studies on the effect of fenbendazole on the liver and liver microsomal enzymes in goats,quail and rats

To compare the effect of fenbendazole on the liver and liver microsomal mono-oxygenases of goats, quail and rats, an oral dose of 25 mg/kg was administered to the animals daily for 9 consecutive days. On the tenth day, blood samples and livers were collected from both the control and the treated animals for preparation of serum and microsomes respectively. Determination of the activities of sorbitol dehydrogenase (SDH), alanine aminotransferase (ALT) and aspartate aminotransferase (AST) in the serum samples showed that there was no significant increase in the activities of these enzymes in the treated animals as compared to their corresponding controls, suggesting no liver damage. Similarly, no significant difference in the amount of microsomal cytochrome P-450 was found between the control and the treated animals of the same species. Compared to their respective controls, the activities of microsomal benzphetamine N-demethylase and aniline hydroxylase were almost unchanged in the treated goats and rats. However, fenbendazole treatment appeared to enhance the activity of these two microsomal enzymes in quail. The results indicate that fenbendazole is not liver toxic to goats, quail or rats at a dose rate of 25 mg/kg.     

纳米氧化铜对雏鸡全血和肝脏铜含量及肝脏铜蓝蛋白mRNA表达的影响

选取1日龄SPF白莱航蛋用公雏63只,随机分为7组,1组受试鸡饲喂基础日粮;2～4组受试鸡分别饲喂以硫酸铜、氧化铜和纳米氧化铜的形式添加铜8mg/kg的饲料;5～7组受试鸡分别饲喂以硫酸铜、氧化铜和纳米氧化铜的形式添加铜175mg/kg的饲料。结果显示,日粮中添加铜8mg/kg或175mg/kg时,纳米氧化铜组受试鸡全血、肝脏铜含量显著高于氧化铜组、硫酸铜组和对照组(P<0.05);纳米氧化铜组受试鸡肝脏血浆铜蓝蛋白(CP)mRNA表达量显著高于硫酸铜组和氧化铜组(P<0.05)。试验至30d,日粮添加铜8mg/kg或175mg/kg时,纳米氧化铜组受试鸡血浆CP活性显著高于硫酸铜组和氧化铜组(P<0.05)。表明纳米氧化铜通过提高脏器组织铜沉积、上调铜酶或铜蛋白的基因表达来提高铜酶活性,从而发挥其生物学作用。