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早期断奶仔猪的断奶应激与腹泻研究 总被引:1,自引:0,他引:1
早期断奶仔猪一方面消化系统发育不成熟,吸收养分能力差,消化酶和胃酸分泌不足,正常的肠道微生物系统尚未建立;另一方面断绝了从母体获得被动免疫的来源,而自身的主动免疫发育尚未成熟。所以断奶应激造成早期断奶仔猪出现消化不良、生长迟滞、抗病力弱等“仔猪早期断奶综合症”。仔猪肠道对日粮抗原过敏从而导致肠道损伤是断奶后腹泻的根本原因。早期补饲、逐步断奶、去母留仔、合理饲喂可预防早期断奶仔猪腹泻。 相似文献
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早期断奶仔猪一方面消化系统发育不成熟,吸收养分能力差,消化酶和胃酸分泌不足,正常的肠道微生物系统尚未建立,断奶应激以及日粮抗原过敏导致肠道损伤从而引起仔猪的腹泻;另一方面断绝了从母体获得被动免疫的来源,而自身的主动免疫发育尚未成熟,易受外界环境的影响,受到有害菌的侵袭,也是造成仔猪断奶后腹泻的一个重要原因。 相似文献
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早期断奶仔猪一方面消化系统发育不成熟,吸收养分能力差,消化酶和胃酸分泌不足,正常的肠道微生物系统尚未建立,断奶应激以及日粮抗原过敏导致肠道损伤从而引起仔猪的腹泻;另一方面断绝了从母体获得被动免疫的来源,而自身的主动免疫发育尚未成熟,易受外界环境的影响,受到有害菌的侵袭,也是造成仔猪断奶后腹泻的一个重要原因. 相似文献
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早期断奶仔猪一方面消化系统发育不成熟,吸收养分能力差,消化酶和胃酸分泌不足,正常的肠道微生物系统尚未建立,断奶应激以及日粮抗原过敏导致肠道损伤从而引起仔猪的腹泻;另一方面断绝了从母体获得被动免疫的来源,而自身的主动免疫发育尚未成熟,易受外界环境的影响,受到有害菌的侵袭,也是造成仔猪断奶后腹泻的一个重要原因. 相似文献
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杨银庆 《河南畜牧兽医(综合版)》2009,(5):39-39
仔猪早期断奶的目的是防止疫病传播、降低生产成本、提高仔猪的生产性能和提高母猪的繁殖率。早期断奶仔猪由于消化功能及免疫系统尚未发育成熟,同时遇到营养和环境等因素的应激。容易出现采食量低、生长发育慢及体重下降、腹泻甚至死亡等现象。因此,需要采取综合措施,防止仔猪断奶应激综合征的发生,提高断奶仔猪的成活率。 相似文献
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梁方印 《河南畜牧兽医(综合版)》2009,30(9)
仔猪早期断奶的目的是防止疫病传播、降低生产成本、提高仔猪的生产性能和提高母猪的繁殖率.早期断奶仔猪由于消化功能及免疫系统尚未发育成熟,同时遇到营养和环境等因素的应激,容易出现采食量低、生长发育慢及体重下降、腹泻甚至死亡等现象. 相似文献
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早期断奶仔猪由于消化系统发育不成熟,消化酶系统不健全,胃酸分泌不足,断奶时母体提供的抗体停止,而仔猪自身的主要免疫系统发育未完善,对疾病和应激抵抗能力下降。加上受生理环境和营养应激等因素影响,导致仔猪食欲差、消化不良、生长抑制、饲料利用率低、腹泻甚至死亡。因此,在早期断奶仔猪饲料中急需添加一些可促进仔猪肠道发育、增强免疫力、提高生产性能、防治下痢等功能的饲料添加剂。 相似文献
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仔猪早期断奶是提高母猪繁殖利用率的重要措施,但早期断奶应激容易导致仔猪断奶后食欲下降,消化机能紊乱,腹泻等“仔猪早期断奶综合症”.发酵豆粕因富含植物小肽,易于动物的消化吸收等多种优点,适用于幼龄、病弱、应激等特殊生理状况下的仔猪.本试验选用市场上常用的一种同类产品与一种新开发的发酵豆粕在断奶仔猪上进行饲喂对比.结果表明:这种新开发的产品对缓解仔猪早期断奶应激引起的胃肠机能紊乱明显优于对照产品,其稀便发生率显著低于对照组(P<0.01);平均日增重、料肉比高于对照组,但差异不显著(P>0.05). 相似文献
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本试验将95头新生迪卡小公猪随机分为4组,第1、2、3组每组19头,均由两头经产母猪哺乳喂养,21日龄断奶。第4组仔猪38头,由4头经产母猪哺乳喂养,35日龄断奶。7日龄起至35日龄各组仔猪分别饲喂不同蛋白质来源的日粮。试验各组仔猪在24、28和35日龄进行屠宰试验,取胃内样品进行酶活性测定。试验结果表明:21日龄断奶使仔猪胃内蛋白酶、脂肪酶活性显著降低,酶活性在1周内不能得到完全恢复,胃蛋白酶活性可在断奶后2周得到恢复;全植物性蛋白日粮使早期断奶仔猪胃内蛋白酶和脂肪酶活性显著降低;日粮中加一定量乳清粉和动物性蛋白质饲料能有效提高胃蛋白酶和脂肪酶的活性。因此,断奶及日粮蛋白质来源显著影响早期断奶仔猪胃内消化酶活性。 相似文献
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早期断奶仔猪营养研究进展 总被引:4,自引:0,他引:4
断奶应激造成早期断奶仔猪生长抑制,制约断奶效果,进而影响其将来生产性能。作者从早期断奶仔猪的消化生理、免疫系统的演变特点出发,就早期断奶仔猪的营养需要及当前对克服断奶应激采取的种种措施,特别是如何制定合理的饲养方案以保证仔猪最佳生长性能方面进行探讨。 相似文献
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《动物营养(英文)》2020,6(3):362-371
