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R. Nayar 《Forest Pathology》1980,10(4):236-242
In the natural sandal reserves arsenic compounds have not proved successful in erradicating the spike disease; benlate has no curative or remission effect; tetracyclines have remission effects but application is not practical in forests due to the low density of sandal per hectare and difficulty in locating diseased trees. Compounds tried by various workers have provided no cure in the field. As vegetative propagation in sandal has not been successful, heat therapy also cannot be taken up. Eucalyptus has failed to control spike. But encouraging results have been obtained in checking the incidence of spike in man made forests by soil working, use of cover crop, interplanting sandal with trees giving shade at different levels, thus reducing the loss of water by evapotranspiration. In the natural reserves where the above conditions are prevalent spike disease has not been reported for the last 80 years.  相似文献   
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Genetic Resources and Crop Evolution - Correcting taxonomic misidentifications is crucial to ensuring the conservation and use of wild relatives in crop breeding programmes. Macrotyloma...  相似文献   
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The genetic bases of leaf rust resistance in wheat (Triticum aestivum L.) line CSP44, selected from the Australian cultivar Condor, and Indian cultivar VL404, were studied. The reaction patterns of CSP44 and VL404 against Indian races 12, 77, 77-1, 77-2, 77-3, 77-4, 77-5 and 108 were different from reaction patterns shown by near-isogenic lines with known adult plant resistance (APR) genes, viz. Lr12, Lr13, Lr22b and Lr34. Although the reaction patterns of CSP44 and VL404 were similar to the near-isogenic line Tc+Lr22a, tests of allelism indicated absence of Lr22a in both CSP44 and VL404. On the basis of genetic studies, their resistances in field tests against race 77-5, the most virulent race from the Indian sub-continent, were each ascribed to two genes. One of the two genes in each wheat was identified to be the non-hypersensitive APR gene Lr34. The second APR genes in CSP44 and VL404 gave hypersensitive reaction types and were recessive and dominant, respectively. The gene in CSP44 was designated Lr48and the gene in VL404, Lr49. This revised version was published online in August 2006 with corrections to the Cover Date.  相似文献   
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One or both eyes of 20 calves were inoculated one or more time with variou(s combinations of microorganism (live oor killed Moraxella bovis, infectious bovine rhinotracheitis virus, bovine adenovirus, bovine parainfluenza-3 virus and Mycoplasma bovoculi) by conjunctival instillation or direct inoculation of the conjunctivea or cornea. The eyes of all the calves received natural or artificial ultraviolet irradiation. Neither the adenovirus nor parainfluenza-3 virus became established in the eye or produced keratoconjunctivitis. Both M. bovis and infectious bovine rhinotracheitis virus became established in the bovine eye and produced disease. Subconjunctival or intracorneal inoculation of M. bovis caused a severe disease, simulating natural infectious bovine keratoconjunctivitis. Only the intracorneal inoculation of mycoplasma produced severe keratoconjunctivits. Eyes that on initial exposure to M. bovis became severly inflamed were more resistant to a second or third exposure to M. bovis, presumably by enhanced local defence mechanisms.  相似文献   
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