实验性雏鸭缺硒病的病理形态学研究

该试验结果表明:①渗出性素质,突出表现为心包积液显著且发生率高,皮下胶样水肿比雏鸡缺硒病轻,剥皮前难以认出;②胸腺坏死,特别是因胸腺发育迟滞而明显小于同龄对照组;③胰腺坏死,体积变小,但与雏鸡缺硒病不同,即未呈现网状纤维显露和所谓纤维化;④骨骼肌变性、同质化、坏死和出血;⑤心肌和十二指肠平滑肌同质化和坏死;⑥肌胃同质化、坏死及钙盐沉着,病变比雏鸡缺硒病发生率高;⑦肝脏显著脂肪变性;⑧淋巴器官(胸腺、法氏囊和脾脏)的淋巴组织坏死。

STUDIES ON PATHO-MORPHOLOGY OFV EXPERIMENTAL SELENIUM-DEFICIENCY IN DUCKLINGS

One-day-old Beijing Ducklings fed on low-selenium corn-soya basal diet(BD)mixed with necessary nutrient except selenium were divided into threegroups.Group I was fed BD+VE(administering 10 mg/kg diet)and intramus-eularly injected VE;Group ⅡBD+VE(10 mg/kg diet),and group Ⅲ BD+Se0.2 ppm+VE.Every week,five ducklings from each group were killed andexamined for eight weeks.The result found in macroscopic and microscopicexaminations showed main lesion changes in selenium-deficiency ducklingswere as follows:1.exudative diathesis,such as outstanding findings showing hydroper-icardium seen conmonly,subcutaneous colloidal oedema,but slighter in thechicks;2.thymus necrosis,especially its size was distinctly smaller than in thecontrol group due to its sluggish developing;3.pancreas necrosis,its size became small,but the reticular fiber andwhat is called fibrosis did not appear as in chieks;4.skeletal muscele degeneration,homogenization,necrosis and haemor-rhagia;5.homogenization and necrosis of smooth muscle of cardiac and duodenum;6.stomaches muscularis homogenization and necrosis,and eyes showingremackably pale;7.obviousl fat degeneration of liver;8.lymph tissue neerosis(thymus,bursa of Fabricius and spleen).