Diarrhea is considered to be associated with microbial dysbiosis caused by infection of pathogens but poorly understood. We herein characterized the colonic microbiota of diarrheal early-weaning piglets infected with porcine circovirus type 2 (PCV2) and Campylobacter. Campylobacter infection significantly decreased species richness and Shannon diversity index of colonic microbiota together with a significant increase in the proportion of Campylobacter and Enterobacteriaceae, whereas no significant difference on the above indexes was observed in piglets infected with PCV2 compared with healthy piglets. PCV2 and Campylobacter infection could disturb the homeostasis of colonic microbiota through deterioration of ecological network within microbial community, and specially Campylobacter performed as a module hub in ecological networks. The microbial dysbiosis caused metabolic dysfunction and led to a remarkable reduction in production of short chain fatty acids, following by a higher pH level in colon cavity. Campylobacter infection disturbed the function of colonic tract barrier observed in terms of significant lower relative expression of claudin-1, occluding, and zonula occludens protein-1 genes, and PCV2 infection induced intestinal inflammation together with a higher permeability of colon. Generally, these results suggested that PCV2 and Campylobacter infection could induce microbial dysbiosis and metabolic dysfunction, and cause intestinal disorder, all of which finally were associated to contribute to the diarrhea of early-weaning piglets. 相似文献
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S. L. Chopra A. C. Blackwood D. G. Dale 《Canadian journal of veterinary research》1963,27(12):290-294
Studies were conducted on the enumeration of 7 groups of fecal microflora including total aerobes, total anaerobes, coliforms, lactobacilli, staphylococci, streptococci and yeasts and molds of 18-day old piglets. These pigs were early weaned (21 days) on different modifications of an early-weaning ration. The above mentioned microflora were enumerated again when some of the pigs in a replicate started scouring. The occurrence of diarrhea was always associated with significant increases in the numbers of coliforms and corresponding decreases in the lactobacilli counts. No other single group of fecal microflora differed in the scouring and non-scouring animals. The composition of the early-weaning ration offered to the animals did not, in itself, influence the fecal microflora to any appreciable extent.
In another series of experiments, enumeration of coliforms and lactobacilli was conducted on samples obtained from different segments of the intestinal tracts of scouring and non-scouring pigs. Increased numbers of coliforms and decreased numbers of lactobacilli were observed at all levels of the intestinal tracts of the scouring animals. However, these changes were more marked in the duodenal samples than in those obtained from other parts of the intestine.
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