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植物Cu,Zn-SOD分子调控机理研究进展
引用本文:姚婕,赵艳玲. 植物Cu,Zn-SOD分子调控机理研究进展[J]. 热带农业科学, 2014, 0(3): 55-61
作者姓名:姚婕  赵艳玲
作者单位:华侨大学化工学院,福建厦门361021
基金项目:国家自然科学基金项目(No.31300497);华侨大学高层次人才科研启动基金项目.
摘    要:铜/锌超氧化物歧化酶(Cu,Zn-SOD)能清除植物体内有害的活性氧(ROS),参与植株遭受逆境胁迫时的应激反应等过程。铜分子伴侣(CCS)可以传递铜离子到Cu,Zn-SOD当中,并将其激活生成有活性的酶分子,依赖CCS协助是Cu,Zn-SOD主要的激活途径。植物在铜缺乏的环境下会诱导启动子结合蛋白(SPL7)和小RNA398(miR398)的表达,miR398通过降解编码Cu,Zn-SOD的mRNA抑制Cu,Zn-SOD的生成,从而调控植物体内铜平衡。本文主要对植物Cu,Zn-SOD激活和调控途径进行综述。

关 键 词:Cu  Zn—SOD  CCS  miR398  SPL7  调控途径

Molecular Regulation Mechanism Research Progress on Plant Cu,Zn-SOD
YAO die ZHAO Yanling. Molecular Regulation Mechanism Research Progress on Plant Cu,Zn-SOD[J]. Chinese Journal of Tropical Agriculture, 2014, 0(3): 55-61
Authors:YAO die ZHAO Yanling
Affiliation:YAO die ZHAO Yanling (College of Chemical Engineering, Huaqiao University, Xiamen, Fujian 361021)
Abstract:Superoxide dismutases (Cu,Zn-SODs) are important antioxidant enzymes that catalyze the disproportionation of superoxide anion to oxygen and hydrogen peroxide to guard cells against superoxide toxicity. The major pathway for activation of copper/zinc SOD (CSD) requiring the CCS copper chaperone to insert copper and activate SOD1 through oxidation of an intramolecular disulfide. Expression of miR398 and SPL7 (for SQUAMOSA promoter binding protein-like7) are induced in response to copper deficiency and miR398 is involved in the degradation of mRNAs encoding copper/zinc superoxide dismutase. This paper reviewed on plant Cu, Zn-SOD activation and regulatory pathways.
Keywords:Cu,Zn-SOD  CCS  miR398  SPL7  regulatory pathways
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