八种盲蝽酯酶同工酶的比较研究

采用聚丙烯酰胺凝胶垂直板电泳法,分析了盲蝽科的绿丽盲蝽[Lygocoris (Apolygus) lucorum(Meyer-Dur)]、三点盲蝽(Adelphocoris taeniphorus Rruter)、黑食蚜盲蝽(Deraeocoris punctulastus Fall)、黑肩绿盲蝽(Cyrtorrhinus lividipennis Reuter)、条赤须盲蝽(Trigonotylus coelestialium Kirkaldy)、中黑苜蓿盲蝽(Adelphocoris suturalis Jak.)、甘薯跳盲蝽(Halticus minutus Reuter)和苜蓿盲蝽(Adelphocoris lineolatus Goeze)成虫的酯酶同工酶谱.结果发现8种盲蝽的同工酶谱存在明显的差异,并具有各自的特征酶谱.最后,探讨了酯酶同工酶电泳法鉴别盲蝽科昆虫种类及其在昆虫种属鉴定、遗传变异、系统发育等方面的可行性.     

Blue-green algae: why they become dominant

The injection of carbon dioxide and the addition of nitrogen and phosphorus to a lake population dominated by blue-green algae results in a rapid shift to dominance by green algae. The basis for the change and its implications are discussed.     

盲蝽卵的扫描电镜观察研究(I)(半翅目:盲蝽科)

记述了植食性盲蝽肉桂泡盾盲蝽PseudodoniellachinensisZheng、八角泡盾盲蝽Pseudodoniellatypica(China&Carvalho)、苜蓿盲蝽Adephocorislineolatus(Geoze)及淡缘厚盲蝽EurystyluscostalisStl4种卵的形态、细微结构及生物学记录.利用扫描电镜对上述种类卵进行了观察、比较.首次揭示出这4种卵的卵壳表面细微结构,卵前极呼吸角形态特征及细微构造.     

Triassic-jurassic tetrapod extinctions: are they real?

Terrestrial vertebrate fossils show that part of the Newark supergroup of the eastern United States, all of the Glen Canyon group of the southwestern United States, and the Upper Stormberg group of southern Africa are Early Jurassic. This new correlation demonstrates that the supposed widespread tetrapod extinction at the Triassic-Jurassic boundary is an artifact of spurious correlation.     

Schizophrenia: diverse approaches to a complex disease

Schizophrenia is a debilitating mental illness that affects 1% of the population. Despite intensive study, its molecular etiology remains enigmatic. Like many common diseases, schizophrenia is multifactorial in origin, with both genetic and environmental contributions likely playing an important role in the manifestation of symptoms. Recent advances based on pharmacological studies, brain imaging analyses, and genetic research are now converging on tantalizing leads that point to a central role for several neurotransmitters, including dopamine, glutamate, and serotonin, that may interface with neurodevelopmental defects reflecting disease-related genetic aberrations. Here, we provide a brief overview of the parallel approaches being used to identify the molecular causes of schizophrenia and discuss possible directions for future research.