鸡传染性喉气管炎防治

鸡传染性喉气管炎是由传染性喉气管炎病毒(ILTV)引起的一种急性上呼吸道传染病,常表现呼吸困难、产蛋鸡产蛋下降和较高的死亡率[1].易感鸡群一旦感染此病,则迅速传开,感染率达90%以上,病死率平均在10%~20%,给养鸡业造成很大的经济损失[2].其症状和病变极易与如禽流感、新城疫、传染性支气管炎等一些常见的呼吸道疾病相混淆,给该病的确诊带来了一定的难度,需要借助实验室方法才能确诊.     

鸡传染性喉气管炎防治

正鸡传染性喉气管炎是鸡的一种急性高度接触性传染病,其由疱疹病毒引起,危害较严重,可以引起鸡大批发病、死亡及造成幼鸡生长停滞、蛋鸡产蛋量下降。病毒主要存在于病鸡的气管组织及渗出物中。病毒最适宜在鸡胚中增殖,死亡胚体变小,鸡胚绒毛尿囊膜增生和坏死,形成混浊的、散在的,边缘隆起,中心凹陷的痘样坏死灶。病毒对乙醚、氯仿等脂溶剂敏感,对外界环境抵抗力不强。1流行病学本病主要危害鸡,各年龄及品种的鸡均可感     

鸡传染性喉气管炎的防治

我县13个乡镇的社员养鸡户,1988年共饲养蛋、肉鸡56万只,于10～11月份(当时的鸡群普遍为5～8月龄)暴发了鸡传染性喉气管炎,共死亡鸡112,000只,死亡率为20％。疫情发生后,我们组织发病较为严重的6个乡镇畜牧兽医站进行了详细的实地调查,共调查81个自然村,1500个养鸡户,调查户共养鸡14,956只,发病12,236只,     

鸡传染性喉气管炎的防治

传染性喉气管炎(ILT)是由A型疱疹病毒引起的鸡的一种急性呼吸道传染病。1936年由Beaudette首次证明其病原是一种滤过性病毒。目前此病在许多国家广泛流行，是危害养鸡业的重要疾病，近几年来，在我国一些养鸡集中的地区，ILT引起产蛋鸡产蛋下降和鸡群的高死亡率，产蛋率一般下降10％～50％。     

鸡传染性喉气管炎的防治

鸡传染性喉气管炎多流行于秋末、冬春季节,以5～8月龄的青年鸡最易感,雏鸡较少发生。感染途径是上呼吸道,主要通过带毒的鸡污染饲料、饮水、用具、羽毛和垫草而传播,感染率可达90%以上。在鸡舍通风不良、潮湿污秽、鸡群密度过大、饲料中缺乏维生素和蛋白质等情况下,常可诱发该病。病状:该病潜伏期为4～12d。最明显的症状是病鸡突然发生咳嗽和气喘。产蛋率明显下降或停止,并发出喘鸣声。鼻孔有分泌物,呼吸极度困难,并可咳出带血黏液。口腔深部见有淡黄色豆腐样附着物常因黏液窒息而死。治疗:病鸡立即隔离,鸡舍用2%热烧碱溶液消毒。治疗可选用…     

鸡传染性喉气管炎的防治

鸡传染性喉气管炎是由疱疹病毒引起的一种急性、接触性呼吸道传染病,主要特征是以呼吸困难,甩血条为特征。多发成鸡,死亡率高达25%。近几年多有发生,给养鸡业带来了较大的经济损失。笔者曾治疗过鸡传染喉气管炎的病例,因诊断治疗及时,并没有造成太大的经济损失1发病情况和症状2012年某养鸡专业户,养了1 000只蛋成鸡发病初期有软皮蛋、鸡蛋大小不等、产蛋量下降     

鸡传染性喉气管炎的防治

<正>冬季是鸡呼吸系统疾病流行季节,海安县部分乡镇养鸡场发生了传染性喉气管炎(ILT)。从未进行过ILT疫苗免疫的养鸡场发病较严重,发病率达50%以上,平均死亡率5%～20%。如有并发症,死亡率高达30%以上,经济损失很大。     

鸡传染性喉气管炎的防治

正鸡传染性喉气管炎是由疱疹病毒引起的鸡的一种急性呼吸道传染病。秋冬季节交替时多发,鸡传染性喉气管炎以育成鸡和成年产蛋鸡多发且症状典型。近年来发病日龄提前,最早可见20日龄的鸡群发病,由于致病毒株的毒力不同,病型可分为急性型和温和型两种。其特征为呼吸困难、咳嗽和咳出血痰。剖检可见喉部及气管黏膜肿胀、充血、出血、有时附着黄色干酪物。近年来该病在商品蛋鸡中发病率有所增加,带来较大的经济损失,应加强防疫,采取综合防治措施。     

鸡传染性喉气管炎的防治

鸡传染性喉气管炎对于鸡养殖者而言会产生较为不利的影响,一旦鸡感染了传染性喉气管炎,就会出现大批量死亡的现象,影响着鸡养殖者的经济效益。基于此,笔者对传染性喉气管炎的病鸡进行诊断分析,提出几点预防与治疗措施,以供相关人员参考。     

雏鸡传染性法氏囊病的诊治

20 0 1年 4月 30日 ,我县城关镇某专业户饲养的 30日龄罗曼雏鸡突然发病 ,根据临床症状和病理剖检典型特征 ,初诊为鸡传染性法氏囊病。经用攻毒 2 0 0 0口服 ,抗病毒和青霉素、链霉素肌肉注射 ,三林合剂饮水 ,迅速控制了病情 ,取得了良好的治疗效果。1　发病情况该户采用立体育雏笼育雏 ,室内火炉保温 ,饲养规模 80 0只 ,定时饲喂 ,自由饮水 ,饲料为某公司生产的蛋小鸡颗粒料。雏鸡在 1日龄接种了马立克氏疫苗 ,7日龄接种了鸡新城疫 +传支Ｈ12 0 二联苗 ,因没有传染性法氏囊疫苗 ,未在 1 4日龄用该疫苗免疫。该鸡群饲养至 30日龄 ,突然发病 …     

Age resistance in chickens against infectious bursal disease

The day old broiler chickens possessing IBD precipitating maternal antibody when exposed either to IBD contaminated environment or challenged intrabursally with virulent virus at weekly intervals indicated 100% susceptibility around 4–5 weeks of age. However, chickens lacking maternal antibody upon intrabursal challenge were found susceptible by 2 weeks of age.     

