蛋鸡脂肪肝综合征的病因及其发病机理

蛋鸡脂肪肝综合症是一种营养代谢性疾病。它是由于鸡产蛋高峰期机体代谢旺盛 ,引起脂肪代谢紊乱 ,导致极低密度脂蛋白合成及转运受阻 ,使肝脏中合成的脂肪酸不能排出 ,造成甘油三酯在肝细胞内过度沉积。它导致青年蛋鸡不明原因的产蛋率下降和死亡率上升。本文对脂肪肝发病的症状、病因、发病机理进行综述     

笼养蛋鸡脂肪肝综合征的防治

脂肪肝出血综合征是由于笼养蛋鸡摄入能量过高的日粮，而运动又受到限制，导致能量代射失调，造成脂肪过度沉积的一种代谢疾病，本病主要发生在笼养鸡。     

高产蛋鸡群暴发脂肪肝综合征的诊治

鸡脂肪肝综合征主要是由于鸡体内脂肪代谢障碍而引起脂肪在肝脏中大量沉积 ,使肝脏发生脂肪变性的一种病症。1　发病概况本县柴镇科技园养殖场安某饲养蛋鸡 5 0 0 0只 ,至 2 60日龄 ,即 2 0 0 1年 4月 18日 ,开始发病 ,并呈急性死亡。用土霉素、四环素、恩诺沙星、氧氟沙星、菌必治等多种抗生素药物治疗均无效果。病鸡临床上不表现任何症状 ,多数为突然死亡 ,死鸡表现为贫血 ,如头部 ,鸡冠苍白 ,每天死亡 10只左右。产蛋母鸡的产蛋量下降 5 %～ 10 %。2　剖检变化腹腔内有凝血块 ,多数鸡肝脏破裂 ,内部有凝血块 ,肝呈土黄色、质脆、一触即碎…     

龙口市蛋鸡脂肪肝综合征的诊断与防治

2004年2月份以来，龙口市许多蛋鸡场大面积发生脂肪肝综合征，个别鸡场发生脂肪肝综合征并发弧菌肝炎和大肠杆菌感染，个别育成鸡群发生严重的弧菌肝炎。脂肪肝是产蛋鸡的一种营养代谢病，发病鸡群多处于开产初期或产蛋高峰期，产蛋率降低．死亡率高低不均。最高死亡率可达5％。据统计，本次发病多见来源于某一个种鸡场的某一个品种的鸡(如海兰)，由于鸡的品种不同，     

大肠杆菌感染引起蛋鸡肿头综合征

随着规模化养鸡业的发展 ,特别是鸡群的密度不断加大 ,鸡的大肠杆菌病日益严重 ,但大肠杆菌感染引发蛋鸡肿头综合征的报道却少见。 2 0 0 1年 9～1 0月 ,海南省某规模化蛋鸡场 32～ 34周龄产蛋鸡发生以单侧性头部肿胀、全眼球炎、鼻腔黏性分泌物增多 ,后期精神沉郁、闭目缩颈、食欲废绝、站立不稳为症状的一种疫情。经临床剖检、血清学诊断、病原分离鉴定、动物回归试验、治疗效果观察诊断为大肠杆菌引起鸡肿头综合征。报告如下。1　发病情况及临床症状　 2 0 0 1年 9月底 ,该场 32～ 34周龄产蛋鸡出现零星死亡 ,当时曾用一些抗生素进行防治…     

产蛋鸡脂肪肝综合征

产蛋鸡脂肪肝综合征 ( FLS)是一种复杂的代谢疾病 ,病因涉及多种因素。其特征是肝细胞中沉积大量脂肪 ,表现为脂肪肝 ,有时肝脏出血 ,鸡体肥胖 ,产蛋减少。有的病鸡因肝功能障碍或肝脏破裂而突然死亡。该病主要发生于产蛋率高或产蛋高峰期的鸡群 [1] 。本病发病率因鸡的品种、品系、日粮组成和环境等因素不同而有差异 ,通常为鸡群的 1 %～ 2 % ,有时高达 5 %～ 7%甚至 1 5 %以上。死亡率一般低于5 % ,但有时高达 2 0 %甚至 30 % [2 ,3 ] ,给养禽业造成了严重的经济损失。由于该病多散发 ,在国内尚未引起有关方面的足够重视。当肝脏出血和…     

蛋鸡脂肪肝综合征诱因及其营养调控措施

蛋鸡脂肪肝综合征是一种营养代谢疾病,多发于产蛋高峰期蛋鸡.本文综述了诱发蛋鸡脂肪肝综合征的主要因素及降低蛋鸡脂肪肝的营养调控措施.     

