寿光地区犬细小病毒病的流行病学调查

犬细小病毒病是犬的一种急性传染病。临床上病犬多以出血性肠炎或非化脓性肌炎为其主要特征。有时其感染率可高达100%,致死率为10%~50%[1]。本论文通过对山东寿光本地宠物医院就诊的疑似犬细小病毒犬中,选择经CPV胶体金快速检测试纸确诊为犬细小病毒病的病例为调查对象,     

犬细小病毒病流行病学调查

犬细小病毒感染是由犬细小病毒引起的一种急性、热性、传染性疾病,临床上以出血性肠炎或心肌炎为主要特征。本研究对泰州某宠物门诊2010年1月－2011年3月期间接诊的260例犬细小病例进行了分析研究,旨在了解泰州地区犬细小病毒病发病率、死亡率与犬年龄、品种、免疫接种、季节等因素之间的关系,并总结防控犬细小病毒病最有效的方法。结果表明,犬细小病毒病发病率及死亡率与犬的年龄、品种、免疫接种、季节有很大的相关性,依此提出了防制对策,以期为防制犬细小病毒病提供参考。     

犬细小病毒病的诊治

犬细小病毒病是由细小病毒引起的一种急性传染病．临床上以出血性肠炎或非脓性心肌炎为主要特征。本病幼犬多发，发病急，死亡率高。现将一典型病例介绍如下。     

犬细小病毒病

本病是犬的一种急性传染病。临床上多以出血性肠炎或非化脓性心肌炎为其主要特征。有时其感染率可高达100%，致死率为10%-50%。犬、猫和貂的细小病毒具有一定的抗原相关性。     

犬细小病毒病诊治

犬细小病毒病是由犬细小病毒引起的一种急性传染病,临床上以出血性肠炎或非化脓性心肌炎为其主要特征。本病一年四季都有发生,本病无明显季节性,但春秋发病率较高,幼犬易感,小于4周龄的吮乳仔犬和大于5岁的成年犬的发病率均很低,2～12月龄幼犬的发病率可高达60%以上,全窝仔犬发     

犬细小病毒病的诊治

犬细小病毒（以下简称CPV）是一种烈性传染病，临床表现以急性出血性肠炎和非化脓性心肌炎为特征。     

犬细小病毒病的临床调查

犬细小病毒病是由犬细小病毒(Canine parvovirus,CPV)引起的一种急性接触性传染病,临床上以发生出血性肠炎的病例多见.广州市怡友动物医院自2005年1月至2006年12月两年间接诊CPV感染犬317例,笔者对此进行了临床调查和分析.     

犬细小病毒病的诊疗

犬细小病毒病是由犬细小病毒引起的一种急性传染病,临床以出血性肠炎、呕吐、严重脱水或心肌炎为特征,多发于2~6月龄幼犬,发病率与死亡率很高,对养犬业的危害很大,笔者在多年的临床中总结出一套比较切实可行的治疗方案,供广大养犬户及同行参考。1病原犬细小病毒(CPV)属细小病毒     

犬细小病毒病的流行病学调查

笔者就重庆市涪陵区江东畜牧兽医站,在多年从事兽医工作期间对犬细小病毒感染病犬进行了流行病学调查和临床综合治疗,共计收治300例,其中疑似犬细小病毒病100例,确诊60例,死亡12例,治愈48例。并对当地区犬细小病毒病的流行情况进行了调查,经调查整理,对数据进行     

犬细小病毒病的诊治

犬细小病毒性肠炎又称犬传染性肠炎,是犬的一种高度接触性烈性传染病。该病临床上以急性出血性肠炎和心肌炎为特征。我们在2007~2008年间接诊的500多个病例中,患细小病毒的犬215个,占42%,早期病例治愈率达90%～     

Canine parvovirus in New Zealand: epidemiological features and diagnostic methods

Data from 763 cases of clinical canine parvovirus disease confirmed at the Ruakura Animal Health Laboratory were examined. The largest number of cases were seen in spring and summer months with the peak incidence in February 1981. The morbidity and mortality rates were highest in young dogs. Sixty-nine percent of all cases occurred in dogs less than six months of age, and 63 percent of dogs seven weeks of age or younger died. The laboratory methods used to diagnose canine parvovirus disease are compareh and discussed.     

Canine parvovirus

Extract

Sir:- The paper Canine parvovirus in New Zealand: epidemiological features and diagnostic methods by G.W. Horner [N.Z. vet. J. 31 (1983): 164-4] suggests “it is probable that many veterinary clinics were heavily contaminated with CPV” quoting an Australian reference in support of his contention.     

Canine parvovirus infection

Extract

Sir — Referring to M. H. Blunt's letter to the Journal for December, 1980 regarding canine parvovirus disease, I would like to take issue with several points.     

Canine parvovirus in Australia: The role of socio-economic factors in disease clusters

To identify clusters of canine parvoviral related disease occurring in Australia during 2010 and investigate the role of socio-economic factors contributing to these clusters, reported cases of canine parvovirus were extracted from an on-line disease surveillance system. Reported residential postcode was used to locate cases, and clusters were identified using a scan statistic. Cases included in clusters were compared to those not included in such clusters with respect to human socioeconomic factors (postcode area relative socioeconomic disadvantage, economic resources, education and occupation) and dog factors (neuter status, breed, age, gender, vaccination status). During 2010, there were 1187 cases of canine parvovirus reported. Nineteen significant (P<0.05) disease clusters were identified, most commonly located in New South Wales. Eleven (58%) clusters occurred between April and July, and the average cluster length was 5.7days. All clusters occurred in postcodes with a significantly (P<0.05) greater level of relative socioeconomic disadvantage and a lower rank in education and occupation, and it was noted that clustered cases were less likely to have been neutered (P=0.004). No significant difference (P>0.05) was found between cases reported from cluster postcodes and those not within clusters for dog age, gender, breed or vaccination status (although the latter needs to be interpreted with caution, since vaccination was absent in most of the cases). Further research is required to investigate the apparent association between indicators of poor socioeconomic status and clusters of reported canine parvovirus diseases; however these initial findings may be useful for developing geographically- and temporally-targeted prevention and disease control programs.     

Canine parvovirus infection in Nigeria

Pups of Nigerian mongrel dogs randomly bought from two village markets for experimental purposes at the University of Nigeria, Nsukka, were observed to be ill within 4 days of purchase. By 13 days after purchase 52.5 per cent of all the animals had died. All sick animals were anorectous and suffered from diarrhoea. The faeces were mostly dark yellow to light brown and a few contained spots of blood. Few animals responded to treatment with fluids, antibiotics and vitamins and death occurred within 24 hours of the onset of clinical signs. In most cases, post mortem and histopathological lesions were similar to those characteristic of canine parvovirus. The sera of the surviving animals tested for haemagglutination-inhibition antibodies to canine parvovirus, using porcine red blood cells, were positive.