八种禾本科干燥花的漂白研究

在室温下用双氧水作漂白剂，研究了2种不同成熟度的8种禾本科干燥的漂白效果，结果表明：1.嫩材的成材率比老材的高，但嫩材所需的漂白时间比老材的长，而且嫩材的褪色效果也没有老材的好。2.含叶绿素较多的花材比含胡萝卜素和黄酮类色素多的花材漂白时间长且漂白白度低，籽粒型花材的漂白时间较绒毛型花材的长，质地松散的花材较质地致密的花材漂白时间短。     

H2O2对月季平面干燥花漂白的影响

在60℃温度条件下,使用H2O2作漂白剂,研究了6种不同H2O2浓度和4种漂液pH值对红色月季平面干花漂白效果的影响.结果表明:漂液浓度越大,漂白速度越快,但超过一定浓度时对材料的损伤越大,漂液适宜浓度在15%～20%之间;随着pH值增大,漂白所用的时间缩短,但当pH值超过一定范围时,漂液开始对材料有损伤,漂液pH值在9时漂白效果最好.     

干花染色技术研究

研究了云南省常见的几种漂白过的花材的染色工艺.结果表明:染液的浓度增加,染色效果显著,染液浓度为3‰时可获得满意效果.温度升高及时间增加,有利于花材的染色,当温度达到70～90℃时染色效果最佳,染液pH在4～6之间为佳.     

压花画的制作方法及应用

干燥花是以真实植物为材料经过干燥、保色、加工整理而成的，可以久置不坏，是具有独特风格的一种观赏花材。它分为立体干燥花和平面干燥花。平面干燥花也称为压花，是将自然界中的植物材料经脱水、保色、压制和干燥处理而成的植物制品。将平面干燥花材按花的色彩、形态、质感、韵律等特点适宜搭配，可构成一幅幅生动活泼的压花艺术画。     

昆明地区野生禾本科干燥花资源的调查研究

对昆明地区的4区8县进行了野生禾本科干燥花资源的调查研究,共筛选出26属33种干燥花资源,并对昆明地区的重要干燥花资源进行了简要评述。     

气调贮藏不同气体成分对网纹甜瓜品质的影响

研究了在１５℃条件下，不同Ｏ２ 浓度（２１％ 、１０％ 、５％ 、３％ ）和不同ＣＯ２ 浓度（０、２％ 、５％ 、１０％ 、１５％ ）的气体组合贮藏网纹甜瓜对其品质的影响，结果表明：在１５℃条件下网纹甜瓜气调贮藏最佳的气体条件是Ｏ２ 浓度３％ ～５％ ，ＣＯ２ 浓度０～５％ ；３％ 的低Ｏ２ 对保持网纹甜瓜的硬度具有重要作用。     

干燥花干燥技术研究进展

干燥花是一种源于自然、同时兼备人造花的长久保存性和鲜花的自然真实性的观赏花材。在干燥花制备工艺过程中,干燥是一个极为重要的环节,直接关系到干燥花的自然性、真实性、长久性。本文综述了近10年来国内外干燥花干燥技术的研究进展,分别叙述传统、现代干燥技术的优缺点,并进行对比分析,指出了现有技术存在的问题和发展的方向,旨在为干燥花干燥工艺的深入研究和规模化生产提供参考。     

气调贮藏不同气体成分对网纹甜瓜品质的影响

研究了在１５℃条件下，不同Ｏ２浓度（２１％、１０％、５％、３％）和不同ＣＯ２浓度（０．２％、５％、１０％１５％）的气体组合贮藏网纹甜瓜对其品质的影响，结果表明：在１５℃条件下网纹甜瓜气调贮藏最佳的气体条件是Ｏ２浓度３％～５％，ＣＯ２浓度０～５％；３％的低Ｏ２对保持网纹甜瓜的硬度具有重要作用。     

硝酸钾在西瓜上的应用效果

土施不同量的ＫＮＯ３，喷施不同浓度的ＫＮＯ３以及在西瓜不同生育期喷施１％ＫＮＯ３，结果表明：对改变西瓜生育期无影响，能明显促进西瓜的营养生长和提高座果率，并能改善西瓜品质。ＫＮＯ３土施用量以５ｋｇ／６６７ｍ＾２为佳，叶面喷施浓度以１％￣１．５％为好。     

雀麦干燥染色技术研究

通过对雀麦茎杆及果穗进行4种不同染料的染色处理,探讨影响干花着色效果的因素及染色技术。结果表明:抽穗后2～3周内并且经过漂白的雀麦染色效果最佳;染料橙黄G的染色速度最快;染料橙黄G和碱性品红与干燥花的结合能力较强。     

乙酰苯胺和2-氯苯磺酰胺诱导IN2-2启动子在芥菜中的表达

分别用不同浓度(50、100、200mg·L-1)的除草剂安全剂类似化合物乙酰苯胺和2-氯苯磺酰胺处理IN2-2::GUS转基因芥菜。结果显示:乙酰苯胺和2-氯苯磺酰胺处理后,IN2-2启动子在芥菜根、叶、花器官的花萼、花瓣、雄蕊及花粉中表达,但不在胚珠中表达。100mg·L-1乙酰苯胺以及50mg·L-12-氯苯磺酰胺适合用于在芥菜中调控IN2-2启动子的表达,乙酰苯胺较2-氯苯磺酰胺诱导表达所需时间更短。高浓度的2-氯苯磺酰胺影响芥菜种子发芽及生长发育,并且明显抑制IN2-2启动子的表达活性。4℃低温胁迫诱导IN2-2启动子在幼苗叶片中表达,IN2-2启动子轻微受150mmol·L-1Na Cl胁迫表达,但不受重金属Gu~(2+)离子的诱导表达。     