Mycoplasma gallinarum infection in commercial layers and onset of fatty liver hemorrhagic syndrome

Fatty liver hemorrhagic syndrome (FLHS) was observed in each of three trials in which commercial layers were utilized to determine the effect of Mycoplasma gallinarum (MGn) on egg and eggshell quality parameters and egg production. In each of three trials, FLHS occurred 31-54 days later in MGn-inoculated hens as compared with the Mycoplasma-clean (control) hens. In trials 1 and 2, no therapeutic intervention was initiated to ameliorate FLHS. In trial 3, therapeutic intervention was instituted and consisted of the addition of 1 pound of choline chloride/ton of feed. Total mortality recorded throughout the duration of each trial and attributable to FLHS was not significantly different between the control and the MGn-inoculated treatment. However, FLHS-associated mortality in each of the three trials was numerically greater for the control treatment.     

The histopathology of fatty liver and kidney syndrome in chicks.

Studies of the general histopathology of the fatty liver and kidney syndrome in chickens have shown abnormal accumulations of the lipid in a variety of organs but no degenerative or inflammatory reactions. Lipid was found in some skeletal muscles, alimentary tract, autonomic ganglia, central nervous system and pineal gland as well as in the liver, kidney and heart. Small amounts of lipid were sometimes seen in the exocrine pancreas, adrenal medulla and epithelium of the thyroid follicles. Lipid deposits in the liver were primarily associated with the hepatic structural unit. The glycogen content of the hepatic cell was reduced. The lipid-metabolising gastrocnemius muscle contained abnormal amounts of lipid but this did not apply to the carbohydrate-metabolising pectoralis major muscle. The thymus did not contain excessive lipid but was significantly smaller in affected than in control birds of similar ages. There was loss of tinctorial distinction between the cortex and medulla of the adrenal gland associated with decreased basophilia of the latter region. Many of these morphological changes can be correlated with previously reported biochemical findings and they are discussed in relation to the hyperlipaemia and hypoglycaemia which characterise the disease.     

脂肪肝综合症对蛋鸡产蛋性能、肝重、腹脂重及肝脏组织学变化的影响研究

蛋鸡脂肪肝综合症 (Fattyliversyndrome ,FLS)或脂肪肝出血性综合症 (FattyLiverHemorrhagicSyndrome ,FLHS)是产蛋鸡的一种常见病。其特征为病鸡肝肿大 ,多脂和易碎。本病常见于产蛋高峰期膘情好的蛋鸡 ,并引起蛋鸡突然死亡 ,造成很大的经济损失。该病病因较复杂 ,目前大多数研究认为 ,饲料因素是脂肪肝综合症的主要病因。本研究旨在通过不同饲料日粮诱发蛋鸡脂肪肝综合症 ,以探讨该病对蛋鸡产蛋性能、肝重、腹脂重及肝脏组织学变化的影响。1　材料与方法1 1　试验鸡选择、分组及管理　选择 1 4周龄体重 1 40 0～ 1 5 0 0g的艾维因蛋…     

The involvement of biotin in preventing the fatty liver and kidney syndrome in chicks.

Three experiments were carried out to investigate the involvement of vitamins in the fatty liver and kidney syndrome. The compounds studied, singly and in combination, were thiamin, riboflavin, nicotinic acid, pyridoxine, pantothenic acid, biotin, folic acid vitamin B12, ascorbic acid, choline and inositol and of these, only biotin prevented the syndrome. The minimum levels of supplemental dietary biotin required to prevent mortality varied from 0-05 to 0-15 mg/kg, depending on the diet. These levels were higher than the amounts required for maximum liveweight.