Dexamethasone reduces infectious bursal disease mortality in chickens

Very virulent infectious bursal disease virus (vvIBDV) causes high mortality in chickens but measures to reduce the mortality have not been explored. Chickens (8–9 weeks) were treated with 3 agents before and during vvIBDV inoculation. Dexamethasone treatment reduced the mortality of infected chickens (40.7% vs. 3.7%; p < 0.001), but treatment with aspirin or vitamin E plus selenium did not affect the mortality. The bursa of Fabricius appeared to have shrunk in both dead and surviving chickens (p < 0.01). The results indicate that dexamethasone can reduce mortality in vvIBDV-infected chickens and may provide therapeutic clues for saving individual birds infected by the virus.     

Immunogenicity of infectious bursal disease viruses in chickens.

Cross-protective properties of infectious bursal disease viruses (IBDVs) were studied. Viruses represented different subtypes of serotype 1, including recently isolated viruses (variants), and a serotype 2 virus. Chickens were vaccinated at 3 weeks of age with inactivated vaccines containing 10(5), 10(6), 10(7), or 10(8) mean tissue-culture infectious dose of a given virus and challenged 2 weeks later using either 10(2) or 10(3.5) mean embryo infectious dose (EID50) of either a standard virus or a variant serotype 1 virus. Protection was evaluated at 5 and 10 days post-challenge, based on gross and microscopic lesions, body weight, and bursa/body-weight ratios. The serotype 2 virus did not confer protection on birds challenged with the serotype 1 viruses. Vaccines made of variant viruses at the low doses protected chickens challenged with the high or low doses of either the standard or the variant viruses. Vaccines made of the standard or variant strains at low doses protected against high or low challenge doses of the standard strain. Vaccines made of the high dose of any of the standard strains protected chickens against the variant virus when the low challenge dose (10(2) EID50) was used, but not when the high challenge dose (10(3.5) EID50) was used. The lowest dose of the standard viruses vaccines required to confer protection against the variant virus varied depending on the strain. Results indicated that protection depended on the strain and dose of both the vaccine and challenge viruses and that the variant strains and standard strains share a common protective antigen(s).     

鸡传染性法氏囊炎并发非典型新城疫的诊治体会

2000年到2002年间,笔者在广饶、垦利、博兴、临淄、寿光等县区搞技术服务时,多次遇到过鸡传染性法氏囊炎和新城疫并发感染的病例,从而积累和总结了一些防治这类传染病的有效经验和措施,在此介绍给大家。1流行特点这类情况多出现在个体养殖户较集中的养殖小区和庭院散养户当中。常发于2～5周龄的肉仔鸡,3周龄前后发病最为常见,偶见于蛋鸡。一年四季均有发生,但以夏季和入冬潮湿季节最为多见。2发病症状病初,鸡群的采食量略有下降,2～3d后采食量明显下降,有个别鸡只开始出现死亡;饮水变化不明显或者略减少(这与单一暴发鸡传染性法氏囊病有所不…     

鸡传染性法氏囊病与球虫病混合感染的诊疗报告

广东省汕头市潮南区两英镇一养鸡户饲养的麻鸡在33日龄时,发生了以精神沉郁、闭目呆立、羽毛松乱、行动迟钝、怕冷打堆、排带有白色红色相间的水样稀便为主要症状,以法氏囊肿大,囊腔内有奶油状物、干酪状物或覆盖着果酱样粘液,两条盲肠高度肿大,肠腔内充满凝固的或新鲜的血液为主要病变的传染病。根据流行病学调查、综合临床症状、病理变化,初步诊断为鸡传染性法氏囊病与球虫病混合感染,后经实验室检查,确诊。经采取综合性治疗措施,取得良好的效果,现将诊疗情况报告如下:1发病情况广东省汕头市潮南区两英镇一黄姓养鸡户,于2004年5月14日购进…     

鸡传染性法氏囊病的诊断

鸡传染性法氏囊炎病是一种主要危害鸡的体液免疫中枢器官——法氏囊的病毒性传染病。牡丹江市某大型养鸡场2006年10月,购自某种鸡场海     

Effect of bursal cell number on the pathogenesis of infectious bursal disease in chickens

The pathogenesis of infectious bursal disease (IBD) in chickens neonatally chemically bursectomized (CB) by cyclophosphamide and subsequently inoculated with various numbers of bursal cells was examined. CB chickens inoculated with at least 62.5 X 10(6) bursal cells were as susceptible to IBD clinical manifestations (as determined by gross and microscopic evaluation of bursal tissues, virus recovery from spleen, and antibody titer) as intact chickens following inoculation with virus at 5 weeks of age. In contrast, CB chickens inoculated with 2.5 X 10(6) or fewer bursal cells were refractory to the IBD clinical manifestations compared with intact chickens or CB chickens inoculated with 62.5 X 10(6) or more bursal cells. Results from this study suggest that the availability of a large number of bursal cells is an essential factor in the development of IBD.