雏鹅氨气中毒的诊治

2009年2月10日,某养鹅户李某从江都购进扬州雏鹅1000只,在一个较为低短的面积60余平方米的塑料薄膜棚内育雏,棚高2米,长15米,宽8米,棚内分成10格,每格育雏100只,没有门窗,四面塑料薄膜下垂到地面,用砖头和本板压紧,基本上不透气,棚内装有6只250W的红外线取暖灯。     

雏鹅霉菌性肺炎并发大肠杆菌的诊治报告

武威市某养殖户张某新购进鹅苗8 000羽。10月中旬,陆续出现喘气、闭目昏睡、腹泻、肺脏表面黄白色结节及心包炎等症状。经痢菌净和氟苯尼考等药物治疗,疗效不显著。经流行病学调查、临床症状、病理变化及实验室诊断,确诊为霉菌性肺炎并发大肠杆菌混合感染。采取中西医结合治疗等综合性防治措施,疫情得到有效控制。现将诊疗情况作一报告,以供广大养殖户参考。     

雏鹅疑似食盐中毒的诊治

本县淤上乡某村民从福建莆田引进雏鹅80余羽，已饲养16d，雏鹅网架舍饲，栏舍内通风良好，饮水清洁，因本地没有雏鹅用专用料，一直以小鸡料代用，近来时有雏鹅发生死亡，每日1～2羽不等，已连续死亡13羽，病鹅表现饮水量激增，口鼻流涎，并频频伸颈摇头，精神萎顿，两肢无力，不愿行走，出现水样腹泻，体温无高温表现，病程较长，经过2～3d后倒地死亡。     

雏鹅马杜拉霉素中毒的诊治

1发病情况某个体养殖户饲养18日龄雏鹅214只。2005年7月份,为防治球虫病,用马杜拉霉素拌料饲喂,但由于称量时失误,将10克马杜拉霉添加到200千克料(纯品添加剂量应为5毫克/千克)中,次日雏鹅出现中毒症状,并不断有雏鹅死亡,3天内共死亡雏鹅165只,死亡率高达78%。2临床症状中毒初雏鹅多表现为神经症状,兴奋不安,乱飞乱跑,口流黏液,头向后仰;后期表现为精神沉郁,食欲部分或全部废绝,部分病鹅拉白色或浅绿色稀便。3剖检变化病死鹅的口、鼻内有黏液,气管出血,腺胃黏膜脱落,心外膜充血,肝脏淤血,小肠黏膜弥漫性出血,肾淤血。4治疗无特效解毒药,采用…     

雏鹅氨气中毒的诊治

一发病情况 本镇李某购进太湖雏鹅1000只,在一面积60余平方米的塑料薄膜棚内育雏。该棚没有门窗,四面塑料薄膜下垂到地面,用砖头和木板压紧,内装有6只250W红外线灯作取暖用。雏鹅在11日龄前一切正常,12日龄时有4只死亡,以后每天都有死亡,至3月5日现场检查发现,棚内共死亡56只。     

雏鹅霉菌性肺炎的诊治

本地 2 0 0 0年雏鹅霉菌性肺炎较往年增多 ,据统计 ,6月 2 0日至 7月 1 0日 ,本市兽医院门诊部共诊治雏鹅霉菌性肺炎 4 3群 ,发病率、死亡率相对较高 ,使养禽户蒙受了一定的经济损失 ,现将诊治情况简述如下 :1　发病情况　2 0 0 0年 6月 2 0日至 7月 1 0日 ,来门诊部就诊且有记载的雏鹅共 85群 ,其中患霉菌性肺炎的有 4 3群 ,约占 50 %,最小发病日龄为 1 0日龄 ,最大为 4 0日龄。当出现精神不振、呼吸困难等症状时 ,用抗菌药物治疗均无明显效果 ;发病后 ,每日都有死亡 ,各群平均死亡率为 1 0 %～ 2 0 %。2　临床症状　 发病雏鹅呼吸困难 ,喘…     

雏鹅大肠杆菌病的诊治

鹅大肠杆菌病是由致病性大肠杆菌引进的传染病,各种日龄鹅都可发病.随着养鹅业的迅猛发展,鹅病也越来越多,近两年来笔者诊治了几群雏鹅发生大肠杆菌病而致大量死亡的病例,现将诊治过程报告如下.     

