共查询到20条相似文献,搜索用时 46 毫秒
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[目的]研究智利小植绥螨(Phytoseiulus persimilis)雌成螨对新疆三种主要害螨土耳其斯坦叶螨(Tetranychus turkestani Ugarov et Nikolski)、截形叶螨(Tetranychus truncatctus Ehara)、朱砂叶螨(Tetranychus cinnabarinus)的捕食功能反应,以及智利小植绥螨个体之间捕食的干扰反应.[方法]在27℃条件下,采用实验生态学方法,用HoUingⅡ型圆盘方程进行拟合.[结果]智利小植绥螨雌成螨和若螨对的反应均属于HollingⅡ型.智利小植绥螨雌成螨的捕食能力(a/Th值)远大于若螨;智利小植绥螨雌成螨对土耳其斯坦叶螨的攻击率最高为0.665,对朱砂叶螨的攻击率最低为0.214;对土耳其斯坦叶螨的捕食能力最强为10.726,对朱砂叶螨的捕食能力最弱为3.754;智利小植绥螨的寻找效应随着天敌密度的增加而降低.[结论]智利小植绥螨对土耳其斯坦叶螨、截形叶螨、朱砂叶螨捕食作用均属于HollingⅡ型,其捕食作用同时受到猎物密度和自身密度的影响. 相似文献
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Identification of a T helper cell-derived lymphokine that activates resting T lymphocytes 总被引:3,自引:0,他引:3
A novel lymphokine with apparent molecular size of 10 to 12 kilodaltons is secreted from helper T cell clones within hours after cross-linking their T cell antigen-MHC (major histocompatibility complex) receptors (T3-Ti). This lymphokine, termed interleukin-4A (IL-4A), stimulates resting lymphocytes by binding to a surface component (or components) of the alternative T11 pathway and subsequently by inducing interleukin-2 (IL-2) receptors. The activation process is neither dependent on antigen specificities of the recruited population or the presence of macrophages. It appears, therefore, that IL-4A is a mediator involved in amplifying the T cell immune response. 相似文献
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The T cell receptor 总被引:51,自引:0,他引:51
The primary structure of T cell receptor proteins and genes is well understood. Immunologists are now trying to understand the properties of these interesting molecules. Evidence suggests that T cell alpha beta receptors recognize a complex of an antigen-derived peptide bound to one of the cell-surface products of the major histocompatibility complex (MHC) genes. It is likely that alpha beta receptors and MHC proteins have coevolved to have some affinity for each other. During T cell development in the thymus, cells bearing self-reactive receptors are deleted by the mechanisms of tolerance, and cells are preferentially allowed to mature if they bear receptors that will be able to recognize antigen plus self-MHC after they have become full-fledged T cells. Some explanations for these phenomena have been tested, but no satisfactory theory can yet be proposed to account for them. 相似文献
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Control of experimental autoimmune encephalomyelitis by T cells responding to activated T cells 总被引:21,自引:0,他引:21
T cell vaccination against experimental autoimmune disease is herein shown to be mediated in part by anti-ergotypic T cells, T cells that recognize and respond to the state of activation of other T cells. The anti-ergotypic response thus combines with the previously shown anti-idiotypic T cell response to regulate autoimmunity. 相似文献
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Proliferation of activated cytotoxic T lymphocytes (CTLs) that recognize foreign histocompatibility antigens is induced by interleukin-2, a potent immunoregulatory molecule originally described as T cell growth factor. Interleukin-2 (IL-2) is widely used to isolate and induce clonal expansion of CTLs for functional studies in vitro and in vivo. However, in studies with CTLs specific for class I and class II histocompatibility antigens, IL-2 rapidly downregulated the lytic activity of some class II-specific CTLs in a time- and dose-dependent manner. Lytic activity of L3T4+ CTLs specific for the murine class II antigen I-Ek was repeatedly up- and downregulated in vitro by alternate exposure to specific (alloantigen) and nonspecific (recombinant IL-2) signals, respectively. These results demonstrate that some CTLs modulate their functional property (cytolysis) while undergoing IL-2-driven cell proliferation without loss of antigen specificity or ability to revert to a lytic phenotype. 相似文献
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Self-nonself discrimination by T cells 总被引:28,自引:0,他引:28
The alpha beta T cell receptor (TCR) recognizes antigens that are presented by major histocompatibility complex (MHC)-encoded cell surface molecules by binding to both the antigen and the MHC molecules. Discrimination of self from nonself antigens and MHC molecules is achieved by negative and positive selection of T cells in the thymus: potentially harmful T cells with receptors that bind to self antigens plus self MHC molecules are deleted before they can mount immune responses. In contrast, the maturation of useful T cells with receptors that bind foreign antigens plus self MHC molecules requires the binding of their receptor to MHC molecules on thymic epithelium in the absence of foreign antigen. The binding of the TCR to either class I or class II MHC molecules directs differentiation of the selected cells into either CD4-8+ (killer) or CD4+8- (helper) T cells, respectively. 相似文献
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