The increasing scarcity of red oaks in Mississippi River floodplain forests: Influence of the residual overstory

Red oaks – cherrybark oak (Quercus pagoda Raf.), willow oak (Quercus phellos L.), water oak (Quercus nigra L.), and Nuttall oak (Quercus texana Buckley; aka: Quercus nuttallii Palmer) – are not regrowing in Mississippi Delta river floodplain forests in the southeastern United States in sufficient numbers to sustain the former species composition and timber and wildlife values. Even if vigorous red oak reproduction becomes established, partial harvesting that does not remove the taller trees will suppress understory red oak height growth more than it will suppress height growth of such other species as sugarberry (Celtis laevigata Willd.), American elm (Ulmus americana L.), cedar elm (Ulmus crassifolia Nutt.), swamp dogwood (Cornus foemina Mill.), green ash (Fraxinus pennsylvanica Marshall), and sweetgum (Liquidambar styraciflua L.). Consequently, the red oaks in these partially harvested stands become increasingly suppressed and probably die; and there is a shift in species composition to the other species. In addition to ensuring vigorous oak reproduction, silvicultural clearcutting or rapid removal of the residual trees following shelterwood or seed tree harvesting to provide full sunlight is needed to ensure red oaks become a dominant part of these future river floodplain stands.     

The WEELS model: methods, results and limitations

Within the European Union (EU)-funded Project ‘Wind Erosion on European Light Soils’ (WEELS), a model was designed and implemented with the aim of predicting the long-term spatial distribution of wind erosion risks in terms of erosion hours and wind-induced soil loss. In order to ensure wide applicability, the model structure consists of a modular combination of different approaches and algorithms, running on available or easily collected topographic and climatological data input. Whereas the ‘WIND’, ‘WIND EROSIVITY’ and ‘SOIL MOISTURE’ modules combine factors that contribute to the temporal variations of climatic erosivity, the ‘SOIL ERODIBILITY’, ‘SURFACE ROUGHNESS’ and ‘LAND USE’ modules predict the temporal soil and vegetation cover variables that control soil erodibility. Preliminary simulations over a 29-year period for the Barnham site (UK) (1970–1998) and a 13-year period for the Grönheim site (Germany) (1981–1993) generally resulted in a higher erosion risk for the English test site, where the total mean soil loss was estimated at 1.56 t ha⁻¹ year⁻¹ and mean maximum soil loss at about 15.5 t ha⁻¹ year⁻¹. The highest rates exceeded 3 t ha⁻¹ in March, September and November. On the northern German test site, the total mean soil loss was 0.43 t ha⁻¹ year⁻¹. The highest erosion rates were predicted in April when they can exceed 2.5 t ha⁻¹. The total mean maximum soil loss at this site of about 10.0 t ha⁻¹ year⁻¹ corresponds to a loss of about 0.65 mm. Predictions based on a land use scenario for the German site revealed that the erosion risk could be reduced significantly by changing land use strategies.     

Coincident scrapie infection and nephritis lead to urinary prion excretion

Prion infectivity is typically restricted to the central nervous and lymphatic systems of infected hosts, but chronic inflammation can expand the distribution of prions. We tested whether chronic inflammatory kidney disorders would trigger excretion of prion infectivity into urine. Urinary proteins from scrapie-infected mice with lymphocytic nephritis induced scrapie upon inoculation into noninfected indicator mice. Prionuria was found in presymptomatic scrapie-infected and in sick mice, whereas neither prionuria nor urinary PrP(Sc) was detectable in prion-infected wild-type or PrP(C)-overexpressing mice, or in nephritic mice inoculated with noninfectious brain. Thus, urine may provide a vector for horizontal prion transmission, and inflammation of excretory organs may influence prion spread.     

Experimental inoculation of raccoons (Procyon lotor) with Spiroplasma mirum and transmissible mink encephalopathy (TME)

The primary objective of this study was to determine whether or not Spiroplasma mirum would be capable of producing lesions of transmissible spongiform encephalopathy (TSE) when inoculated in raccoons (Procyon lotor) and, if that was possible, to compare the clinicopathological findings with those of transmissible mink encephalopathy (TME) in the same experimental model. For this purpose, 5 groups (n = 5) of raccoon kits were inoculated intracerebrally with either S. mirum and/or TME. Two other groups (n = 5) of raccoon kits served as sham-inoculated controls. All animals inoculated with TME, either alone or in combination, showed clinical signs of neurologic disorder and were euthanized within 6 mo post-inoculation (MPI). None of the carcasses revealed gross lesions. Spongiform encephalopathy was observed by light microscopy and the presence of abnormal disease-causing prion protein (PrP(d)) was detected by immunohistochemistry (IHC) and Western blot (WB) techniques in only the raccoons administered TME. Raccoons inoculated with Spiroplasma, but not administered TME agent, were euthanized at 30 MPI. They did not show clinical neurologic signs, their brains did not have lesions of spongiform encephalopathy, and their tissues were negative for S. mirum by polymerase chain reaction (PCR) and for PrP(d) by IHC and WB techniques. The results of this study indicate that Spiroplasma mirum does not induce TSE-like disease in raccoons.     

Adrenocorticotropic hormone, but not trilostane, causes severe adrenal hemorrhage, vacuolization, and apoptosis in rats

Adrenal necrosis has been reported as a complication of trilostane application in dogs with hyperadrenocorticism. One suspicion was that necrosis results from the increase of adrenocorticotropic hormone (ACTH) during trilostane therapy. The aim of the current study was to assess the effects of ACTH and trilostane on adrenal glands of rats. For experiment 1, 36 rats were divided into 6 groups. Groups 1.1 to 1.4 received ACTH in different doses (60, 40, 20, and 10 μg/d) infused subcutaneously with osmotic minipumps for 16 wk. Group 1.5 received saline, and group 1.6 received no therapy. For experiment 2, 24 rats were divided into 3 groups. Group 2.1 and 2.2 received 5 and 50 mg/kg trilostane/d orally mixed into chocolate pudding for 16 wk. Eight control rats received pudding alone. At the end of the experiments, adrenal glands were assessed for necrosis by histology and immunohistochemistry; levels of endogenous ACTH and nucleosomes were assessed in the blood. Rats treated with 60 μg ACTH/d showed more hemorrhage and vacuolization and increased numbers of apoptotic cells in the adrenal glands than rats treated with 20 or 10 μg ACTH/d, trilostane, or control rats. Rats treated with 60 μg ACTH/d had a higher amount of nucleosomes in the blood compared with rats treated with 10 μg ACTH/d, trilostane, or saline. We conclude that in healthy rats ACTH, but not trilostane, causes adrenal degeneration in a dose-dependent manner. Results of this study support the hypothesis that adrenal gland lesions seen in trilostane-treated dogs are caused by ACTH and not by trilostane.     

温室盆栽郁金香促成栽培

Longwood花园的郁金香花展是自1921年以来一直流传下来的传统。在11月到翌年4月期间,温室里进行的盆栽郁金香花展非常引人注目。盆栽郁金香种植促成盆栽郁金香,首先应该选择高质量的种球,其级别是12+级即球根的周长是12cm或者更大,这个级别对     

美国杜邦花园水仙盆栽生产技术

美国杜邦花园每年冬天都会生产成千上万盆水仙,让人眼前一亮,暖意浓浓,预示着春天的到来。本文作者为杜邦花园种植者,他从春化处理到开花销售,详尽介绍了水仙盆栽生产成功的诀窍及其种植流程。