共查询到20条相似文献,搜索用时 15 毫秒
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Kim L Morley PS McCluskey BJ Mumford EL Swenson SL Salman MD 《Journal of the American Veterinary Medical Association》2000,216(9):1399-1404
OBJECTIVE: To report clinical and serologic findings in horses with oral vesicular lesions that were consistent with vesicular stomatitis (VS) but apparently were not associated with VS virus (VSV) infection. DESIGN: Serial case study. ANIMALS: 8 horses. PROCEDURE: Horses were quarantined after appearance of oral lesions typical of VS. Severity of clinical signs was scored every 2 to 5 days for 3 months. Serum samples were tested for antibodies by use of competitive ELISA (cELISA), capture ELISA for IgM, serum neutralization, and complement fixation (CF). Virus isolation was attempted from swab specimens of active lesions. RESULTS: 2 horses with oral vesicular lesions on day 1 had antibodies (cELISA and CF) against VSV; however, results of CF were negative by day 19. Five of the 6 remaining horses were seronegative but developed oral lesions by day 23. Virus isolation was unsuccessful for all horses. CONCLUSIONS AND CLINICAL RELEVANCE: Horses were quarantined for 75 days in compliance with state and federal regulations. However, evidence suggests that oral lesions were apparently not associated with VSV infection. The occurrence in livestock of a vesicular disease that is not caused by VSV could confound efforts to improve control of VS in the United States and could impact foreign trade. Vesicular stomatitis is of substantial economic and regulatory concern. 相似文献
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Swine vesicular disease (SVD) is a notifiable viral disease of pigs included on the Office International des Epizooties List A. The first outbreak of the disease was recognized in Italy in 1966. Subsequently, the disease has been reported in many European and Asian countries. The causative agent of the disease is SVD virus which is currently classified as a porcine variant of human coxsackievirus B5 and a member of the genus enterovirus in the family picornaviridae. From a clinical point of view, SVD is relatively unimportant, rarely causing deaths and usually only a minor setback to finishing schedules. However, the clinical signs which it produces are indistinguishable from those caused by foot-and-mouth disease, and its presence prevents international trade in pigs and pig products. This article reviews recent findings on all aspects of the virus and the disease which it causes. 相似文献
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J T Done 《The Veterinary record》1992,131(17):399-400
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兔传染性水疱口炎是由水疱性口炎病毒引起的兔的一种急性传染病。其特征是口腔黏膜发生水疱性炎症并大量流涎,故又称“流涎病”。水疱性口炎病毒属于弹状病毒科水疱性病毒属,病毒粒子呈子弹状或圆柱性,有囊膜,大小为176nm×69nm。该病毒可在7~13日龄鸡胚绒毛尿囊膜上及尿囊内生长,1~2d内使鸡胚死亡。另外,该病毒能在多种细胞中繁殖并产生血凝素。在4℃条件下该病毒能存活30d;-20℃能长期存活。2%氢氧化钠、1%福尔马林能在数分钟杀死该病毒;加热至60℃或直射阳光下,病毒很快死亡。该病毒主要存在于病兔的水疱液、水疱皮、口腔黏膜坏死组 相似文献
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H Liebermann P Schulze R Riebe R Koitzsch 《Archiv fuer experimentelle veterinaermedizin》1976,30(3):433-440
Titration of SVDV on primary pig kidney cell cultures revealed a plating efficiency of less than or equal to 0,9 X 10(-3). Concentration and purification of the SVD-Virus propagated on pig kidney cell cultures were done by chloroform treatment, adsorption, differential- and density gradient centrifugation. The following physical parameters were found: SVDV is an isometrical RNA-virus having a diameter of 25,1 +/- 1,0 nm. It is resistent to the action of chloroform, ether and pH. The virus has a sedimentation coefficient of 156 +/- 3S and a bouyant density in CsCl of 1,33 +/- 0,01 g/ml. Within the family of picornaviruses the SVDV belongs to the subgroup of enteroviruses and can be distinguished from the foot-and-mouth disease virus by the difference in pH-sensitivity and bouyant density in CsCl. 相似文献