雏鹅霉菌性肺炎的诊治

近年来辽宁省锦州地区养鹅生产发展迅速,除了一些病毒病严重影响养鹅业外,霉菌病也困绕着养鹅户。2005年进入6月以来雨水较多,雏鹅霉菌性肺炎较往年增多,发病率和死亡率相对较高,养鹅户遭受一定的经济损失,现将该病诊治情况介绍如下。1发病情况2005年6月上旬,笔者先后接待了4～     

雏鹅黄曲霉毒素中毒的诊治

黄曲霉毒素中毒是畜禽的一种中毒性疾病,是由于畜禽食入被黄曲霉或寄生曲霉产生的黄曲霉毒素污染的饲料引起。这两种霉菌能产生一组毒性极强的代谢产物,其中黄曲霉毒素B1（AFTB1）的毒性最大。黄曲霉毒素主要对肝脏造成损害,可引起肝细胞变性、坏死。临床上以肝功能障碍、全身出血、消化机能紊乱、腹水、神经症状等为特征。     

雏鹅霉菌性肺炎的诊治体会

2006年5～6月,我市发生雏鹅霉菌性肺炎病鹅死亡率从20%～70%不等,造成很大的经济损失,本人对发病鹅进行了诊治,并实地跟踪调查病情及防治效果,现综述如下。     

家禽霉菌毒素中毒症

本文从临床症状、病理变化等方面概述了黄曲霉毒素、麦角、单端孢霉毒素、串珠镰刀霉菌毒素、赭曲霉毒素、卵孢霉素、红青霉素等引起的家禽中毒。     

猪霉菌毒素中毒的新概念

霉菌毒素是真菌的次级代谢产物,可以降低畜禽生产性能和改变新陈代谢(Wannemacher等,1991)。动物采食被霉菌毒素污染的饲料而引发的病     

鹅艾美球虫对雏鹅的致病性及病理学研究

用纯种鹅艾美球虫孢子化卵囊分别人工感染9日龄和18日龄雏鹅,进行致病性与病理学研究.以0.01×104个/只、0.1×104个/只、1.0 × 104个/只剂量感染9日龄雏鹅,死亡率分别为0、10%和60%;以0.5×104个/只、3.0×104个/只剂量感染18日龄雏鹅,死亡率分别为20%和75%.雏鹅发病的临床症状和死亡鹅的病理变化基本一致,潜伏期5 d～5.5 d,病程3.5 d～4 d.病变主要在小肠下段,尤其是空肠后段与回肠前段最严重,呈急性出血-坏死性或出血-卡他性肠炎.大量配子体或卵囊寄生在肠绒毛基部尤其是肠腺上皮细胞中,引起肠黏膜上皮坏死、脱落以及肠壁水肿、出血和炎性细胞浸润等.结果表明:鹅艾美球虫对雏鹅有很强的致病性;致病性主要发生在配子生殖阶段,是由大量的配子体或卵囊在肠腺上皮细胞内发育破坏了肠道黏膜组织结构与功能所致.     

The feeding of penicillin to goslings

A total of 180 crossbred Chinese/Emden goslings were either housed indoors for 8 weeks or kept inside for 4 weeks and then allowed to graze. Within each experimental group the birds were fed on diets with or without added penicillin. The antibiotic was included at either 55 mg/kg or 110 mg/kg.

Only when the goslings were fed throughout on a diet supplemented with penicillin at an inclusion rate of 110 mg/kg was there significant growth promotion.

