The aftershock signature of supershear earthquakes

Recent studies show that earthquake faults may rupture at speeds exceeding the shear wave velocity of rocks. This supershear rupture produces in the ground a seismic shock wave similar to the sonic boom produced by a supersonic airplane. This shock wave may increase the destruction caused by the earthquake. We report that supershear earthquakes are characterized by a specific pattern of aftershocks: The fault plane itself is remarkably quiet whereas aftershocks cluster off the fault, on secondary structures that are activated by the supershear rupture. The post-earthquake quiescence of the fault shows that friction is relatively uniform over supershear segments, whereas the activation of off-fault structures is explained by the shock wave radiation, which produces high stresses over a wide zone surrounding the fault.     

Laboratory earthquakes along inhomogeneous faults: directionality and supershear

We report on the experimental observation of spontaneously nucleated ruptures occurring on frictionally held bimaterial interfaces with small amounts of wave speed mismatch. Rupture is always found to be asymmetric bilateral. In one direction, rupture always propagates at the generalized Rayleigh wave speed, whereas in the opposite direction it is subshear or it transitions to supershear. The lack of a preferred rupture direction and the conditions leading to supershear are discussed in relation to existing theory and to the earthquake sequence in Parkfield, California, and in North Anatolia.     

Observation of long supershear rupture during the magnitude 8.1 Kunlunshan earthquake

The 2001 Kunlunshan earthquake was an extraordinary event that produced a 400-km-long surface rupture. Regional broadband recordings of this event provide an opportunity to accurately observe the speed at which a fault ruptures during an earthquake, which has important implications for seismic risk and for understanding earthquake physics. We determined that rupture propagated on the 400-km-long fault at an average speed of 3.7 to 3.9 km/s, which exceeds the shear velocity of the brittle part of the crust. Rupture started at sub-Rayleigh wave velocity and became supershear, probably approaching 5 km/s, after about 100 km of propagation.     

A radically different mechanism for S-adenosylmethionine-dependent methyltransferases

Methylation of small molecules and macromolecules is crucial in metabolism, cell signaling, and epigenetic programming and is most often achieved by S-adenosylmethionine (SAM)-dependent methyltransferases. Most employ an S(N)2 mechanism to methylate nucleophilic sites on their substrates, but recently, radical SAM enzymes have been identified that methylate carbon atoms that are not inherently nucleophilic via the intermediacy of a 5'-deoxyadenosyl 5'-radical. We have determined the mechanisms of two such reactions targeting the sp(2)-hybridized carbons at positions 2 and 8 of adenosine 2503 in 23S ribosomal RNA, catalyzed by RlmN and Cfr, respectively. In neither case is a methyl group transferred directly from SAM to the RNA; rather, both reactions proceed by a ping-pong mechanism involving intermediate methylation of a conserved cysteine residue.     

A mechanical trigger for the trot-gallop transition in horses.

It is widely thought that animals switch gaits at speeds that minimize energetic cost. Horses naturally switched from a trot to a gallop at a speed where galloping required more energy than trotting, and thus, the gait transition actually increased the energetic cost of running. However, by galloping at this speed, the peak forces on the muscles, tendons, and bones, and presumably the chance of injury, are reduced. When the horses carried weights, they switched from a trot to a gallop at a lower speed but at the same critical level of force. These findings suggest that the trot-gallop transition is triggered when musculoskeletal forces reach a critical level.     

A powerful chiral counterion strategy for asymmetric transition metal catalysis

Traditionally, transition metal-catalyzed enantioselective transformations rely on chiral ligands tightly bound to the metal to induce asymmetric product distributions. Here we report high enantioselectivities conferred by a chiral counterion in a metal-catalyzed reaction. Two different transformations catalyzed by cationic gold(I) complexes generated products in 90 to 99% enantiomeric excess with the use of chiral binaphthol-derived phosphate anions. Furthermore, we show that the chiral counterion can be combined additively with chiral ligands to enable an asymmetric transformation that cannot be achieved by either method alone. This concept of relaying chiral information via an ion pair should be applicable to a vast number of metal-mediated processes.     

A common explosion mechanism for type Ia supernovae

Type Ia supernovae, the thermonuclear explosions of white dwarf stars composed of carbon and oxygen, were instrumental as distance indicators in establishing the acceleration of the universe's expansion. However, the physics of the explosion are debated. Here we report a systematic spectral analysis of a large sample of well-observed type Ia supernovae. Mapping the velocity distribution of the main products of nuclear burning, we constrain theoretical scenarios. We find that all supernovae have low-velocity cores of stable iron-group elements. Outside this core, nickel-56 dominates the supernova ejecta. The outer extent of the iron-group material depends on the amount of nickel-56 and coincides with the inner extent of silicon, the principal product of incomplete burning. The outer extent of the bulk of silicon is similar in all supernovae, having an expansion velocity of approximately 11,000 kilometers per second and corresponding to a mass of slightly over one solar mass. This indicates that all the supernovae considered here burned similar masses and suggests that their progenitors had the same mass. Synthetic light-curve parameters and three-dimensional explosion simulations support this interpretation. A single explosion scenario, possibly a delayed detonation, may thus explain most type Ia supernovae.     

