基于响应面法的马铃薯分选机参数优化及试验*

为减少马铃薯在分选过程中的跌落损伤,探讨分选机的工作条件对马铃薯跌落损伤的影响,以“荷兰15”马铃薯为试验材料,以马铃薯分选机的输送速度、剔除力、马铃薯的下落高度和含水率为试验因素,以马铃薯的跌落损伤等级为响应值,进行二次回归正交旋转试验,通过Design-Expert 8.0.6软件对试验结果畸形方差分析,并通过响应面探究各试验因素对马铃薯的跌落损伤等级的影响规律,确定马铃薯分选机的最佳工作参数。跌落试验结果表明:对马铃薯跌落损伤等级的影响程度主次顺序为马铃薯含水率、下落高度、输送速度、剔除力。当马铃薯输送速度为0.3 m/s,剔除力为15 N,下落高度为45 cm,含水率为75.36%时,在该参数组合下分选机对马铃薯跌落损伤等级最小。试验所得马铃薯的实际跌落损伤等级为0.84,该结果与马铃薯跌落损伤预测等级具有良好的拟合性。     

极端低温对晚熟柑桔产业的影响及冻后补救措施

2020年冬季的极端低温天气对全国晚熟柑桔产业造成了较为严重的影响。为将冻害损失降至最低程度,以南充地区为例,通过实地调查、分析对比冻害发生情况,提出了晚熟柑桔冻害发生后的补救措施,为促进南充及相似产区晚熟柑桔产业有序恢复及后续发展提供参考。     

Influence of phosphate-buffered sucrose solution on early graft function in feline renal autotransplantation

Graft perfusion with cold heparinized saline has known to induce ischemia and reperfusion injury in feline kidney transplantation. In this study, the effects of phosphate-buffered sucrose solution and heparinized saline solution on early kidney graft function were compared in feline kidney autotransplantation. Perfusion of grafts with or without hypothermic storage with chilled phosphate-buffered sucrose solution prevented ischemia and reperfusion injury despite a very short ischemic time. The results of our study suggest that phosphate-buffered sucrose perfusion and storage solution should be effective to reduce ischemia and reperfusion injury despite a very short ischemic time in feline kidney transplantation.     

低温预贮对‘红阳’猕猴桃果实冷害及CBF转录因子表达的影响

以‘红阳’猕猴桃果实为试材,研究低温预贮[(5±1)℃3d]对‘红阳’猕猴桃果实冷害及转录因子CBF(C-repeat binding factor)表达的影响。结果表明,低温预贮处理可以有效地降低‘红阳’猕猴桃果实冷害率和冷害指数,显著减少膜脂过氧化产物丙二醛(MDA)的积累和抑制脂氧合酶(LOX)活性的增加,有效地抑制果实呼吸速率和乙烯释放速率,保持较高的过氧化物酶(POD)活性,降低多酚氧化酶(PPO)活性,促进CBF转录因子的表达,在贮藏末期失水较少,保持较高的好果率。可见,低温预贮处理可以减轻‘红阳’猕猴桃果实在低温贮藏过程中的冷害发生,促进CBF转录因子的表达,对控制‘红阳’猕猴桃果实冷害有较好的作用。     

云南师范大学校园植物受冻害调查与植物合理配置分析

2012年末至2013年初,昆明出现的极端低温和持续低温天气,导致云南师范大学校园的部分植物遭受冻害。对受冻害植物进行调查,包括云南师范大学呈贡校区内的植物,总计71科178种。划分植物受冻害级别,分析不同受害级别植物的数量与受害程度,对冻害原因进行分析和讨论,提出植物合理配置和植物防冻害保护建议。     

2013年滇西石斛寒害情况调查

报道2013年冬云南滇西7个县(市)人工栽培石斛寒害灾情和灾后恢复情况;调查发现,不同石斛种抗寒性有很大差异,尖刀唇石斛、报春石斛、铁皮石斛寒害率较低,抗寒性强,而晶帽石斛、兜唇石斛、齿瓣石斛寒害率较高,抗寒性明显较弱。     

Protective effect of emodin on lung injury induced by hepatic fibrosis in rats

AIM:To explore the protective effect of emodin on lung injury induced by hepatic fibrosis in rats. METHODS:The hepatic fibrosis rat model was established with multiple pathogenic factors (CCl 4, ethanol, high fat, high cholesterol and low choline) and treated with different doses (20 mg/kg and 40 mg/kg) of emodin for 4 weeks. The hepatic index was measured. The biochemical indexes, endotoxin, homocysteine, albumin, aspartate aminotransferase,alanine aminotransferase, total bilirubin, total cholesterol and triglyceride, and hepatic fibrosis indexes, hyaluronic acid, laminin, collagen IV and procollagen Ⅲ, were detected. The histopathological changes of the liver were observed. The pulmonary index was determined. The histopathological changes of the lungs were observed. The levels of tumor necrosis factor α (TNF-α), malondialdehyde (MDA), nitric oxide (NO) and peroxynitrite (ONOO-) in the lung tissues were analyzed. RESULTS:The rat hepatic fibrosis model was successfully established. In model group, lung edema and inflammation occurred, and the pulmonary index and the levels of TNF-α, MDA, NO and ONOO- in the lung tissues were increased significantly. In emodin treatment groups, the pulmonary indexes were lower than that in model group. The pathological injury of the lung tissues was alleviated. The levels of TNF-α, MDA, NO and ONOO- in the lung tissues were decreased. CONCLUSION:Emodin has a protective effect on lung injury induced by hepatic fibrosis in rats.