H2O2 和Ca2+ 对受低温胁迫香蕉幼苗抗冷性的影响

 研究表明, 香蕉幼苗喷施H₂O₂ 或CaCl₂ 或二者结合使用, 能提高其POD 活性, 降低膜渗漏率, 增加可溶性糖含量及减缓叶绿素降解, 从而提高抗寒力, 二者结合表现出协同效应。H₂O₂ 对提高POD活性, 降低膜渗漏率和减缓Chl. b 降解的效果优于CaCl₂ ; CaCl₂ 则在提高可溶性糖含量和减少Chl. a 降解方面占优。相关性分析表明, POD 活性与膜渗漏率的负相关性达到显著水平, 高POD 活性有利于减少低温下膜渗漏。     

Roles of SARS-CoV-2 receptor ACE2 in various diseases

It is well know that severe acute respiratory syndrome coronavirus 2 （SARS-CoV-2） can enter host cells through angiotensin-converting enzyme 2 （ACE2）, which is a key step in the development of coronavirus disease 2019 （COVID-19）. At the same time, ACE2 is expressed in a variety of tissues and regulates many biological functions, including inflammation, cell proliferation and oxidative stress, as a key negative regulator of the renin-angiotensin system. It plays an important role in the changes of physiological functions and pathological diseases of multiple systems and organs. Therefore, in addition to COVID-2019, ACE2 is also a potential therapeutic target for the diseases such as acute lung injury, cardiovascular diseases, cancer, and kidney diseases. This article reviews the mechanism of ACE2 in the above diseases and new strategies for therapeutic application.     

过氧化氢脲在食用菌生产中应用研究

过氧化氢脲可有效抑制空气中的微生物．其效果与过氧乙酸及甲醛相近。还可拌入培养料中使用，效果很好。     

L-carnitine inhibits H2O2-induced rat cardiomyocyte apoptosis through suppressing Ca2+/CaMKII signaling pathway

AIM：To examine the inhibitory effect of L-carnitine on hydrogen peroxide (H2O2)-induced apoptosis of rat cardiomyocytes and to further explore the underlying mechanisms. METHODS：Primarily cultured neonatal rat myocardial cells were prepared and challenged by 200 μmol/L H2O2 to induce cell apoptosis. In order to evaluate the effects of Ca2+ chelator 1,2-bis(2-aminophenoxy)ethane-N, N, N′, N′-tetraacetic acid (BAPTA), calmodulin-dependent protein kinase II (CaMKII) inhibitor KN93 and L-carnitine on cell viability, apoptosis, resting intracellular free Ca2+ concentration (［Ca2+］i) and phospho-CaMKII (p-CaMKII) expression, these three agents were added 30 min or 1 h prior to H2O2 stimulation. Cell viability was measured by MTT assay and apoptosis was determined by flow cytomertry. The ［Ca2+］i was measured by laser confocal scanning. Cleaved caspase-3 and p-CaMKII expression was detected using Western blotting. RESULTS：Upon 200 μmol/L H2O2 stimulation for 12 h, cell viability decreased and apoptotic rate increased significantly compared with control．Pretreament with L-carnitine, BAPTA and KN93 significantly increased cell viability and decreased apoptosis．Furthermore, intracellular Ca2+ overload triggered by H2O2 could be greatly relieved by L-carnitine and BAPTA pretreatment, but not affected by KN93. H2O2-stimulated cleaved caspase-3 and p-CaMKII expression was also significantly inhibited by all these three agents. CONCLUSION：L-carnitine inhibits H2O2-induced rat cardiomyocyte apoptosis possibly via suppressing Ca2+/CaMKII signaling pathway.     

L-Carnitine inhibits H2O2-mediated NFATc3 nuclear translocation

AIM:To explore the effect of L-carnitine on nuclear factor of activated T-cells,cytoplasmic 3 (NFATc3) in cardiomyocytes under H2O2 stimulation. METHODS:Primary cultured neonatal rat myocardial cells were stimulated by H2O2 at concentration of 200 μmol/L for 12 h to induce oxidative stress injury. In treatment group, L-carnitine and cyclosporin A (CsA), a specific inhibitor of calcineurin (CaN), were administered 30 min prior to H2O2 stimulation. After treatment, total, cytoplasmic and nuclear NFATc3 protein levels were determined by Western blotting. The method of immunofluoresence was used to evaluate the distribution of NFATc3. RESULTS:H2O2 treatment produced no effect on the expression of total NFATc3, but caused its translocation from the cytosolic to nuclear compartment, which was greatly blunted by L-carnitine pretreatment. CONCLUSION: L-carnitine antagonized oxidative stress injury via alleviating NFATc3 nuclear translocation.