A mechanism for social selection and successful altruism

Within the framework of neo-Darwinism, with its focus on fitness, it has been hard to account for altruism behavior that reduces the fitness of the altruist but increases average fitness in society. Many population biologists argue that, except for altruism to close relatives, human behavior that appears to be altruistic amounts to reciprocal altruism, behavior undertaken with an expectation of reciprocation, hence incurring no net cost to fitness. Herein is proposed a simple and robust mechanism, based on human docility and bounded rationality that can account for the evolutionary success of genuinely altruistic behavior. Because docility-receptivity to social influence-contributes greatly to fitness in the human species, it will be positively selected. As a consequence, society can impose a "tax" on the gross benefits gained by individuals from docility by inducing docile individuals to engage in altruistic behaviors. Limits on rationality in the face of environmental complexity prevent the individual from avoiding this "tax." An upper bound is imposed on altruism by the condition that there must remain a net fitness advantage for docile behavior after the cost to the individual of altruism has been deducted.     

A general mechanism for network-dosage compensation in gene circuits

Coping with variations in network dosage is crucial for maintaining optimal function in gene networks. We explored how network structure facilitates network-level dosage compensation. By using the yeast galactose network as a model, we combinatorially deleted one of the two copies of its four regulatory genes and found that network activity was robust to the change in network dosage. A mathematical analysis revealed that a two-component genetic circuit with elements of opposite regulatory activity (activator and inhibitor) constitutes a minimal requirement for network-dosage invariance. Specific interaction topologies and a one-to-one interaction stoichiometry between the activating and inhibiting agents were additional essential elements facilitating dosage invariance. This mechanism of network-dosage invariance could represent a general design for gene network structure in cells.     

A formation mechanism for catalytically grown helix-shaped graphite nanotubes

The concept of a spatial-velocity hodograph is introduced to describe quantitatively the extrusion of a carbon tubule from a catalytic particle. The conditions under which a continuous tubular surface can be generated are discussed in terms of this hodograph, the shape of which determines the geometry of the initial nanotube. The model is consistent with all observed tubular shapes and explains why the formation process induces stresses that may lead to "spontaneous" plastic deformation of the tubule. This result is due to the violation of the continuity condition, that is, to the mismatch between the extrusion velocity by the catalytic particle, required to generate a continuous tubular surface, and the rate of carbon deposition.     

A structural mechanism for MscS gating in lipid bilayers

The mechanosensitive channel of small conductance (MscS) is a key determinant in the prokaryotic response to osmotic challenges. We determined the structural rearrangements associated with MscS activation in membranes, using functorial measurements, electron paramagnetic resonance spectroscopy, and computational analyses. MscS was trapped in its open conformation after the transbilayer pressure profile was modified through the asymmetric incorporation of lysophospholipids. The transition from the closed to the open state is accompanied by the downward tilting of the transmembrane TM1-TM2 hairpin and by the expansion, tilt, and rotation of the TM3 helices. These movements expand the permeation pathway, leading to an increase in accessibility to water around TM3. Our open MscS model is compatible with single-channel conductance measurements and supports the notion that helix tilting is associated with efficient pore widening in mechanosensitive channels.     

A bond-fluctuation mechanism for stochastic switching in wired molecules

Stochastic on-off conductivity switching observed in phenylene-ethynylene oligomers has been explained in terms of changes in ring conformations, or electron localization, or both. We report the observation of stochastic on-off switching in the simplest of wired molecules: octanedithiol, decanedithiol, and dodecanedithiol bonded on an Au(111) surface. Stochastic switching was observed even when a top gold contact was pressed on by a conducting atomic force microscope tip at constant force. The rate of switching increased substantially at 60 degrees C, a temperature at which these films are commonly annealed. Because such switching in alkanethiols is unlikely to be caused by internal molecular electronic changes and cannot be fully accounted for by breaking of the top contact, we argue that the cause is the well-known mobility of molecules tethered to gold via a thiol linkage.     

裂缝对渗流场影响的数值模拟方法研究

【目的】研究裂缝对渗流场影响的数值模拟方法,为岩石、混凝土等材料裂缝渗流场的变化研究提供支持。【方法】在等效连续介质模型框架下提出渗透系数张量修正法,并基于流量等效原理推导出修正单元的渗透系数张量矩阵。应用有限元分析软件,分别建立裂缝位于构件边界和构件内部时的分析模型,应用渗透系数张量修正法得到的渗透系数张量表达式定义裂缝单元的渗透特性,将模拟所得渗流场与裂缝真实存在的数值分析结果进行比较,并通过改变修正单元的尺寸分析渗透系数张量修正法对单元尺寸的依赖性。【结果】修正单元为10倍裂缝宽度时,与精确解相比,各分析模型所得渗流量及相应断面孔隙水压的误差均小于0.5%。就单元尺寸依赖性而言,为满足工程精度要求,从渗流量角度分析,修正单元的尺寸应不大于600倍裂缝宽度;从孔隙水压角度分析,修正单元的尺寸应不大于200倍裂缝宽度。【结论】所建立的等效处理方法合理可行,当修正单元的尺寸不大于200倍裂缝宽度时,无论是渗流量还是孔隙水压,该处理方法均能满足工程精度要求。