No significant differences in the weights of the liver, gizzard, small intestine or caeca, or in the average weights of 1 cm lengths of the small intestines and caeca could be detected.     

鹅源沙门菌分离鉴定及系统进化分析

从发病死亡雏鹅尸体所采集的病料中分离获得3株细菌,并进行了培养特性、形态特征、生化特性、药物敏感性、致病性等表观性状的研究,以及对其系统发育进行分析。16S rRNA基因测序表明,菌株RCg-1,菌株RCg-3与肠炎沙门菌同源性最高,达到99.2%-99.4%;3株分离菌2.5×107CFU能够导致试验组雏鹅和小鼠100%死亡。3株分离菌对头孢噻吩、头孢曲松、先锋霉素ⅴ、恩诺沙星最敏感,对乙酰螺旋霉素、青霉素耐药。     

Nitrogen requirement for maintenance in Yangzhou goslings

1. Experiments were conducted to determine the nitrogen (N) requirement for maintenance (N(m)) in Yangzhou goslings. 2. At 56 d old, 18 birds were divided into three equal groups and fed on diets with equal metabolisable energy (ME) and different contents of crude protein (CP; low, medium and high) in an N balance test. N(m) was estimated from the relationship between N output (N(E)) and N intake (N(I)). Following the above N balance test, 16 goslings at the same age were used in a N balance test with an N-free diet. The birds were divided into 4 equal groups and offered an N-free diet at intakes of 90, 70, 50 and 0 g per d, respectively. 3. The estimated N(m) was 240 mg/kg BW(0.75) per d in the N balance test. The result from the N-free diet trial gave a value of 244 mg/kg BW(0.75) per d, confirming the result of the N balance test. The lower intake of N-free diet resulted in more N(E), suggesting that protein catabolism may occur in the body of birds to meet N(m) when dietary N(I) was very low. 4. It was concluded that the N(m) of Yangzhou goslings was about 240 mg/kg BW(0.75).     

一起雏鹅小鹅瘟的诊断与治疗

小鹅瘟是雏鹅的一种急性或亚急性败血症,依感染时的年龄,可分为最急性、急性和亚急性。15日龄以内的锥鹅发病常为急性,无明显前驱症状,一经发现即极度衰弱,倒地乱划,抽搐死亡,死亡率高达60％以上。2008年6月9日,灵武市东塔镇某鹅场6日龄雏鹅出现大批死亡,发病率52％,死亡率100％。采用特异性治疗、补液治疗等措施后,病情得到基本控制。现将情况报告如下。     

Large-scale mitochondrial DNA deletions in weak goslings

1. Evidence has accumulated in mammals to support the idea that mitochondrial DNA (mtDNA) deletions and mutations might contribute to ageing and reproductive failure. White Roman geese were monitored to evaluate the effect of large-scale deletions of mtDNA in an avian species. 2. A total of 340 samples from 114 dead embryos, 111 weak goslings, and 115 normal goslings were used in this experiment. The regions of these two large-scale mtDNA deletions, ΔmtDNA6829 and ΔmtDNA6992, were between the COI and ND5 genes. A 3·6% (4 out of 111) positive sample was detected in the weak goslings. In contrast, no large-scale mitochondrial DNA deletions were detected in either the dead embryos (0 out of 114) or the normal goslings (0 out of 115). 3. Large-scale mtDNA deletions may be a factor causing weak goslings.     

Viral tracheitis in goslings in Saskatchewan.

Up to 12% mortality occurred in goslings between 4 and 11 days of age in two sequential hatches from a single breeder flock. Respiratory signs were noted before the birds died. The most consistent necropsy finding was a white opaque plug of fibrin and cellular debris in the trachea. The tracheal epithelium was hyperplastic and metaplastic with intranuclear inclusion bodies present in superficial cells. An adenovirus was isolated in both chicken and goose embryo liver cells.